Ultradian hydrocortisone replacement alters neuronal processing, emotional ambiguity, affect and fatigue in adrenal insufficiency: The PULSES trial.


Journal

Journal of internal medicine
ISSN: 1365-2796
Titre abrégé: J Intern Med
Pays: England
ID NLM: 8904841

Informations de publication

Date de publication:
Jan 2024
Historique:
medline: 20 12 2023
pubmed: 20 10 2023
entrez: 19 10 2023
Statut: ppublish

Résumé

Primary adrenal insufficiency (PAI) mortality and morbidity remain unacceptably high, possibly arising as glucocorticoid replacement does not replicate natural physiology. A pulsatile subcutaneous pump can closely replicate cortisol's circadian and ultradian rhythm. To assess the effect of pump therapy on quality of life, mood, functional neuroimaging, behavioural/cognitive responses, sleep and metabolism. A 6-week randomised, crossover, double-blinded and placebo-controlled feasibility study of usual dose hydrocortisone in PAI administered as either pulsed subcutaneous or standard care in Bristol, United Kingdom (ISRCTN67193733). Participants were stratified by adrenal insufficiency type. All participants who received study drugs are included in the analysis. The primary outcome, the facial expression recognition task (FERT), occurred at week 6. Between December 2014 and 2017, 22 participants were recruited - 20 completed both arms, and 21 were analysed. The pump was well-tolerated. No change was seen in the FERT primary outcome; however, there were subjective improvements in fatigue and mood. Additionally, functional magnetic resonance imaging revealed differential neural processing to emotional cues and visual stimulation. Region of interest analysis identified the left amygdala and insula, key glucocorticoid-sensitive regions involved in emotional ambiguity. FERT post hoc analysis confirmed this response. There were four serious adverse events (AE): three intercurrent illnesses requiring hospitalisation (1/3, 33.3% pump) and a planned procedure (1/1, 100% pump). There was a small number of expected AEs: infusion site bruising/itching (3/5, 60% pump), intercurrent illness requiring extra (3/7, 42% pump) and no extra (4/6, 66% pump) steroid. These findings support the administration of hormone therapy that mimics physiology.

Sections du résumé

BACKGROUND BACKGROUND
Primary adrenal insufficiency (PAI) mortality and morbidity remain unacceptably high, possibly arising as glucocorticoid replacement does not replicate natural physiology. A pulsatile subcutaneous pump can closely replicate cortisol's circadian and ultradian rhythm.
OBJECTIVES OBJECTIVE
To assess the effect of pump therapy on quality of life, mood, functional neuroimaging, behavioural/cognitive responses, sleep and metabolism.
METHODS METHODS
A 6-week randomised, crossover, double-blinded and placebo-controlled feasibility study of usual dose hydrocortisone in PAI administered as either pulsed subcutaneous or standard care in Bristol, United Kingdom (ISRCTN67193733). Participants were stratified by adrenal insufficiency type. All participants who received study drugs are included in the analysis. The primary outcome, the facial expression recognition task (FERT), occurred at week 6.
RESULTS RESULTS
Between December 2014 and 2017, 22 participants were recruited - 20 completed both arms, and 21 were analysed. The pump was well-tolerated. No change was seen in the FERT primary outcome; however, there were subjective improvements in fatigue and mood. Additionally, functional magnetic resonance imaging revealed differential neural processing to emotional cues and visual stimulation. Region of interest analysis identified the left amygdala and insula, key glucocorticoid-sensitive regions involved in emotional ambiguity. FERT post hoc analysis confirmed this response. There were four serious adverse events (AE): three intercurrent illnesses requiring hospitalisation (1/3, 33.3% pump) and a planned procedure (1/1, 100% pump). There was a small number of expected AEs: infusion site bruising/itching (3/5, 60% pump), intercurrent illness requiring extra (3/7, 42% pump) and no extra (4/6, 66% pump) steroid.
CONCLUSIONS CONCLUSIONS
These findings support the administration of hormone therapy that mimics physiology.

Identifiants

pubmed: 37857352
doi: 10.1111/joim.13721
doi:

Substances chimiques

Glucocorticoids 0
Hydrocortisone WI4X0X7BPJ

Types de publication

Randomized Controlled Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

51-67

Subventions

Organisme : Medical Research Council
ID : MR/J012548/1
Pays : United Kingdom

Informations de copyright

© 2023 The Authors. Journal of Internal Medicine published by John Wiley & Sons Ltd on behalf of Association for Publication of The Journal of Internal Medicine.

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Auteurs

Georgina Russell (G)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.
North Bristol NHS Trust, Bristol, UK.

Konstantinos Kalafatakis (K)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
Department of Informatics and Telecommunications, Human-Computer Interaction Laboratory, University of Ioannina, Arta, Greece.
Clinical Research and Imaging Centre, University of Bristol, Bristol, UK.
Faculty of Medicine and Dentistry (Malta Campus), Queen Mary University of London, Victoria, Malta.

Claire Durant (C)

Department of Brain Sciences, Faculty of Medicine, Imperial College London, London, UK.

Nicola Marchant (N)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.

Jamini Thakrar (J)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
Clinical Research and Imaging Centre, University of Bristol, Bristol, UK.

Russell Thirard (R)

Bristol Trials Centre, Bristol Medical School, University of Bristol, Bristol, UK.

Jade King (J)

University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.
North Bristol NHS Trust, Bristol, UK.

Jane Bowles (J)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.

Thomas Upton (T)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.

Ngoc Jade Thai (NJ)

Clinical Research and Imaging Centre, University of Bristol, Bristol, UK.
Neurosciences and Mental Health, Liverpool Health Partners, Liverpool, UK.

Jonathan C W Brooks (JCW)

School of Psychology, University of East Anglia, Norwich, UK.

Aileen Wilson (A)

Clinical Research and Imaging Centre, University of Bristol, Bristol, UK.

Kirsty Phillips (K)

University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.

Stuart Ferguson (S)

School of Medicine, University of Tasmania, Hobart, Tasmania, Australia.

Meryem Grabski (M)

Division of Psychology and Language Sciences, UCL, London, UK.

Chris A Rogers (CA)

Bristol Trials Centre, Bristol Medical School, University of Bristol, Bristol, UK.

Theodoros Lampros (T)

Department of Informatics and Telecommunications, Human-Computer Interaction Laboratory, University of Ioannina, Arta, Greece.

Sue Wilson (S)

Department of Brain Sciences, Faculty of Medicine, Imperial College London, London, UK.

Catherine Harmer (C)

Department of Psychiatry, Oxford University and Oxford Health NHS Foundation Trust, Oxford, UK.

Marcus Munafo (M)

MRC Integrative Epidemiology Unit, School of Psychological Science, University of Bristol, Bristol, UK.

Stafford L Lightman (SL)

Laboratories of Integrative Neuroscience and Endocrinology, Bristol Medical School, University of Bristol, Bristol, UK.
University Hospital Bristol and Weston NHS Foundation Trust, Bristol, UK.

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