Developmental dynamics of homoarginine, ADMA and SDMA plasma levels from birth to adolescence.


Journal

Amino acids
ISSN: 1438-2199
Titre abrégé: Amino Acids
Pays: Austria
ID NLM: 9200312

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 26 09 2022
accepted: 15 08 2023
medline: 4 12 2023
pubmed: 31 8 2023
entrez: 30 8 2023
Statut: ppublish

Résumé

Guanidino compounds such as dimethylarginines (SDMA, ADMA) and L-homoarginine ((L-)hArg) can interfere with bioavailability and function of the main NO-donor L-arginine (L-Arg). High ADMA and SDMA but low L-hArg concentrations have been associated with cardio- and cerebrovascular events and mortality in adults. The role of guanidino compounds in paediatric patients remains less clear. We, therefore, compared guanidino compound levels in plasma samples of 57 individuals with chronic kidney disease (CKD) and 141 individuals without CKD from the age of 0 to 17 years, including patients with different comorbidities by correlation and regression analyses. We found highest hArg, SDMA and ADMA concentrations in neonates (Kruskal-Wallis, p < 0.001 for all). From the age of 1 year on, hArg levels increased, whereas SDMA und ADMA levels further decreased in children. SDMA and ADMA are higher in children with CKD independent of GFR (mean factor 1.92 and 1.38, respectively, p < 0.001 for both), and SDMA is strongly correlated with creatinine concentration in children with CKD (Spearman's rho 0.74, p < 0.001). We provide guanidino compound levels in a large sample covering all paediatric age groups for the first time. Our data can be used to assess the role of guanidino compounds such as hArg in disease states, i.e. cerebro- and cardiovascular disorders in childhood and adolescence.

Identifiants

pubmed: 37648945
doi: 10.1007/s00726-023-03318-w
pii: 10.1007/s00726-023-03318-w
pmc: PMC10689515
doi:

Substances chimiques

Homoarginine 156-86-5
Arginine 94ZLA3W45F

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1381-1388

Subventions

Organisme : Else Kröner-Fresenius-Stiftung
ID : 2018-EKES.04

Informations de copyright

© 2023. The Author(s).

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Auteurs

Florence Baach (F)

Department of Paediatrics, University Medical Center Hamburg-Eppendorf, Martinistraße 52, 20246, Hamburg, Germany.

Boglarka Meyer (B)

Department of Paediatrics, University Medical Center Hamburg-Eppendorf, Martinistraße 52, 20246, Hamburg, Germany.

Jun Oh (J)

Department of Paediatrics, University Medical Center Hamburg-Eppendorf, Martinistraße 52, 20246, Hamburg, Germany.

Susanne Lezius (S)

Department of Medical Biometry and Epidemiology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Rainer Böger (R)

Institute of Clinical Pharmacology and Toxicology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Edzard Schwedhelm (E)

Institute of Clinical Pharmacology and Toxicology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
German Centre for Cardiovascular Research (DZHK E.V.), Partner Site Hamburg/Kiel/Lübeck, Hamburg, Germany.

Chi-Un Choe (CU)

Department of Neurology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
Department of Neurology, Klinikum Itzehoe, Robert-Koch-Strasse 2, 25524, Itzehoe, Germany.

Axel Neu (A)

Department of Paediatrics, University Medical Center Hamburg-Eppendorf, Martinistraße 52, 20246, Hamburg, Germany. axel@enp.org.
VAMED Klinik Geesthacht, Johannes-Ritter-Strasse 100, 21502, Geesthacht, Germany. axel@enp.org.

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