Microbe-induced gene silencing boosts crop protection against soil-borne fungal pathogens.


Journal

Nature plants
ISSN: 2055-0278
Titre abrégé: Nat Plants
Pays: England
ID NLM: 101651677

Informations de publication

Date de publication:
09 2023
Historique:
received: 12 12 2022
accepted: 02 08 2023
medline: 19 9 2023
pubmed: 1 9 2023
entrez: 31 8 2023
Statut: ppublish

Résumé

Small RNA (sRNA)-mediated trans-kingdom RNA interference (RNAi) between host and pathogen has been demonstrated and utilized. However, interspecies RNAi in rhizospheric microorganisms remains elusive. In this study, we developed a microbe-induced gene silencing (MIGS) technology by using a rhizospheric beneficial fungus, Trichoderma harzianum, to exploit an RNAi engineering microbe and two soil-borne pathogenic fungi, Verticillium dahliae and Fusarium oxysporum, as RNAi recipients. We first detected the feasibility of MIGS in inducing GFP silencing in V. dahliae. Then by targeting a fungal essential gene, we further demonstrated the effectiveness of MIGS in inhibiting fungal growth and protecting dicotyledon cotton and monocotyledon rice plants against V. dahliae and F. oxysporum. We also showed steerable MIGS specificity based on a selected target sequence. Our data verify interspecies RNAi in rhizospheric fungi and the potential application of MIGS in crop protection. In addition, the in situ propagation of a rhizospheric beneficial microbe would be optimal in ensuring the stability and sustainability of sRNAs, avoiding the use of nanomaterials to carry chemically synthetic sRNAs. Our finding reveals that exploiting MIGS-based biofungicides would offer straightforward design and implementation, without the need of host genetic modification, in crop protection against phytopathogens.

Identifiants

pubmed: 37653339
doi: 10.1038/s41477-023-01507-9
pii: 10.1038/s41477-023-01507-9
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1409-1418

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Nature Limited.

Références

Chen, X. & Rechavi, O. Plant and animal small RNA communications between cells and organisms. Nat. Rev. Mol. Cell Biol. 23, 185–203 (2022).
Lopez-Gomollon, S. & Baulcombe, D. C. Roles of RNA silencing in viral and non-viral plant immunity and in the crosstalk between disease resistance systems. Nat. Rev. Mol. Cell Biol. 23, 645–662 (2022).
pubmed: 35710830 doi: 10.1038/s41580-022-00496-5
Guo, Z., Li, Y. & Ding, S. W. Small RNA-based antimicrobial immunity. Nat. Rev. Immunol. 19, 31–44 (2019).
pubmed: 30301972 doi: 10.1038/s41577-018-0071-x
Zhao, J. H. & Guo, H. S. RNA silencing: from discovery and elucidation to application and perspectives. J. Integr. Plant Biol. 64, 476–498 (2022).
pubmed: 34964265 doi: 10.1111/jipb.13213
Brown, K. M. et al. Expanding RNAi therapeutics to extrahepatic tissues with lipophilic conjugates. Nat. Biotechnol. 40, 1500–1508 (2022).
Hu, B. et al. Therapeutic siRNA: state of the art. Signal Transduct. Target. Ther. 5, 101 (2020).
pubmed: 32561705 pmcid: 7305320 doi: 10.1038/s41392-020-0207-x
Setten, R. L., Rossi, J. J. & Han, S. P. The current state and future directions of RNAi-based therapeutics. Nat. Rev. Drug Discov. 18, 421–446 (2019).
pubmed: 30846871 doi: 10.1038/s41573-019-0017-4
Zhang, T. et al. Cotton plants export microRNAs to inhibit virulence gene expression in a fungal pathogen. Nat. Plants 2, 16153 (2016).
pubmed: 27668926 doi: 10.1038/nplants.2016.153
Weiberg, A. et al. Fungal small RNAs suppress plant immunity by hijacking host RNA interference pathways. Science 342, 118–123 (2013).
pubmed: 24092744 pmcid: 4096153 doi: 10.1126/science.1239705
Silvestri, A. et al. In silico analysis of fungal small RNA accumulation reveals putative plant mRNA targets in the symbiosis between an arbuscular mycorrhizal fungus and its host plant. BMC Genomics 20, 169 (2019).
pubmed: 30832582 pmcid: 6399891 doi: 10.1186/s12864-019-5561-0
Šečić, E. et al. A novel plant-fungal association reveals fundamental sRNA and gene expression reprogramming at the onset of symbiosis. BMC Biol. 19, 171 (2021).
pubmed: 34429124 pmcid: 8385953 doi: 10.1186/s12915-021-01104-2
Dalakouras, A. et al. A beneficial fungal root endophyte triggers systemic RNA silencing and DNA methylation of a host reporter gene. RNA Biol. 20, 20–30 (2023).
pubmed: 36573793 doi: 10.1080/15476286.2022.2159158
Wong-Bajracharya, J. et al. The ectomycorrhizal fungus Pisolithus microcarpus encodes a microRNA involved in cross-kingdom gene silencing during symbiosis. Proc. Natl Acad. Sci. USA 119, e2103527119(2022).
Tsikou, D. et al. Systemic control of legume susceptibility to rhizobial infection by a mobile microRNA. Science 362, 233–236 (2018).
pubmed: 30166437 doi: 10.1126/science.aat6907
Ren, B., Wang, X., Duan, J. & Ma, J. Rhizobial tRNA-derived small RNAs are signal molecules regulating plant nodulation. Science 365, 919–922 (2019).
pubmed: 31346137 doi: 10.1126/science.aav8907
Zhang, T. et al. Host-induced gene silencing of the target gene in fungal cells confers effective resistance to the cotton wilt disease pathogen Verticillium dahliae. Mol. Plant 9, 939–942 (2016).
pubmed: 26925819 doi: 10.1016/j.molp.2016.02.008
Nowara, D. et al. HIGS: host-induced gene silencing in the obligate biotrophic fungal pathogen Blumeria graminis. Plant Cell 22, 3130–3141 (2010).
pubmed: 20884801 pmcid: 2965548 doi: 10.1105/tpc.110.077040
Qiao, Y. et al. Small RNAs in plant immunity and virulence of filamentous pathogens. Annu. Rev. Phytopathol. 59, 265–288 (2021).
pubmed: 34077241 doi: 10.1146/annurev-phyto-121520-023514
Zhao, J. H., Zhang, T., Liu, Q. Y. & Guo, H. S. Trans-kingdom RNAs and their fates in recipient cells: advances, utilization, and perspectives. Plant Commun. 2, 100167 (2021).
pubmed: 33898979 pmcid: 8060725 doi: 10.1016/j.xplc.2021.100167
Koch, A. et al. An RNAi-based control of Fusarium graminearum infections through spraying of long dsRNAs involves a plant passage and is controlled by the fungal silencing machinery. PLoS Pathog. 12, e1005901 (2016).
pubmed: 27737019 pmcid: 5063301 doi: 10.1371/journal.ppat.1005901
Mitter, N. et al. Clay nanosheets for topical delivery of RNAi for sustained protection against plant viruses. Nat. Plants 3, 16207 (2017).
pubmed: 28067898 doi: 10.1038/nplants.2016.207
Hochella, M. F. Jr et al. Natural, incidental, and engineered nanomaterials and their impacts on the Earth system. Science 363, eaau8299 (2019).
Zhao, P., Zhao, Y. L., Jin, Y., Zhang, T. & Guo, H. S. Colonization process of Arabidopsis thaliana roots by a green fluorescent protein-tagged isolate of Verticillium dahliae. Protein Cell 5, 94–98 (2014).
pubmed: 24481631 pmcid: 3956967 doi: 10.1007/s13238-013-0009-9
Xu, Y. et al. Genetical and O-glycoproteomic analyses reveal the roles of three protein O-mannosyltransferases in phytopathogen Fusarium oxysporum f.sp. cucumerinum. Fungal Genet. Biol. 134, 103285 (2020).
pubmed: 31648060 doi: 10.1016/j.fgb.2019.103285
Jin, Y. et al. A fungal milRNA mediates epigenetic repression of a virulence gene in Verticillium dahliae. Phil. Trans. R. Soc. Lond. B 374, 20180309 (2019).
doi: 10.1098/rstb.2018.0309
Chang, S. S., Zhang, Z. & Liu, Y. RNA interference pathways in fungi: mechanisms and functions. Annu. Rev. Microbiol. 66, 305–323 (2012).
pubmed: 22746336 pmcid: 4617789 doi: 10.1146/annurev-micro-092611-150138
Kwon, S., Tisserant, C., Tulinski, M., Weiberg, A. & Feldbrugge, M. Inside-out: from endosomes to extracellular vesicles in fungal RNA transport. Fungal Biol. Rev. 34, 89–99 (2020).
doi: 10.1016/j.fbr.2020.01.001
Zand Karimi, H. et al. Arabidopsis apoplastic fluid contains sRNA- and circular RNA-protein complexes that are located outside extracellular vesicles. Plant Cell 34, 1863–1881 (2022).
pubmed: 35171271 pmcid: 9048913 doi: 10.1093/plcell/koac043
Djonovic, S., Pozo, M. J. & Kenerley, C. M. Tvbgn3, a beta-1,6-glucanase from the biocontrol fungus Trichoderma virens, is involved in mycoparasitism and control of Pythium ultimum. Appl. Environ. Microbiol. 72, 7661–7670 (2006).
pubmed: 16997978 pmcid: 1694269 doi: 10.1128/AEM.01607-06
Viterbo, A., Ramot, O., Chemin, L. & Chet, I. Significance of lytic enzymes from Trichoderma spp. in the biocontrol of fungal plant pathogens. Antonie van Leeuwenhoek 81, 549–556 (2002).
pubmed: 12448750 doi: 10.1023/A:1020553421740
Zhang, T., Zhao, J.-H., Fang, Y.-Y., Guo, H.-S. & Jin, Y. Exploring the effectiveness and durability of trans-kingdom silencing of fungal genes in the vascular pathogen Verticillium dahliae. Int. J. Mol. Sci. 23, 2742 (2022).
pubmed: 35269884 pmcid: 8910871 doi: 10.3390/ijms23052742
Shimizu, T., Yaegashi, H., Ito, T. & Kanematsu, S. Systemic RNA interference is not triggered by locally-induced RNA interference in a plant pathogenic fungus, Rosellinia necatrix. Fungal Genet. Biol. 76, 27–35 (2015).
pubmed: 25677378 doi: 10.1016/j.fgb.2015.02.001
Hammond, T. M. & Keller, N. P. RNA silencing in Aspergillus nidulans is independent of RNA-dependent RNA polymerases. Genetics 169, 607–617 (2005).
pubmed: 15545645 pmcid: 1449118 doi: 10.1534/genetics.104.035964
Song, X. S. et al. Secondary amplification of siRNA machinery limits the application of spray-induced gene silencing. Mol. Plant Pathol. 19, 2543–2560 (2018).
pubmed: 30027625 pmcid: 6638038 doi: 10.1111/mpp.12728
Middleton, H., Yergeau, E., Monard, C., Combier, J. P. & El Amrani, A. Rhizospheric plant–microbe interactions: miRNAs as a key mediator. Trends Plant Sci. 26, 132–141 (2021).
pubmed: 33036916 doi: 10.1016/j.tplants.2020.09.005
Liu, H. & Brettell, L. E. Plant defense by VOC-induced microbial priming. Trends Plant Sci. 24, 187–189 (2019).
pubmed: 30738790 doi: 10.1016/j.tplants.2019.01.008
Liu, H., Macdonald, C. A., Cook, J., Anderson, I. C. & Singh, B. K. An ecological loop: host microbiomes across multitrophic interactions. Trends Ecol. Evol. 34, 1118–1130 (2019).
pubmed: 31422890 doi: 10.1016/j.tree.2019.07.011
Harman, G. E., Howell, C. R., Viterbo, A., Chet, I. & Lorito, M. Trichoderma species—opportunistic, avirulent plant symbionts. Nat. Rev. Microbiol. 2, 43–56 (2004).
pubmed: 15035008 doi: 10.1038/nrmicro797
Benitez, T., Rincon, A. M., Limon, M. C. & Codon, A. C. Biocontrol mechanisms of Trichoderma strains. Int. Microbiol. 7, 249–260 (2004).
pubmed: 15666245
Sarrocco, S. et al. Histopathological studies of sclerotia of phytopathogenic fungi parasitized by a GFP transformed Trichoderma virens antagonistic strain. Mycol. Res. 110, 179–187 (2006).
pubmed: 16388938 doi: 10.1016/j.mycres.2005.08.005
Steyaert, J. M., Ridgway, H. J., Elad, Y. & Stewart, A. Genetic basis of mycoparasitism: a mechanism of biological control by species of Trichoderma. N. Z. J. Crop Hortic. Sci. 31, 281–291 (2003).
doi: 10.1080/01140671.2003.9514263
Wang, S., Xing, H., Hua, C., Guo, H. S. & Zhang, J. An improved single-step cloning strategy simplifies the Agrobacterium tumefaciens-mediated transformation (ATMT)-based gene-disruption method for Verticillium dahliae. Phytopathology 106, 645–652 (2016).
Gao, F. et al. A glutamic acid-rich protein identified in Verticillium dahliae from an insertional mutagenesis affects microsclerotial formation and pathogenicity. PLoS ONE 5, e15319 (2010).
pubmed: 21151869 pmcid: 2998422 doi: 10.1371/journal.pone.0015319
Zhao, Y. L., Zhang, T. & Guo, H. S. Penetration assays, fungal recovery and pathogenicity assays for Verticillium dahliae. Bio Protoc. 7, e2133 (2017).
pubmed: 34458454 pmcid: 8376503 doi: 10.21769/BioProtoc.2133
Bleackley, M. R. et al. Extracellular vesicles from the cotton pathogen Fusarium oxysporum f. sp. vasinfectum induce a phytotoxic response in plants. Front. Plant Sci. 10, 1610 (2019).
pubmed: 31998330 doi: 10.3389/fpls.2019.01610
Chan, W. et al. Induction of amphotericin B resistance in susceptible Candida auris by extracellular vesicles. Emerg. Microbes Infect. 11, 1900–1909 (2022).
pubmed: 35786393 pmcid: 9341352 doi: 10.1080/22221751.2022.2098058
Smith, T., Heger, A. & Sudbery, I. UMI-tools: modeling sequencing errors in Unique Molecular Identifiers to improve quantification accuracy. Genome Res. 27, 491–499 (2017).
pubmed: 28100584 pmcid: 5340976 doi: 10.1101/gr.209601.116

Auteurs

Han-Guang Wen (HG)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China.

Jian-Hua Zhao (JH)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China. zhao_jian_hua@hotmail.com.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China. zhao_jian_hua@hotmail.com.

Bo-Sen Zhang (BS)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China.

Feng Gao (F)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China.

Xue-Ming Wu (XM)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China.

Yong-Sheng Yan (YS)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.

Jie Zhang (J)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China.

Hui-Shan Guo (HS)

State Key Laboratory of Plant Genomics, Institute of Microbiology, the Chinese Academy of Sciences, Beijing, China. guohs@im.ac.cn.
CAS Center for Excellence in Biotic Interactions, University of the Chinese Academy of Sciences, Beijing, China. guohs@im.ac.cn.

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