Favorable survival outcomes in epidermal growth factor receptor (EGFR)-mutant non-small cell lung cancer sequentially treated with a tyrosine kinase inhibitor and osimertinib in a real-world setting.


Journal

Journal of cancer research and clinical oncology
ISSN: 1432-1335
Titre abrégé: J Cancer Res Clin Oncol
Pays: Germany
ID NLM: 7902060

Informations de publication

Date de publication:
Sep 2023
Historique:
received: 13 12 2022
accepted: 03 05 2023
medline: 31 7 2023
pubmed: 18 5 2023
entrez: 17 5 2023
Statut: ppublish

Résumé

EGFR tyrosine kinase inhibitor (TKI) therapy in EGFR-mutated lung cancer is limited by acquired resistance. In half of the patients treated with first/second-generation (1st/2nd gen) TKI, resistance is associated with EGFR p.T790M mutation. Sequential treatment with osimertinib is highly active in such patients. Currently, there is no approved targeted second-line option for patients receiving first-line osimertinib, which thus may not be the best choice for all patients. The present study aimed to evaluate the feasibility and efficacy of a sequential TKI treatment with 1st/2nd gen TKI, followed by osimertinib in a real-world setting. Patients with EGFR-mutated lung cancer treated at two major comprehensive cancer centers were retrospectively analyzed by the Kaplan-Meier method and log rank test. A cohort of 150 patients, of which 133 received first-line treatment with a first/second gen EGFR TKI, and 17 received first-line osimertinib, was included. Median age was 63.9 years, 55% had ECOG performance score of ≥ 1. First-line osimertinib was associated with prolonged progression-free survival (P = 0.038). Since the approval of osimertinib (February 2016), 91 patients were under treatment with a 1st/2nd gen TKI. Median overall survival (OS) of this cohort was 39.3 months. At data cutoff, 87% had progressed. Of those, 92% underwent new biomarker analyses, revealing EGFR p.T790M in 51%. Overall, 91% of progressing patients received second-line therapy, which was osimertinib in 46%. Median OS with sequenced osimertinib was 50 months. Median OS of patients with p.T790M-negative progression was 23.4 months. Real-world survival outcomes of patients with EGFR-mutated lung cancer may be superior with a sequenced TKI strategy. Predictors of p.T790M-associated resistance are needed to personalize first-line treatment decisions.

Identifiants

pubmed: 37198447
doi: 10.1007/s00432-023-04839-3
pii: 10.1007/s00432-023-04839-3
pmc: PMC10374675
doi:

Substances chimiques

osimertinib 3C06JJ0Z2O
Tyrosine Kinase Inhibitors 0
ErbB Receptors EC 2.7.10.1
Protein Kinase Inhibitors 0
Aniline Compounds 0
EGFR protein, human EC 2.7.10.1

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

9243-9252

Informations de copyright

© 2023. The Author(s).

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Auteurs

Oliver Kraskowski (O)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Jan A Stratmann (JA)

Department of Internal Medicine, Hematology/Oncology, University Hospital Frankfurt, Frankfurt, Germany.

Marcel Wiesweg (M)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Wilfried Eberhardt (W)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Martin Metzenmacher (M)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Kurt W Schmid (KW)

Institute of Pathology, West German Cancer Center, University Hospital Essen, Essen, Germany.
German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.

Thomas Herold (T)

Institute of Pathology, West German Cancer Center, University Hospital Essen, Essen, Germany.

Hans-Ulrich Schildhaus (HU)

Institute of Pathology, West German Cancer Center, University Hospital Essen, Essen, Germany.
German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.

Kaid Darwiche (K)

Section of Interventional Pneumology, Department of Pneumology, West German Cancer Center, University Medicine Essen-Ruhrlandklinik, Essen, Germany.

Clemens Aigner (C)

German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.
Department of Thoracic Surgery and Surgical Endoscopy, West German Cancer Center, University Medicine Essen-Ruhrlandklinik, Essen, Germany.

Martin Stuschke (M)

German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.
Department of Radiation Oncology, West German Cancer Center, University Hospital Essen, University Duisburg-Essen, Essen, Germany.

Katharina Laue (K)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Gregor Zaun (G)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Stefan Kasper (S)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.
German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.

Jörg Hense (J)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.

Martin Sebastian (M)

Department of Internal Medicine, Hematology/Oncology, University Hospital Frankfurt, Frankfurt, Germany.
German Cancer Consortium (DKTK), Partner Site University Hospital Frankfurt, Frankfurt, Germany.

Martin Schuler (M)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany.
German Cancer Consortium (DKTK), Partner Site University Hospital Essen, Essen, Germany.

Michael Pogorzelski (M)

Department of Medical Oncology, West German Cancer Center, University Hospital Essen, Hufelandstr. 55, 46147, Essen, Germany. michael.pogorzelski@uk-essen.de.

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