Cochlear aqueduct revisited: A histological study using human fetuses.

Cavitation Cochlear aqueduct Cochlear vein Human fetuses Perilymphatic space Scala tympani Subarachnoid space

Journal

Annals of anatomy = Anatomischer Anzeiger : official organ of the Anatomische Gesellschaft
ISSN: 1618-0402
Titre abrégé: Ann Anat
Pays: Germany
ID NLM: 100963897

Informations de publication

Date de publication:
Apr 2024
Historique:
received: 23 04 2023
revised: 04 12 2023
accepted: 20 02 2024
medline: 18 3 2024
pubmed: 29 2 2024
entrez: 28 2 2024
Statut: ppublish

Résumé

The cochlear aqueduct (CA) connects between the perilymphatic space of the cochlea and the subarachnoid space in the posterior cranial fossa. The study aimed to examine 1) whether cavitation of the CA occurs on the subarachnoid side or the cochlear side and 2) the growth and/or degeneration of the CA and its concomitant vein. We examined paraffin-embedded histological sections from human fetuses: 15 midterm fetuses (crown-rump length or CRL, 39-115 mm) and 12 near-term fetuses (CRL, 225-328 mm). A linear mesenchymal condensation, i.e., a likely candidate of the CA anlage, was observed without the accompanying vein at 9-10 weeks. The vein appeared until 15 weeks, but it was sometimes distant from the CA. At 10-12 weeks, the subarachnoid space (or the epidural space) near the glossopharyngeal nerve rapidly protruded into the CA anlage and reached the scala tympani, in which cavitation was gradually on-going but without epithelial lining. However, CA cavitation did not to occur in the anlage. At the opening to the scala, the epithelial-like lining of the CA lost its meningeal structure. At near-term, the CA was often narrowed and obliterated. The CA develops from meningeal tissues when the cavitation of the scala begins. The latter cavitation seemed to reduce tissue stiffness leading, to meningeal protrusion. The so-called anlage of CA might be a phylogenetic remnant of the glossopharyngeal nerve branch. A course of cochlear veins appears to be determined by a rule different from the CA development.

Sections du résumé

BACKGROUND AND AIM OBJECTIVE
The cochlear aqueduct (CA) connects between the perilymphatic space of the cochlea and the subarachnoid space in the posterior cranial fossa. The study aimed to examine 1) whether cavitation of the CA occurs on the subarachnoid side or the cochlear side and 2) the growth and/or degeneration of the CA and its concomitant vein.
METHODS METHODS
We examined paraffin-embedded histological sections from human fetuses: 15 midterm fetuses (crown-rump length or CRL, 39-115 mm) and 12 near-term fetuses (CRL, 225-328 mm).
RESULTS RESULTS
A linear mesenchymal condensation, i.e., a likely candidate of the CA anlage, was observed without the accompanying vein at 9-10 weeks. The vein appeared until 15 weeks, but it was sometimes distant from the CA. At 10-12 weeks, the subarachnoid space (or the epidural space) near the glossopharyngeal nerve rapidly protruded into the CA anlage and reached the scala tympani, in which cavitation was gradually on-going but without epithelial lining. However, CA cavitation did not to occur in the anlage. At the opening to the scala, the epithelial-like lining of the CA lost its meningeal structure. At near-term, the CA was often narrowed and obliterated.
CONCLUSION CONCLUSIONS
The CA develops from meningeal tissues when the cavitation of the scala begins. The latter cavitation seemed to reduce tissue stiffness leading, to meningeal protrusion. The so-called anlage of CA might be a phylogenetic remnant of the glossopharyngeal nerve branch. A course of cochlear veins appears to be determined by a rule different from the CA development.

Identifiants

pubmed: 38417484
pii: S0940-9602(24)00028-1
doi: 10.1016/j.aanat.2024.152236
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

152236

Informations de copyright

Copyright © 2024 Elsevier GmbH. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of Competing Interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Kwang Ho Cho (KH)

Department of Neurology, Wonkwang University School of Medicine and Hospital, Institute of Wonkwang Medical Science, Iksan, Republic of Korea. Electronic address: neurlogy@wonkwang.ac.kr.

Ji Hyun Kim (JH)

Department of Anatomy, Jeonbuk National University Medical School, Jeonju, Republic of Korea. Electronic address: 407kk@hanmail.net.

Yohei Honkura (Y)

Department of Otolaryngology-Head and Neck Surgery, Tohoku University Graduate School of Medicine, Sendai, Japan. Electronic address: y-honkura@orl.med.tohoku.ac.jp.

Masahito Yamamoto (M)

Department of Anatomy, Division of Basic Medical Science, Tokai University School of Medicine, Japan. Electronic address: masahitoyamamoto0412@gmail.com.

Gen Murakami (G)

Division of Internal Medicine, Cupid Clinic, Iwamizawa, Japan. Electronic address: g.murakamisaori@gmail.com.

Jose Francisco Rodríguez-Vázquez (JF)

Department of Anatomy and Embryology, School of Medicine, Complutense University, Madrid, Spain. Electronic address: jfrodvaz@ucm.es.

Yukio Katori (Y)

Department of Otolaryngology-Head and Neck Surgery, Tohoku University Graduate School of Medicine, Sendai, Japan. Electronic address: entsendai@yahoo.co.jp.

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