Plasma angiopoietin-2 and its association with heart failure in patients with atrial fibrillation.


Journal

Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology
ISSN: 1532-2092
Titre abrégé: Europace
Pays: England
ID NLM: 100883649

Informations de publication

Date de publication:
04 07 2023
Historique:
received: 04 05 2023
accepted: 24 05 2023
medline: 23 10 2023
pubmed: 18 7 2023
entrez: 18 7 2023
Statut: ppublish

Résumé

Several biomarkers are associated with clinical outcomes in patients with atrial fibrillation (AF), but a causal relationship has not been established. This study aimed to evaluate angiopoietin-2, a novel candidate biomarker of endothelial inflammation and vascular remodelling, in patients with AF. Angiopoietin-2 was measured in plasma obtained from patients with AF treated with aspirin monotherapy (exploration cohort, n = 2987) or with oral anticoagulation (validation cohort, n = 13 079). Regression models were built to assess the associations between angiopoietin-2, clinical characteristics, and outcomes. In both cohorts, plasma angiopoietin-2 was independently associated with AF on the baseline electrocardiogram and persistent/permanent AF, age, history of heart failure, female sex, tobacco use/smoking, body mass index, renal dysfunction, diabetes, and N-terminal pro-B-type natriuretic peptide (NT-proBNP). Angiopoietin-2 was independently associated with subsequent hospitalization for heart failure after adjusting for age, creatinine, and clinical characteristics in the exploration cohort [c-index 0.79, 95% confidence interval (CI) 0.75-0.82; third vs. first quartile, hazard ratio (HR) 1.74, 95% CI 1.26-2.41] and in the validation cohort (c-index 0.76, 95% CI 0.74-0.78; HR 1.58, 95% CI 1.37-1.82). In both cohorts, the association persisted when also adjusting for NT-proBNP (P ≤ 0.001). In full multivariable models also adjusted for NT-proBNP, angiopoietin-2 did not show statistically significant associations with ischaemic stroke, cardiovascular and all-cause death, or major bleeding that were consistent across the two cohorts. In patients with AF, plasma levels of angiopoietin-2 were independently associated with subsequent hospitalization for heart failure and provided incremental prognostic value to clinical risk factors and NT-proBNP.

Identifiants

pubmed: 37461214
pii: 7225558
doi: 10.1093/europace/euad200
pmc: PMC10359110
pii:
doi:

Substances chimiques

Angiopoietin-2 0
Biomarkers 0
Peptide Fragments 0
Natriuretic Peptide, Brain 114471-18-0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Sanofi-Aventis and Bristol-Myers Squibb
Organisme : Bristol-Myers Squibb and Pfize
Organisme : Roche Diagnostics
Organisme : Bristol-Myers Squibb and Pfizer
Organisme : Swedish Society for Medical Research
ID : S17-0133
Organisme : Swedish Heart-Lung Foundation
ID : 20200722
Organisme : Uppsala University Hospital

Informations de copyright

© The Author(s) 2023. Published by Oxford University Press on behalf of the European Society of Cardiology.

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Auteurs

Alexander P Benz (AP)

Population Health Research Institute, McMaster University, 237 Barton St. E., Hamilton, Ontario L8L 2X2, Canada.
Department of Cardiology, University Medical Center Mainz, Johannes Gutenberg-University, Langenbeckstr. 1, Mainz 55131, Germany.

Ziad Hijazi (Z)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.
Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden.

Johan Lindbäck (J)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.

Stuart J Connolly (SJ)

Population Health Research Institute, McMaster University, 237 Barton St. E., Hamilton, Ontario L8L 2X2, Canada.

John W Eikelboom (JW)

Population Health Research Institute, McMaster University, 237 Barton St. E., Hamilton, Ontario L8L 2X2, Canada.

Peter Kastner (P)

Roche Diagnostics GmbH, Penzberg, Germany.

André Ziegler (A)

Roche Diagnostics GmbH, Penzberg, Germany.

John H Alexander (JH)

Duke Clinical Research Institute, Duke University, Durham, NC, USA.

Christopher B Granger (CB)

Duke Clinical Research Institute, Duke University, Durham, NC, USA.

Renato D Lopes (RD)

Duke Clinical Research Institute, Duke University, Durham, NC, USA.

Jonas Oldgren (J)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.
Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden.

Agneta Siegbahn (A)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.
Department of Medical Sciences, Clinical Chemistry, Uppsala University, Uppsala, Sweden.

Lars Wallentin (L)

Uppsala Clinical Research Center, Uppsala University, Uppsala, Sweden.
Department of Medical Sciences, Cardiology, Uppsala University, Uppsala, Sweden.

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