Metabolic features of treatment-refractory major depressive disorder with suicidal ideation.


Journal

Translational psychiatry
ISSN: 2158-3188
Titre abrégé: Transl Psychiatry
Pays: United States
ID NLM: 101562664

Informations de publication

Date de publication:
15 Dec 2023
Historique:
received: 06 09 2022
accepted: 29 11 2023
revised: 18 10 2023
medline: 15 12 2023
pubmed: 15 12 2023
entrez: 14 12 2023
Statut: epublish

Résumé

Peripheral blood metabolomics was used to gain chemical insight into the biology of treatment-refractory Major Depressive Disorder with suicidal ideation, and to identify individualized differences for personalized care. The study cohort consisted of 99 patients with treatment-refractory major depressive disorder and suicidal ideation (trMDD-SI n = 52 females and 47 males) and 94 age- and sex-matched healthy controls (n = 48 females and 46 males). The median age was 29 years (IQR 22-42). Targeted, broad-spectrum metabolomics measured 448 metabolites. Fibroblast growth factor 21 (FGF21) and growth differentiation factor 15 (GDF15) were measured as biomarkers of mitochondrial dysfunction. The diagnostic accuracy of plasma metabolomics was over 90% (95%CI: 0.80-1.0) by area under the receiver operator characteristic (AUROC) curve analysis. Over 55% of the metabolic impact in males and 75% in females came from abnormalities in lipids. Modified purines and pyrimidines from tRNA, rRNA, and mRNA turnover were increased in the trMDD-SI group. FGF21 was increased in both males and females. Increased lactate, glutamate, and saccharopine, and decreased cystine provided evidence of reductive stress. Seventy-five percent of the metabolomic abnormalities found were individualized. Personalized deficiencies in CoQ10, flavin adenine dinucleotide (FAD), citrulline, lutein, carnitine, or folate were found. Pathways regulated by mitochondrial function dominated the metabolic signature. Peripheral blood metabolomics identified mitochondrial dysfunction and reductive stress as common denominators in suicidal ideation associated with treatment-refractory major depressive disorder. Individualized metabolic differences were found that may help with personalized management.

Identifiants

pubmed: 38097555
doi: 10.1038/s41398-023-02696-9
pii: 10.1038/s41398-023-02696-9
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

393

Subventions

Organisme : NCRR NIH HHS
ID : UL1 RR024153
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR000005
Pays : United States

Informations de copyright

© 2023. The Author(s).

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Auteurs

Lisa A Pan (LA)

Department of Psychiatry, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA. lisapanmd@newhopemolecular.com.
School of Public Health, Department of Human Genetics, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA. lisapanmd@newhopemolecular.com.
Panomics Mental Health Initiative, Pittsburgh, PA, USA. lisapanmd@newhopemolecular.com.
New Hope Molecular, LLC, Pittsburgh, PA, USA. lisapanmd@newhopemolecular.com.
New Hope Molecular, LLC, 750 Washington Rd, Suite 19, Pittsburgh, PA, 15228, USA. lisapanmd@newhopemolecular.com.

Jane C Naviaux (JC)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA.
Department of Neurosciences, University of California, San Diego School of Medicine, San Diego, CA, USA.

Lin Wang (L)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA.
Department of Medicine, University of California, San Diego School of Medicine, San Diego, CA, USA.

Kefeng Li (K)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA.
Department of Medicine, University of California, San Diego School of Medicine, San Diego, CA, USA.

Jonathan M Monk (JM)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA.
Department of Medicine, University of California, San Diego School of Medicine, San Diego, CA, USA.

Sai Sachin Lingampelly (SS)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA.
Department of Medicine, University of California, San Diego School of Medicine, San Diego, CA, USA.

Anna Maria Segreti (AM)

Department of Psychiatry, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.

Kaitlyn Bloom (K)

Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.

Jerry Vockley (J)

Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.

Mark A Tarnopolsky (MA)

Department of Pediatrics, McMaster University, Hamilton, Ontario, Canada.

David N Finegold (DN)

School of Public Health, Department of Human Genetics, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.
Panomics Mental Health Initiative, Pittsburgh, PA, USA.
New Hope Molecular, LLC, Pittsburgh, PA, USA.

David G Peters (DG)

Department of Psychiatry, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.
School of Public Health, Department of Human Genetics, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.
Panomics Mental Health Initiative, Pittsburgh, PA, USA.
Department of Obstetrics, Gynecology & Reproductive Sciences, University of Pittsburgh, Pittsburgh, PA, USA.

Robert K Naviaux (RK)

The Mitochondrial and Metabolic Disease Center, University of California, San Diego School of Medicine, San Diego, CA, USA. rnaviaux@health.ucsd.edu.
Department of Medicine, University of California, San Diego School of Medicine, San Diego, CA, USA. rnaviaux@health.ucsd.edu.
Department of Pediatrics, University of California, San Diego School of Medicine, San Diego, CA, USA. rnaviaux@health.ucsd.edu.
Department of Pathology, University of California, San Diego School of Medicine, San Diego, CA, USA. rnaviaux@health.ucsd.edu.

Classifications MeSH