BAFFR expression in circulating T follicular helper (CD4


Journal

Lupus
ISSN: 1477-0962
Titre abrégé: Lupus
Pays: England
ID NLM: 9204265

Informations de publication

Date de publication:
Aug 2023
Historique:
medline: 8 8 2023
pubmed: 18 7 2023
entrez: 17 7 2023
Statut: ppublish

Résumé

Circulating T follicular helper (cTfh) and T peripheral helper (Tph) subpopulations are shown to be higher in systemic lupus erythematosus (SLE) patients and have been involved in promoting extrafollicular B cell responses. However, a possible association with the B cell activating factor (BAFF), a cytokine mainly related to B cell responses and disease activity in SLE, has not been investigated. Therefore, this study aimed to evaluate the association of cTfh and Tph subpopulations with the BAFF system expression and clinical activity in SLE patients. This study included 43 SLE patients and 12 healthy subjects (HS). The identification of cTfh (CD4 Compared with HS, SLE patients showed a significantly increased percentage of cTfh and Tph cells, higher in patients with clearly active disease. SLE patients had markedly higher IL-21-producing cTfh and Tph cells than HS. Both subpopulations were positively correlated with the disease activity in SLE patients. Tph cells were negatively correlated with CD19 This study emphasizes the importance of cTfh and Tph cells in SLE pathogenesis. Besides the importance of IL-21, our results suggest that BAFFR could play a role in cTfh and Tph subpopulations in the autoimmunity context.

Sections du résumé

BACKGROUND BACKGROUND
Circulating T follicular helper (cTfh) and T peripheral helper (Tph) subpopulations are shown to be higher in systemic lupus erythematosus (SLE) patients and have been involved in promoting extrafollicular B cell responses. However, a possible association with the B cell activating factor (BAFF), a cytokine mainly related to B cell responses and disease activity in SLE, has not been investigated. Therefore, this study aimed to evaluate the association of cTfh and Tph subpopulations with the BAFF system expression and clinical activity in SLE patients.
METHODS METHODS
This study included 43 SLE patients and 12 healthy subjects (HS). The identification of cTfh (CD4
RESULTS RESULTS
Compared with HS, SLE patients showed a significantly increased percentage of cTfh and Tph cells, higher in patients with clearly active disease. SLE patients had markedly higher IL-21-producing cTfh and Tph cells than HS. Both subpopulations were positively correlated with the disease activity in SLE patients. Tph cells were negatively correlated with CD19
CONCLUSIONS CONCLUSIONS
This study emphasizes the importance of cTfh and Tph cells in SLE pathogenesis. Besides the importance of IL-21, our results suggest that BAFFR could play a role in cTfh and Tph subpopulations in the autoimmunity context.

Identifiants

pubmed: 37460408
doi: 10.1177/09612033231189804
doi:

Substances chimiques

B-Cell Maturation Antigen 0
CXCR5 protein, human 0
Programmed Cell Death 1 Receptor 0
Receptors, CXCR5 0
TNFRSF13C protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1093-1104

Auteurs

Nefertari Sagrero-Fabela (N)

Doctorado en Ciencias Biomédicas (DCB), Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

Pablo C Ortíz-Lazareno (PC)

División de Inmunología, Centro de Investigación Biomédica de Occidente (CIBO), Instituto Mexicano del Seguro Social (IMSS), Guadalajara, México.

Diana C Salazar-Camarena (DC)

Grupo de Inmunología Molecular, Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

Alvaro Cruz (A)

Instituto de Investigación en Ciencias Biomédicas (IICB), Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

Sergio Cerpa-Cruz (S)

Departamento de Reumatología, Hospital Civil de Guadalajara Fray Antonio Alcalde, Guadalajara, México.

José F Muñoz-Valle (JF)

Instituto de Investigación en Ciencias Biomédicas (IICB), Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

Miguel Marín-Rosales (M)

Grupo de Inmunología Molecular, Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.
Hospital General de Occidente, Secretaría de Salud Jalisco, Guadalajara, México.

Jhonatan A Alvarez-Gómez (JA)

Doctorado en Ciencias en Biología Molecular en Medicina (DCBMM), Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

Claudia A Palafox-Sánchez (CA)

Grupo de Inmunología Molecular, Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.
Instituto de Investigación en Ciencias Biomédicas (IICB), Centro Universitario de Ciencias de la Salud, Universidad de Guadalajara, Guadalajara, México.

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Classifications MeSH