Analysis of random mutations in Salmonella Gallinarum dihydropteroate synthase conferring sulfonamide resistance.

Dihydropteroate synthase Mutant analysis Protein modeling Salmonella enterica serovar Gallinarum Sulfonamide

Journal

Archives of microbiology
ISSN: 1432-072X
Titre abrégé: Arch Microbiol
Pays: Germany
ID NLM: 0410427

Informations de publication

Date de publication:
31 Oct 2023
Historique:
received: 29 08 2023
accepted: 29 09 2023
revised: 26 09 2023
medline: 2 11 2023
pubmed: 31 10 2023
entrez: 31 10 2023
Statut: epublish

Résumé

In bacteria and primitive eukaryotes, sulfonamide antibiotics block the folate pathway by inhibiting dihydropteroate synthase (FolP) that combines para-aminobenzoic acid (pABA) and dihydropterin pyrophosphate (DHPP) to form dihydropteroic acid (DHP), a precursor for tetrahydrofolate synthesis. However, the emergence of resistant strains has severely compromised the use of pABA mimetics as sulfonamide drugs. Salmonella enterica serovar Gallinarum (S. Gallinarum) is a significant source of antibiotic-resistant infections in poultry. Here, a sulfonamide-resistant FolP mutant library of S. Gallinarum was generated through random mutagenesis. Among resistant strains, substitution of amino acid Arginine 171 with Proline (R171P) in the FolP protein conferred the highest resistance against sulfonamide. Substitution of Phe28 with Leu or Ile (F28L/I) led to modest sulfonamide resistance. Structural modeling indicates that R171P and Phenylalanine 28 with leucine or isoleucine (F28L/I) substitution mutations are located far from the substrate-binding site and cause insignificant conformational changes in the FolP protein. Rather, in silico studies suggest that the mutations altered the stability of the protein, potentially resulting in sulfonamide resistance. Identification of specific mutations in FolP that confer resistance to sulfonamide would contribute to our understanding of the molecular mechanisms of antibiotic resistance.

Identifiants

pubmed: 37906281
doi: 10.1007/s00203-023-03696-5
pii: 10.1007/s00203-023-03696-5
doi:

Substances chimiques

Dihydropteroate Synthase EC 2.5.1.15
4-Aminobenzoic Acid TL2TJE8QTX
Anti-Bacterial Agents 0
Sulfanilamide 21240MF57M
Sulfonamides 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

363

Subventions

Organisme : Starting growth Technological R&D Program (TIPS Program) funded by the Ministry of SMEs and Startups (MSS, Korea) in 2021
ID : No. S3130592

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer-Verlag GmbH Germany, part of Springer Nature.

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Auteurs

Taner Duysak (T)

Department of Microbiology, Chonnam National University Medical School, Gwangju, 61468, Korea.
Basic Medical Research Building, Odysseus Bio, Chonnam National University Medical College, 322 Seoyang-ro, Hwasun, 58128, Jeonnam, Korea.

Jae-Ho Jeong (JH)

Department of Microbiology, Chonnam National University Medical School, Gwangju, 61468, Korea.

Kwangsoo Kim (K)

Basic Medical Research Building, Odysseus Bio, Chonnam National University Medical College, 322 Seoyang-ro, Hwasun, 58128, Jeonnam, Korea.

Jeong-Sun Kim (JS)

Department of Chemistry, Chonnam National University, Gwangju, 61186, Korea. jsunkim@chonnam.ac.kr.

Hyon E Choy (HE)

Department of Microbiology, Chonnam National University Medical School, Gwangju, 61468, Korea. hyonchoy@jnu.ac.kr.
Basic Medical Research Building, Odysseus Bio, Chonnam National University Medical College, 322 Seoyang-ro, Hwasun, 58128, Jeonnam, Korea. hyonchoy@jnu.ac.kr.

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