Azathioprine-induced vanishing bile duct syndrome: The value of early thiopurine metabolism assessment.


Journal

British journal of clinical pharmacology
ISSN: 1365-2125
Titre abrégé: Br J Clin Pharmacol
Pays: England
ID NLM: 7503323

Informations de publication

Date de publication:
08 2023
Historique:
revised: 13 05 2023
received: 03 03 2023
accepted: 17 05 2023
medline: 21 7 2023
pubmed: 23 5 2023
entrez: 23 5 2023
Statut: ppublish

Résumé

About 15% to 28% of patients treated with thiopurines experienced adverse drug reactions, such as haematological and hepatic toxicities. Some of these related to the polymorphic activity of the thiopurine S-methyltransferase (TPMT), the key detoxifying enzyme of thiopurine metabolism. We report here a case of thiopurine-induced ductopenia with a comprehensive pharmacological analysis on thiopurine metabolism. A 34-year-old woman, with a medical history of severe systemic lupus erythematosus with recent introduction of azathioprine therapy, presented with mild fluctuating transaminase blood levels consistent with a hepatocellular pattern, which evolved to a cholestatic pattern over the next weeks. A blood thiopurine metabolite assay revealed low 6-thioguanine nucleotides (6-TGN) level and a dramatically increased 6-methylmercaptopurine ribonucleotides (6-MMPN) level, together with an unfavourable [6-MMPN:6-TGN] metabolite ratio and a high TPMT activity. After a total of about 6 months of thiopurine therapy, a transjugular liver biopsy revealed a ductopenia, and azathioprine discontinuation led to further clinical improvement. In line with previous reports from the literature, our case supports the fact that ductopenia is a rare adverse drug reaction of azathioprine. The mechanism of reaction is unknown but may involve high 6-MMPN blood level, due to unusual thiopurine metabolism (switched metabolism). Early therapeutic drug monitoring with measurement of 6-TGN and 6-MMPN blood levels may help physicians to identify patients at risk of similar duct injury.

Identifiants

pubmed: 37218386
doi: 10.1111/bcp.15797
doi:

Substances chimiques

Azathioprine MRK240IY2L
Immunosuppressive Agents 0
Thioguanine FTK8U1GZNX
Thionucleotides 0
Methyltransferases EC 2.1.1.-
Mercaptopurine E7WED276I5
Guanine Nucleotides 0

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

2625-2630

Informations de copyright

© 2023 The Authors. British Journal of Clinical Pharmacology published by John Wiley & Sons Ltd on behalf of British Pharmacological Society.

Références

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Auteurs

Laurent Chouchana (L)

Centre Régional de Pharmacovigilance, Service de Pharmacologie périnatale, pédiatrique et adulte, Hôpital Cochin, AP-HP.Centre, Université Paris Cité, Paris, France.

Benoit Terris (B)

Service de Pathologie, Hôpital Cochin, AP-HP.Centre, Université Paris Cité, Paris, France.

Philippe Sogni (P)

Service d'hépatologie, Hôpital Cochin, AP-HP.Centre, Université Paris Cité, Paris, France.

Jean-Marc Treluyer (JM)

Centre Régional de Pharmacovigilance, Service de Pharmacologie périnatale, pédiatrique et adulte, Hôpital Cochin, AP-HP.Centre, Université Paris Cité, Paris, France.

Nathalie Costedoat-Chalumeau (N)

Centre de référence maladies auto-immunes et systémiques rares d'Ile de France, Département de Médecine interne, Hôpital Cochin, AP-HP.Centre, Université Paris Cité, Paris, France.
INSERM U 1153, Center for Epidemiology and Statistics Sorbonne Paris Cité (CRESS), Paris, France.

Marie-Anne Loriot (MA)

Unité de Pharmacogénétique, Service de Biochimie, Hôpital européen Georges Pompidou, AP-HP.Centre, Université Paris Cité, Paris, France.
INSERM U1147, Médecine Personnalisée Pharmacogénomique et Optimisation Thérapeutique (MEPPOT), Paris, France.

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