Neutrophilic dermatosis and hidradenitis suppurativa in patients with Behçet's disease: A neutrophilic disease in the spectrum of autoinflammatory syndromes.

Anti-TNFα Behçet's disease Hidradenitis suppurativa Neutrophilic dermatosis Pyoderma gangrenosum SAPHO Tocilizumab Ustekinumab

Journal

Seminars in arthritis and rheumatism
ISSN: 1532-866X
Titre abrégé: Semin Arthritis Rheum
Pays: United States
ID NLM: 1306053

Informations de publication

Date de publication:
Aug 2023
Historique:
received: 06 02 2023
revised: 05 05 2023
accepted: 10 05 2023
medline: 10 7 2023
pubmed: 20 5 2023
entrez: 19 5 2023
Statut: ppublish

Résumé

Association of neutrophilic dermatosis (ND), hidradenitis suppurativa (HS) and Behçet's disease (BD) and shared efficacy of TNFα axis blockade suggests common physiopathology. To investigate the clinical features and therapeutic response of ND and HS associated with BD. We identified 20 patients with ND or HS associated with BD among 1462 patients with BD. We analysed 20 (1.4%) patients diagnosed with ND or HS associated with BD: 13 HS, 6 pyoderma gangrenosum (PG), and 1 SAPHO. Our 6 PG cases over 1462 BD patients accounts for 400/100 000 prevalence. Thirteen had bipolar aphthosis, 6 vascular, 5 neurologic, and 4 ocular involvements. All PG occurred on limbs and had typical histology with constant dermal neutrophilic infiltrate. All HS had the classical axillary-mammary phenotype. Sixty-nine percent (69%) of HS were Hurley 1 stage. Treatment consisted mainly in colchicine (n = 20), glucocorticoids (n = 12), and anti-TNFα (n = 9). Interesting results with complete or partial responses were obtained with anti-TNFα (9 cases), ustekinumab (3 cases) and tocilizumab (1 case) to treat refractory ND or HS associated with BD. PG seems overrepresented in patients with BD. Biotherapies such as anti-TNFα, ustekinumab and tocilizumab appear to be promising to treat refractory ND or HS associated with BD.

Sections du résumé

BACKGROUND BACKGROUND
Association of neutrophilic dermatosis (ND), hidradenitis suppurativa (HS) and Behçet's disease (BD) and shared efficacy of TNFα axis blockade suggests common physiopathology.
OBJECTIVES OBJECTIVE
To investigate the clinical features and therapeutic response of ND and HS associated with BD.
METHODS METHODS
We identified 20 patients with ND or HS associated with BD among 1462 patients with BD.
RESULTS RESULTS
We analysed 20 (1.4%) patients diagnosed with ND or HS associated with BD: 13 HS, 6 pyoderma gangrenosum (PG), and 1 SAPHO. Our 6 PG cases over 1462 BD patients accounts for 400/100 000 prevalence. Thirteen had bipolar aphthosis, 6 vascular, 5 neurologic, and 4 ocular involvements. All PG occurred on limbs and had typical histology with constant dermal neutrophilic infiltrate. All HS had the classical axillary-mammary phenotype. Sixty-nine percent (69%) of HS were Hurley 1 stage. Treatment consisted mainly in colchicine (n = 20), glucocorticoids (n = 12), and anti-TNFα (n = 9). Interesting results with complete or partial responses were obtained with anti-TNFα (9 cases), ustekinumab (3 cases) and tocilizumab (1 case) to treat refractory ND or HS associated with BD.
CONCLUSION CONCLUSIONS
PG seems overrepresented in patients with BD. Biotherapies such as anti-TNFα, ustekinumab and tocilizumab appear to be promising to treat refractory ND or HS associated with BD.

Identifiants

pubmed: 37207416
pii: S0049-0172(23)00066-5
doi: 10.1016/j.semarthrit.2023.152224
pii:
doi:

Substances chimiques

Ustekinumab FU77B4U5Z0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

152224

Informations de copyright

Copyright © 2023 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None declared

Auteurs

Hélène Bugaut (H)

Department of Internal Medicine and Clinical Immunology, Sorbonne University, Pitié-Salpêtrière Hospital, APHP, 75013 Paris France, Centre de référence Maladies Autoimmunes systémiques rares, Centre de référence Maladies Autoinflammatoires et amylose, 83 boulevard de l'hôpital, Paris 75013, France.

Stéphane Barete (S)

Unit of Dermatology, Pitié-Salpêtrière Hospital, APHP, DMU3ID, Sorbonne Université, Paris 75013, France.

Martine Bagot (M)

Dermatology, Saint-Louis Hospital, Université Paris Cité, Paris 75010, France.

Jean-David Bouaziz (JD)

Dermatology, Saint-Louis Hospital, Université Paris Cité, Paris 75010, France.

François Le Pelletier de Glatigny (F)

Department of Pathology, Pitié-Salpêtrière Hospital, Sorbonne Université, Paris 75013, France.

Yves Gallien (Y)

Biostatistic, Saint-Louis Hospital, Université Paris Cité, Paris 75010, France.

Lucie Biard (L)

Biostatistic, Saint-Louis Hospital, Université Paris Cité, Paris 75010, France.

Fanny Domont (F)

Department of Internal Medicine and Clinical Immunology, Sorbonne University, Pitié-Salpêtrière Hospital, APHP, 75013 Paris France, Centre de référence Maladies Autoimmunes systémiques rares, Centre de référence Maladies Autoinflammatoires et amylose, 83 boulevard de l'hôpital, Paris 75013, France.

Patrice Cacoub (P)

Department of Internal Medicine and Clinical Immunology, Sorbonne University, Pitié-Salpêtrière Hospital, APHP, 75013 Paris France, Centre de référence Maladies Autoimmunes systémiques rares, Centre de référence Maladies Autoinflammatoires et amylose, 83 boulevard de l'hôpital, Paris 75013, France.

David Saadoun (D)

Department of Internal Medicine and Clinical Immunology, Sorbonne University, Pitié-Salpêtrière Hospital, APHP, 75013 Paris France, Centre de référence Maladies Autoimmunes systémiques rares, Centre de référence Maladies Autoinflammatoires et amylose, 83 boulevard de l'hôpital, Paris 75013, France. Electronic address: david.saadoun@aphp.fr.

Cloé Comarmond (C)

Department of Internal Medicine and Clinical Immunology, Lariboisière Hospital, Université Paris Cité, Paris 75010, France.

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Classifications MeSH