Serum neurofilament and glial fibrillary acidic protein in idiopathic and seropositive transverse myelitis.

Glial fibrillary acidic protein Multiple sclerosis Myelin oligodendrocyte glycoprotein antibody-associated disease Neurofilament Neuromyelitis optica spectrum disorder Transverse myelitis

Journal

Multiple sclerosis and related disorders
ISSN: 2211-0356
Titre abrégé: Mult Scler Relat Disord
Pays: Netherlands
ID NLM: 101580247

Informations de publication

Date de publication:
Nov 2023
Historique:
received: 03 03 2023
revised: 22 06 2023
accepted: 20 08 2023
medline: 10 11 2023
pubmed: 10 9 2023
entrez: 9 9 2023
Statut: ppublish

Résumé

Serum levels of neurofilament light chain (NfL) and glial fibrillary acidic protein (GFAP) reflect the disease activity and disability in central nervous system (CNS) demyelinating diseases. However, the clinical significance of NfL and GFAP in idiopathic transverse myelitis (iTM), an inflammatory spinal cord disease with unknown underlying causes, remains unclear. This study aimed to investigate NfL and GFAP levels in iTM and their association with the clinical parameters compared with those in TM with disease-specific antibodies such as anti-aquaporin 4 or myelin oligodendrocyte glycoprotein antibodies (sTM). We collected serum and clinical data of 365 patients with CNS inflammatory diseases from 12 hospitals. The serum NfL and GFAP levels were measured in patients with iTM (n = 37) and sTM (n = 39) using ultrasensitive single-molecule array assays. Regression analysis was performed to investigate the associations between serum levels of NfL and GFAP and the clinical parameters such as higher EDSS scores (EDSS ≥ 4.0). Mean NfL levels were not significantly different between iTM (50.29 pg/ml) and sTM (63.18 pg/ml) (p = 0.824). GFAP levels were significantly lower in iTM (112.34 pg/ml) than in sTM (3814.20 pg/ml) (p = 0.006). NfL levels correlated with expanded disability status scale (EDSS) scores in sTM (p = 0.001) but not in iTM (p = 0.824). Disease duration also correlated with higher EDSS scores in sTM (p = 0.017). NfL levels and disease duration correlated with EDSS scores in sTM, and GFAP levels could be a promising biomarker to differentiate iTM from sTM.

Sections du résumé

BACKGROUND BACKGROUND
Serum levels of neurofilament light chain (NfL) and glial fibrillary acidic protein (GFAP) reflect the disease activity and disability in central nervous system (CNS) demyelinating diseases. However, the clinical significance of NfL and GFAP in idiopathic transverse myelitis (iTM), an inflammatory spinal cord disease with unknown underlying causes, remains unclear. This study aimed to investigate NfL and GFAP levels in iTM and their association with the clinical parameters compared with those in TM with disease-specific antibodies such as anti-aquaporin 4 or myelin oligodendrocyte glycoprotein antibodies (sTM).
METHODS METHODS
We collected serum and clinical data of 365 patients with CNS inflammatory diseases from 12 hospitals. The serum NfL and GFAP levels were measured in patients with iTM (n = 37) and sTM (n = 39) using ultrasensitive single-molecule array assays. Regression analysis was performed to investigate the associations between serum levels of NfL and GFAP and the clinical parameters such as higher EDSS scores (EDSS ≥ 4.0).
RESULTS RESULTS
Mean NfL levels were not significantly different between iTM (50.29 pg/ml) and sTM (63.18 pg/ml) (p = 0.824). GFAP levels were significantly lower in iTM (112.34 pg/ml) than in sTM (3814.20 pg/ml) (p = 0.006). NfL levels correlated with expanded disability status scale (EDSS) scores in sTM (p = 0.001) but not in iTM (p = 0.824). Disease duration also correlated with higher EDSS scores in sTM (p = 0.017).
CONCLUSION CONCLUSIONS
NfL levels and disease duration correlated with EDSS scores in sTM, and GFAP levels could be a promising biomarker to differentiate iTM from sTM.

Identifiants

pubmed: 37688927
pii: S2211-0348(23)00458-3
doi: 10.1016/j.msard.2023.104957
pii:
doi:

Substances chimiques

Glial Fibrillary Acidic Protein 0
Aquaporin 4 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

104957

Informations de copyright

Copyright © 2023 Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors have no conflicts of interest to disclose.

Auteurs

Hye Lim Lee (HL)

Department of Neurology, Korea University, College of Medicine, Seoul, Korea.

Jin Myoung Seok (JM)

Department of Neurology, Soonchunhyang University Hospital Cheonan, Soonchunhyang University College of Medicine, Cheonan, Korea.

Yeon Hak Chung (YH)

Department of Neurology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea; Neuroscience Center, Samsung Medical Center, Seoul, Korea.

Ju-Hong Min (JH)

Department of Neurology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea; Neuroscience Center, Samsung Medical Center, Seoul, Korea.

Seol-Hee Baek (SH)

Department of Neurology, Korea University, College of Medicine, Seoul, Korea.

Sung Min Kim (SM)

Department of Neurology, Seoul National University, College of Medicine, Seoul, Korea.

Eunhee Sohn (E)

Department of Neurology, Chungnam National University, College of Medicine, Daejeon, Korea.

Juhyeon Kim (J)

Department of Neurology, Gyeongsang Institute of Health Science, Gyeongsang National University, College of Medicine, Jinju, Korea.

Sa-Yoon Kang (SY)

Department of Neurology, Jeju National University, College of Medicine, Jeju, Korea.

Yoon-Ho Hong (YH)

Department of Neurology, Seoul National University, College of Medicine, Seoul, Korea.

Ha Young Shin (HY)

Department of Neurology, Yonsei University College of Medicine, Seoul, Korea.

Joong-Yang Cho (JY)

Department of Neurology, Ilsan Paik Hospital, Inje University College of Medicine, Goyang, Korea.

Jeeyoung Oh (J)

Department of Neurology, Konkuk University School of Medicine, Konkuk University Medical Center, Seoul, Korea.

Sang-Soo Lee (SS)

Department of Neurology, Chungbuk National University, College of Medicine, Chungbuk, Korea.

Sunyoung Kim (S)

Department of Neurology, University of Ulsan College of Medicine, Ulsan University Hospital, Ulsan, Korea.

Su-Hyun Kim (SH)

Department of Neurology, Research Institute and Hospital of National Cancer Center, Goyang, Korea.

Ho Jin Kim (HJ)

Department of Neurology, Research Institute and Hospital of National Cancer Center, Goyang, Korea.

Byung-Jo Kim (BJ)

Department of Neurology, Korea University, College of Medicine, Seoul, Korea. Electronic address: bjkim@skku.edu.

Byoung Joon Kim (BJ)

Department of Neurology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea; Neuroscience Center, Samsung Medical Center, Seoul, Korea. Electronic address: nukbj@korea.ac.kr.

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Classifications MeSH