Change in Eosinophil Count in Patients with Heart Failure Treated with Anakinra.

cardiorespiratory fitness eosinophils exercise test heart failure injection site reaction interleukin-1 interleukin-1 receptor antagonist protein

Journal

Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052

Informations de publication

Date de publication:
11 04 2023
Historique:
received: 24 03 2023
revised: 07 04 2023
accepted: 10 04 2023
medline: 17 5 2023
pubmed: 16 5 2023
entrez: 16 5 2023
Statut: epublish

Résumé

Interleukin-1 blockade with anakinra leads to a transient increase in eosinophil blood count (eosinophils) in patients with acute myocardial infarction. We aimed to investigate the effect of anakinra on changes in eosinophils in patients with heart failure (HF) and their correlation with cardiorespiratory fitness (CRF). We measured eosinophils in 64 patients with HF (50% females), 55 (51-63) years of age, before and after treatment, and, in a subset of 41 patients, also after treatment cessation. We also evaluated CRF, measuring peak oxygen consumption (VO Treatment with anakinra significantly and transiently increased eosinophils, from 0.2 [0.1-0.3] to 0.3 [0.1-0.4] × 10 Patients with HF treated with anakinra experience a transient increase in eosinophils, which is associated with ISR and a greater improvement in peak VO

Sections du résumé

BACKGROUND
Interleukin-1 blockade with anakinra leads to a transient increase in eosinophil blood count (eosinophils) in patients with acute myocardial infarction. We aimed to investigate the effect of anakinra on changes in eosinophils in patients with heart failure (HF) and their correlation with cardiorespiratory fitness (CRF).
METHODS
We measured eosinophils in 64 patients with HF (50% females), 55 (51-63) years of age, before and after treatment, and, in a subset of 41 patients, also after treatment cessation. We also evaluated CRF, measuring peak oxygen consumption (VO
RESULTS
Treatment with anakinra significantly and transiently increased eosinophils, from 0.2 [0.1-0.3] to 0.3 [0.1-0.4] × 10
CONCLUSION
Patients with HF treated with anakinra experience a transient increase in eosinophils, which is associated with ISR and a greater improvement in peak VO

Identifiants

pubmed: 37190038
pii: cells12081129
doi: 10.3390/cells12081129
pmc: PMC10137267
pii:
doi:

Substances chimiques

Interleukin 1 Receptor Antagonist Protein 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NCRR NIH HHS
ID : KL2RR031989
Pays : United States
Organisme : NHLBI NIH HHS
ID : 1R34HL118348
Pays : United States
Organisme : NCRR NIH HHS
ID : UL1TR000058
Pays : United States
Organisme : NHLBI NIH HHS
ID : 1R34HL117026
Pays : United States
Organisme : NCRR NIH HHS
ID : UL1 RR031990
Pays : United States

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Auteurs

Michele Golino (M)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Medicine and Surgery, University of Insubria, 21100 Varese, Italy.

Francesco Moroni (F)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Internal Medicine, University of Virginia, Charlottesville, VA 22904, USA.
Department of Medicine, Università Milano-Bicocca, 20126 Milan, Italy.

Marco Giuseppe Del Buono (MG)

Department of Cardiovascular Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, 00168 Rome, Italy.

Justin M Canada (JM)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

Azita H Talasaz (AH)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Pharmacotherapy and Outcome Sciences, Virginia Commonwealth University, Richmond, VA 23284, USA.

Sebastian Piñel (S)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

James Mbualungu (J)

Robert M. Berne Cardiovascular Research Center, Division of Cardiology-Heart and Vascular Center, University of Virginia, Charlottesville, VA 22904, USA.

Alessandra Vecchiè (A)

Medicina Generale 1, Medical Center, Department of Internal Medicine, Ospedale di Circolo e Fondazione Macchi, ASST Sette Laghi, 21100 Varese, Italy.

Ai-Chen Jane Ho (AJ)

Department of Pharmacotherapy and Outcome Sciences, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Clinical & Administrative Sciences, School of Pharmacy, Notre Dame of Maryland University, Baltimore, MD 21210, USA.

Georgia K Thomas (GK)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

Salvatore Carbone (S)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Kinesiology & Health Sciences, College of Humanities & Sciences, Virginia Commonwealth University, Richmond, VA 23284, USA.

Hayley E Billingsley (HE)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Kinesiology & Health Sciences, College of Humanities & Sciences, Virginia Commonwealth University, Richmond, VA 23284, USA.

Jeremy Turlington (J)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

Roshanak Markley (R)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

Cory Trankle (C)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.

Roberto De Ponti (R)

Department of Medicine and Surgery, University of Insubria, 21100 Varese, Italy.

Benjamin Van Tassell (B)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Department of Pharmacotherapy and Outcome Sciences, Virginia Commonwealth University, Richmond, VA 23284, USA.

Antonio Abbate (A)

Pauley Heart Center, Virginia Commonwealth University, Richmond, VA 23284, USA.
Robert M. Berne Cardiovascular Research Center, Division of Cardiology-Heart and Vascular Center, University of Virginia, Charlottesville, VA 22904, USA.

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Classifications MeSH