Transcription factor networks link B-lymphocyte development and malignant transformation in leukemia.
B-ALL
B-lymphocytes
transcription factors
Journal
Genes & development
ISSN: 1549-5477
Titre abrégé: Genes Dev
Pays: United States
ID NLM: 8711660
Informations de publication
Date de publication:
01 08 2023
01 08 2023
Historique:
pmc-release:
01
02
2024
medline:
22
9
2023
pubmed:
7
9
2023
entrez:
6
9
2023
Statut:
ppublish
Résumé
Rapid advances in genomics have opened unprecedented possibilities to explore the mutational landscapes in malignant diseases, such as B-cell acute lymphoblastic leukemia (B-ALL). This disease is manifested as a severe defect in the production of normal blood cells due to the uncontrolled expansion of transformed B-lymphocyte progenitors in the bone marrow. Even though classical genetics identified translocations of transcription factor-coding genes in B-ALL, the extent of the targeting of regulatory networks in malignant transformation was not evident until the emergence of large-scale genomic analyses. There is now evidence that many B-ALL cases present with mutations in genes that encode transcription factors with critical roles in normal B-lymphocyte development. These include
Identifiants
pubmed: 37673459
pii: gad.349879.122
doi: 10.1101/gad.349879.122
pmc: PMC10546977
doi:
Substances chimiques
Transcription Factors
0
Types de publication
Journal Article
Review
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
703-723Informations de copyright
© 2023 Sigvardsson; Published by Cold Spring Harbor Laboratory Press.
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