Anticonvulsant potential of Grewia tiliaefolia in pentylenetetrazole induced epilepsy: insights from in vivo and in silico studies.


Journal

Metabolic brain disease
ISSN: 1573-7365
Titre abrégé: Metab Brain Dis
Pays: United States
ID NLM: 8610370

Informations de publication

Date de publication:
10 2023
Historique:
received: 16 06 2022
accepted: 07 06 2023
medline: 18 9 2023
pubmed: 12 7 2023
entrez: 12 7 2023
Statut: ppublish

Résumé

Epilepsy, a chronic neurological condition, impacts millions of individuals globally and remains a significant contributor to both illness and mortality. Available antiepileptic drugs have serious side effects which warrants to explore different medicinal plants used for the management of epilepsy reported in Traditional Indian Medicinal System (TIMS). Therefore, we explored the antiepileptic potential of the Grewia tiliaefolia (Tiliaeceae) which is known for its neuroprotective properties. Aerial parts of G. tiliaefolia were subjected to extraction with increasing order of polarity viz. hexane, chloroform and methanol. Antioxidant potential of hexane, chloroform and methanol extracts of G. tiliaefolia was evaluated by 2,2-diphenyl-1-picryl-hydrazyl-hydrate (DPPH) assay, total antioxidant capacity (TAC) assay, reducing power assay (RPA) and DNA nicking assay. Additionally, quantitative antioxidant assays were also conducted to quantify total phenolic (TPC) and total flavonoid content (TFC). As revealed by in vitro assays, methanol extract was found to contain more phenolic content. Hence, the methanol extract was further explored for its anticonvulsant potential in pentylenetetrazole (PTZ) induced acute seizures in mice. The methanol extract (400 mg/kg) significantly increased the latency to occurrence of myoclonic jerks and generalized tonic clonic seizures (GTCS). Additionally, it also reduced duration and seizure severity score associated with GTCS. The Grewia tiliaefolia methanol extract was further screened by Ultra High-Performance Liquid Chromatography (UHPLC) for presence of polyphenolic compounds, among which gallic acid and kaempferol were present in higher amount and were further analysed by in silico study to predict their possible binding sites and type of interactions these compounds show with gamma amino butyric acid (GABA) receptor and glutamate α amino-3- hydroxyl-5-methyl-4-isoxazolepropionic acid (Glu-AMPA) receptor. It was revealed that gallic acid and kaempferol had shown agonistic interaction for GABA receptor and antagonistic interaction for Glu-AMPA receptor. We concluded that G. tiliaefolia showed anticonvulsant potential possibly because of gallic acid and kaempferol possibly mediated through GABA and Glu-AMPA receptor.

Identifiants

pubmed: 37436587
doi: 10.1007/s11011-023-01252-0
pii: 10.1007/s11011-023-01252-0
doi:

Substances chimiques

Anticonvulsants 0
Pentylenetetrazole WM5Z385K7T
n-hexane 2DDG612ED8
Hexanes 0
Kaempferols 0
Antioxidants 0
Methanol Y4S76JWI15
Chloroform 7V31YC746X
Receptors, AMPA 0
Plant Extracts 0
Gallic Acid 632XD903SP
gamma-Aminobutyric Acid 56-12-2

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2355-2367

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Ankita Rajput (A)

Department of Botanical and Environmental Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Palvi Sharma (P)

Department of Botanical and Environmental Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Nitish Kumar (N)

Department of Pharmaceutical Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Hasandeep Singh (H)

Department of Pharmaceutical Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Tanveer Singh (T)

Department of Neuroscience and Experimental Therapeutics, College of Medicine, Texas A&M University Health Science Center, Bryan, TX, 77807, USA.

Sharabjit Singh (S)

Department of Botanical and Environmental Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Preet Mohinder Singh Bedi (PM)

Department of Pharmaceutical Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Balbir Singh (B)

Department of Pharmaceutical Sciences, Guru Nanak Dev University, Amritsar, Punjab, India.

Saroj Arora (S)

Department of Botanical and Environmental Sciences, Guru Nanak Dev University, Amritsar, Punjab, India. dr.sarojarora@gmail.com.

Sarabjit Kaur (S)

Department of Pharmaceutical Sciences, Guru Nanak Dev University, Amritsar, Punjab, India. sarabjit.pharma@gndu.ac.in.

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