Detection of Mpox Virus Using Microbial Cell-free DNA: the Potential of Pathogen-Agnostic Sequencing for Rapid Identification of Emerging Pathogens.

HIV metagenomic sequencing microbial cell-free DNA monkeypox mpox outbreak surveillance

Journal

The Journal of infectious diseases
ISSN: 1537-6613
Titre abrégé: J Infect Dis
Pays: United States
ID NLM: 0413675

Informations de publication

Date de publication:
12 Oct 2023
Historique:
received: 31 07 2023
revised: 09 10 2023
accepted: 10 10 2023
medline: 12 10 2023
pubmed: 12 10 2023
entrez: 12 10 2023
Statut: aheadofprint

Résumé

The 2022 global outbreak of mpox virus (MPXV) highlighted challenges with PCR detection as divergent strains emerged and atypical presentations limited the applicability of swab sampling. Recommended testing in the United States requires a swab of lesions, which arise late in infection and may be unrecognized. We present MPXV detections using plasma microbial cell-free DNA (mcfDNA) sequencing. Fifteen plasma samples from 12 case-patients were characterized through mcfDNA sequencing. Assay performance was confirmed through in-silico inclusivity and exclusivity assessments. MPXV isolates were genotyped using mcfDNA, and phylodynamic information imputed using publicly available sequences. MPXV mcfDNA was detected in 12 case-patients. Mpox was not suspected in 5, with one having documented resolution of mpox >6 months previously. Six had moderate to severe mpox, supported by high MPXV mcfDNA concentration; 4 died. In 7 case-patients, mcfDNA sequencing detected co-infections. Genotyping by mcfDNA sequencing identified 22 MPXV mutations at 10 genomic loci in 9 case-patients. Consistent with variation observed in the 2022 outbreak, 21/22 variants were G > A/C > T. Phylogenetic analyses imputed isolates to sublineages arising at different time points and from different geographic locations. We demonstrate the potential of plasma mcfDNA sequencing to detect, quantify, and, for acute infections with high sequencing coverage, subtype MPXV using a single non-invasive test. Sequencing plasma mcfDNA may augment existing mpox testing in vulnerable patient populations or in patients with atypical symptoms or unrecognized mpox. Strain type information may supplement disease surveillance and facilitate tracking emerging pathogens.

Sections du résumé

BACKGROUND BACKGROUND
The 2022 global outbreak of mpox virus (MPXV) highlighted challenges with PCR detection as divergent strains emerged and atypical presentations limited the applicability of swab sampling. Recommended testing in the United States requires a swab of lesions, which arise late in infection and may be unrecognized. We present MPXV detections using plasma microbial cell-free DNA (mcfDNA) sequencing.
METHODS METHODS
Fifteen plasma samples from 12 case-patients were characterized through mcfDNA sequencing. Assay performance was confirmed through in-silico inclusivity and exclusivity assessments. MPXV isolates were genotyped using mcfDNA, and phylodynamic information imputed using publicly available sequences.
RESULTS RESULTS
MPXV mcfDNA was detected in 12 case-patients. Mpox was not suspected in 5, with one having documented resolution of mpox >6 months previously. Six had moderate to severe mpox, supported by high MPXV mcfDNA concentration; 4 died. In 7 case-patients, mcfDNA sequencing detected co-infections. Genotyping by mcfDNA sequencing identified 22 MPXV mutations at 10 genomic loci in 9 case-patients. Consistent with variation observed in the 2022 outbreak, 21/22 variants were G > A/C > T. Phylogenetic analyses imputed isolates to sublineages arising at different time points and from different geographic locations.
CONCLUSIONS CONCLUSIONS
We demonstrate the potential of plasma mcfDNA sequencing to detect, quantify, and, for acute infections with high sequencing coverage, subtype MPXV using a single non-invasive test. Sequencing plasma mcfDNA may augment existing mpox testing in vulnerable patient populations or in patients with atypical symptoms or unrecognized mpox. Strain type information may supplement disease surveillance and facilitate tracking emerging pathogens.

Identifiants

pubmed: 37824825
pii: 7310835
doi: 10.1093/infdis/jiad452
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© The Author(s) 2023. Published by Oxford University Press on behalf of Infectious Diseases Society of America.

Auteurs

Sarah Y Park (SY)

Karius Inc; Redwood City, CA.

Martin S Lindner (MS)

Karius Inc; Redwood City, CA.

Kevin Brick (K)

Karius Inc; Redwood City, CA.

Nicholas Noll (N)

Karius Inc; Redwood City, CA.

Rachid Ounit (R)

Karius Inc; Redwood City, CA.

Luis J Noa (LJ)

AdventHealth Orlando; Orlando, FL.

Rabeeya Sabzwari (R)

Edward Hines, Jr. VA Hospital; Hines, IL.

Ronald Trible (R)

Georgia Infectious Diseases, PC; Atlanta, GA.

Jason C Sniffen (JC)

AdventHealth Orlando; Orlando, FL.

Prerana Roth (P)

Prisma Health - Upstate; Greenville, SC.

Amir Khan (A)

Carle Foundation Hospital; Urbana, IL.

Anamaria Rodriguez (A)

Orlando Health; Orlando, FL.

Syeda Sahra (S)

Oklahoma University Medical Center; Oklahoma City, OK.

Michael J Davis (MJ)

University of Minnesota; Minneapolis, MN.

Inderjeet S Brar (IS)

Baptist Memorial Health Care; Memphis, TN.

Gayathri Balasundaram (G)

Karius Inc; Redwood City, CA.

Frederick S Nolte (FS)

Karius Inc; Redwood City, CA.

Timothy A Blauwkamp (TA)

Karius Inc; Redwood City, CA.

Bradley A Perkins (BA)

Karius Inc; Redwood City, CA.

Sivan Bercovici (S)

Karius Inc; Redwood City, CA.

Classifications MeSH