Circulating hormones and risk of gastric cancer by subsite in three cohort studies.


Journal

Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association
ISSN: 1436-3305
Titre abrégé: Gastric Cancer
Pays: Japan
ID NLM: 100886238

Informations de publication

Date de publication:
Nov 2023
Historique:
received: 20 03 2023
accepted: 20 06 2023
medline: 13 11 2023
pubmed: 17 7 2023
entrez: 16 7 2023
Statut: ppublish

Résumé

Obesity has been positively associated with gastric cancer. Excess fat impacts hormones, which have been implicated in carcinogenesis. We investigated obesity-related hormones and cardia gastric cancer (CGC) and non-cardia gastric cancer (NCGC) risk. Nested case-control studies were conducted within the European Prospective Investigation into Cancer and Nutrition (EPIC) cohort (61 CGCs, and 172 NCGCs and matched controls) and the Alpha-Tocopherol, Beta-Carotene Cancer Prevention (ATBC) study (100 CGCs and 65 NCGCs and matched controls); serum hormones were measured. In UK-Biobank (n = 458,713), we included 137 CGCs and 92 NCGCs. Sex-specific analyses were conducted. For EPIC and ATBC, odds ratios (ORs), and for UK-Biobank hazard ratios (HRs), were estimated using conditional logistic regression and Cox regression, respectively. Insulin-like growth-factor-1 was positively associated with CGC and NCGC in EPIC men (OR Some obesity-related hormones influence CGC and NCGC risk.

Sections du résumé

BACKGROUND BACKGROUND
Obesity has been positively associated with gastric cancer. Excess fat impacts hormones, which have been implicated in carcinogenesis. We investigated obesity-related hormones and cardia gastric cancer (CGC) and non-cardia gastric cancer (NCGC) risk.
METHODS METHODS
Nested case-control studies were conducted within the European Prospective Investigation into Cancer and Nutrition (EPIC) cohort (61 CGCs, and 172 NCGCs and matched controls) and the Alpha-Tocopherol, Beta-Carotene Cancer Prevention (ATBC) study (100 CGCs and 65 NCGCs and matched controls); serum hormones were measured. In UK-Biobank (n = 458,713), we included 137 CGCs and 92 NCGCs. Sex-specific analyses were conducted. For EPIC and ATBC, odds ratios (ORs), and for UK-Biobank hazard ratios (HRs), were estimated using conditional logistic regression and Cox regression, respectively.
RESULTS RESULTS
Insulin-like growth-factor-1 was positively associated with CGC and NCGC in EPIC men (OR
CONCLUSIONS CONCLUSIONS
Some obesity-related hormones influence CGC and NCGC risk.

Identifiants

pubmed: 37455285
doi: 10.1007/s10120-023-01414-0
pii: 10.1007/s10120-023-01414-0
pmc: PMC10640529
doi:

Substances chimiques

Hormones 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

969-987

Subventions

Organisme : Cancer Research UK
ID : 20189
Pays : United Kingdom
Organisme : Cancer Research UK
ID : 25004
Pays : United Kingdom
Organisme : Cancer Research UK
ID : EDDPMA-NOV22/100009
Pays : United Kingdom
Organisme : Cancer Research UK
ID : PRCRPG-NOV22/100001
Pays : United Kingdom

Informations de copyright

© 2023. The Author(s).

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Auteurs

Harinakshi Sanikini (H)

Department of Epidemiology and Biostatistics, School of Public Health, Imperial College London, St. Mary's Campus, Norfolk Place, London, W2 1PG, UK. h.sanikini@imperial.ac.uk.

Carine Biessy (C)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Sabina Rinaldi (S)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Anne-Sophie Navionis (AS)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Audrey Gicquiau (A)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Pekka Keski-Rahkonen (P)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Agneta Kiss (A)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Stephanie J Weinstein (SJ)

Metabolic Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Demetrius Albanes (D)

Metabolic Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Antonio Agudo (A)

Unit of Nutrition and Cancer, Catalan Institute of Oncology-ICO, L'Hospitalet de Llobregat, Spain.
Nutrition and Cancer Group, Epidemiology, Public Health, Cancer Prevention and Palliative Care Program, Bellvitge Biomedical Research Institute-IDIBELL, L'Hospitalet de Llobregat, Spain.

Mazda Jenab (M)

Section of Nutrition and Metabolism, International Agency for Research on Cancer, Lyon, France.

Elio Riboli (E)

Department of Epidemiology and Biostatistics, School of Public Health, Imperial College London, St. Mary's Campus, Norfolk Place, London, W2 1PG, UK.

Marc J Gunter (MJ)

Department of Epidemiology and Biostatistics, School of Public Health, Imperial College London, St. Mary's Campus, Norfolk Place, London, W2 1PG, UK.

Gwen Murphy (G)

Cancer Screening and Prevention Research Group, Department of Surgery and Cancer, Imperial College London, London, UK.

Amanda J Cross (AJ)

Department of Epidemiology and Biostatistics, School of Public Health, Imperial College London, St. Mary's Campus, Norfolk Place, London, W2 1PG, UK.
Cancer Screening and Prevention Research Group, Department of Surgery and Cancer, Imperial College London, London, UK.

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Classifications MeSH