Preservation of Circumferential and Radial Left Ventricular Function as a Mitigating Mechanism for Impaired Longitudinal Strain in Early Cardiac Amyloidosis.


Journal

Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography
ISSN: 1097-6795
Titre abrégé: J Am Soc Echocardiogr
Pays: United States
ID NLM: 8801388

Informations de publication

Date de publication:
Dec 2023
Historique:
received: 05 05 2023
revised: 05 07 2023
accepted: 01 08 2023
medline: 5 12 2023
pubmed: 14 8 2023
entrez: 13 8 2023
Statut: ppublish

Résumé

In patients with cardiac amyloidosis (CA), left ventricular ejection fraction (LVEF) is frequently preserved, despite commonly reduced global longitudinal strain (GLS). We hypothesized that nonlongitudinal contraction may initially serve as a mitigating mechanism to maintain cardiac output and studied the relationship between global circumferential (GCS) and radial (GRS) strain with LVEF and extracellular volume (ECV), a marker of amyloid burden. Patients with CA who underwent cardiac magnetic resonance (CMR; n = 140, 70.7 ± 11.5 years, 66% male) or echocardiography (n = 67, 71 ± 13 years, 66% male) and normal controls (CMR, n = 20; echocardiography, n = 45) were retrospectively identified, and GCS, GLS, and GRS were quantified using feature-tracking CMR or speckle-tracking echocardiography and compared between CA patients with preserved and reduced LVEF (CA While echocardiography-derived GLS was impaired in both CA Among CA patients with preserved LVEF, preserved GCS and GRS, despite near-universally impaired GLS, may be explained by an initial predominantly subendocardial involvement, where mostly longitudinal fibers are located. If confirmed in future studies, these findings may facilitate identification of patients with early stages of CA, when treatments may be most effective.

Sections du résumé

BACKGROUND BACKGROUND
In patients with cardiac amyloidosis (CA), left ventricular ejection fraction (LVEF) is frequently preserved, despite commonly reduced global longitudinal strain (GLS). We hypothesized that nonlongitudinal contraction may initially serve as a mitigating mechanism to maintain cardiac output and studied the relationship between global circumferential (GCS) and radial (GRS) strain with LVEF and extracellular volume (ECV), a marker of amyloid burden.
METHODS METHODS
Patients with CA who underwent cardiac magnetic resonance (CMR; n = 140, 70.7 ± 11.5 years, 66% male) or echocardiography (n = 67, 71 ± 13 years, 66% male) and normal controls (CMR, n = 20; echocardiography, n = 45) were retrospectively identified, and GCS, GLS, and GRS were quantified using feature-tracking CMR or speckle-tracking echocardiography and compared between CA patients with preserved and reduced LVEF (CA
RESULTS RESULTS
While echocardiography-derived GLS was impaired in both CA
CONCLUSIONS CONCLUSIONS
Among CA patients with preserved LVEF, preserved GCS and GRS, despite near-universally impaired GLS, may be explained by an initial predominantly subendocardial involvement, where mostly longitudinal fibers are located. If confirmed in future studies, these findings may facilitate identification of patients with early stages of CA, when treatments may be most effective.

Identifiants

pubmed: 37574149
pii: S0894-7317(23)00418-2
doi: 10.1016/j.echo.2023.08.005
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1290-1301

Informations de copyright

Copyright © 2023 American Society of Echocardiography. Published by Elsevier Inc. All rights reserved.

Auteurs

Jeremy A Slivnick (JA)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Cristiane Singulane (C)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Deyu Sun (D)

David Geffen School of Medicine, University of California Los Angeles, Los Angeles, California.

Derek Eshun (D)

Division of Cardiology, Northwestern Medicine, Chicago, Illinois.

Akhil Narang (A)

Division of Cardiology, Northwestern Medicine, Chicago, Illinois.

Steven Mazzone (S)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Karima Addetia (K)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Amit R Patel (AR)

Division of Cardiology, University of Virginia, Charlottesville, Virginia.

Karolina M Zareba (KM)

Division of Cardiology, The Ohio State University Wexner Medical Center, Columbus, Ohio.

Suzanne Smart (S)

Division of Cardiology, The Ohio State University Wexner Medical Center, Columbus, Ohio.

Jung Woo Kwon (JW)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Aliya Husain (A)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Brittany Cody (B)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Seth Scheetz (S)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Federico M Asch (FM)

Division of Cardiology, Medstar Health, Washington, District of Columbia.

Akash Goyal (A)

Division of Cardiology, The Ohio State University Wexner Medical Center, Columbus, Ohio.

Nitasha Sarswat (N)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Victor Mor-Avi (V)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois.

Roberto M Lang (RM)

Division of Cardiology, University of Chicago Medical Center, Chicago, Illinois. Electronic address: rlang@bsd.uchicago.edu.

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Classifications MeSH