Incidence, Predictors, and Prognostic Impact of Bleeding Events After TAVR According to VARC-3 Criteria.


Journal

JACC. Cardiovascular interventions
ISSN: 1876-7605
Titre abrégé: JACC Cardiovasc Interv
Pays: United States
ID NLM: 101467004

Informations de publication

Date de publication:
25 09 2023
Historique:
received: 01 05 2023
revised: 05 07 2023
accepted: 11 07 2023
medline: 29 9 2023
pubmed: 7 9 2023
entrez: 7 9 2023
Statut: ppublish

Résumé

The updated Valve Academic Research Consortium (VARC) definition for bleeding events after transcatheter aortic valve replacement (TAVR) lacks of clinical validation. The aim of this study was to determine the incidence, predictors, and clinical impact of bleeding events following TAVR as defined by recent VARC-3 criteria. A total of 2,384 consecutive patients with severe symptomatic aortic stenosis undergoing TAVR were included. Early (at index hospitalization) and late (after hospital discharge) bleeding complications were defined according to VARC-3 criteria as type 1, 2, 3, or 4. Baseline, procedural, and follow-up (24 [IQR: 12-43] months) data were prospectively collected. Bleeding events occurred in 761 patients (31.9%): types 1, 2, 3, and 4 in 169 (22.2%), 399 (52.4%), 149 (19.6%), and 44 (5.8%) patients, respectively. The primary vascular access site and gastrointestinal locations were the most common bleeding sources among early and late bleeding events, respectively. Female sex, thoracotomy access, larger (14-F) sheath use, and dual antiplatelet therapy determined an increased risk of early bleeding events (P < 0.02 for all). The use of the radial artery for secondary access was associated with a significant risk reduction of early bleeding (P < 0.001). Type 2 and type 3 events were associated with an increased mortality risk at 30-day (HR: 2.94 [95% CI: 1.43-6.03; P = 0.003] and HR: 4.91 [95% CI: 2.19-11.03; P < 0.001], respectively) and 1-year (HR: 1.86 [95% CI: 1.28-2.69; P = 0.001] and HR: 2.28 [95% CI: 1.41-3.66; P = 0.001], respectively) follow-up. A similar prognostic pattern was observed when applying VARC-2 criteria but with a much lower global incidence of early bleeding events (19% vs 27%; P < 0.001). Bleeding events after TAVR were associated with poorer short- and long-term survival. The magnitude of this correlation was proportional to bleeding severity defined according to VARC-3 criteria. Further studies on bleeding prevention following TAVR are warranted to improve procedural safety and patient prognosis.

Sections du résumé

BACKGROUND
The updated Valve Academic Research Consortium (VARC) definition for bleeding events after transcatheter aortic valve replacement (TAVR) lacks of clinical validation.
OBJECTIVES
The aim of this study was to determine the incidence, predictors, and clinical impact of bleeding events following TAVR as defined by recent VARC-3 criteria.
METHODS
A total of 2,384 consecutive patients with severe symptomatic aortic stenosis undergoing TAVR were included. Early (at index hospitalization) and late (after hospital discharge) bleeding complications were defined according to VARC-3 criteria as type 1, 2, 3, or 4. Baseline, procedural, and follow-up (24 [IQR: 12-43] months) data were prospectively collected.
RESULTS
Bleeding events occurred in 761 patients (31.9%): types 1, 2, 3, and 4 in 169 (22.2%), 399 (52.4%), 149 (19.6%), and 44 (5.8%) patients, respectively. The primary vascular access site and gastrointestinal locations were the most common bleeding sources among early and late bleeding events, respectively. Female sex, thoracotomy access, larger (14-F) sheath use, and dual antiplatelet therapy determined an increased risk of early bleeding events (P < 0.02 for all). The use of the radial artery for secondary access was associated with a significant risk reduction of early bleeding (P < 0.001). Type 2 and type 3 events were associated with an increased mortality risk at 30-day (HR: 2.94 [95% CI: 1.43-6.03; P = 0.003] and HR: 4.91 [95% CI: 2.19-11.03; P < 0.001], respectively) and 1-year (HR: 1.86 [95% CI: 1.28-2.69; P = 0.001] and HR: 2.28 [95% CI: 1.41-3.66; P = 0.001], respectively) follow-up. A similar prognostic pattern was observed when applying VARC-2 criteria but with a much lower global incidence of early bleeding events (19% vs 27%; P < 0.001).
CONCLUSIONS
Bleeding events after TAVR were associated with poorer short- and long-term survival. The magnitude of this correlation was proportional to bleeding severity defined according to VARC-3 criteria. Further studies on bleeding prevention following TAVR are warranted to improve procedural safety and patient prognosis.

Identifiants

pubmed: 37676226
pii: S1936-8798(23)01045-2
doi: 10.1016/j.jcin.2023.07.005
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2262-2274

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2023 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Funding Support and Author Disclosures Dr Real is supported by the Fundacion Interhospitalaria Para la Investigación Cardiovascular (Madrid, Spain). Dr Nuche is a recipient of a grant from Fundación Alfonso Martín Escudero (Madrid, Spain). Dr Rodés-Cabau has received institutional research grants and speaker and consultant fees from Edwards Lifesciences and Medtronic, and holds the Research Chair “Fondation Famille Jacques Larivière” for the Development of Structural Heart Disease Interventions (Laval University, Quebec City, Canada). All other authors have reported that they have no relationships relevant to the contents of this paper to disclose.

Auteurs

Marisa Avvedimento (M)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Carlos Real (C)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada; Department of Cardiology, Hospital Clinico San Carlos, Madrid, Spain.

Jorge Nuche (J)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Julio Farjat-Pasos (J)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Attilio Galhardo (A)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Kim-Hoang Trinh (KH)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Mathieu Robichaud (M)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Robert Delarochellière (R)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Jean-Michel Paradis (JM)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Anthony Poulin (A)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Eric Dumont (E)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Dimitris Kalavrouziotis (D)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Siamak Mohammadi (S)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Mélanie Côté (M)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada.

Josep Rodés-Cabau (J)

Quebec Heart & Lung Institute, Laval University, Quebec City, Quebec, Canada; Clínic Barcelona, Barcelona, Spain. Electronic address: josep.rodes@criucpq.ulaval.ca.

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