First-in-human Study With LIS1, a Next-generation Porcine Low Immunogenicity Antilymphocyte Immunoglobulin in Kidney Transplantation.


Journal

Transplantation
ISSN: 1534-6080
Titre abrégé: Transplantation
Pays: United States
ID NLM: 0132144

Informations de publication

Date de publication:
01 Jul 2024
Historique:
medline: 10 7 2024
pubmed: 10 7 2024
entrez: 10 7 2024
Statut: ppublish

Résumé

Polyclonal rabbit antithymocyte globulins (ATGs) are commonly used in organ transplantation as induction. Anti- N -glycolylneuraminic acid carbohydrate antibodies which develop in response to rabbit carbohydrate antigens might lead to unwanted systemic inflammation. LIS1, the first new generation of antilymphocyte globulins (ALGs) derived from double knockout swine, lacking carbohydrate xenoantigens was already tested in nonhuman primates and rodent models. This open-label, single-site, dose escalation, first-in-human, phase 1 study evaluated the safety, T cell depletion, pharmacokinetics, and pharmacodynamics of LIS1. In an ascending dose cohort (n = 5), a primary kidney transplant recipient at low immunologic risk (panel reactive antibody [PRA] < 20%), received LIS1 for 5 d at either 0.6, 1, 3, 6, or 8 mg/kg. After each patient completed treatment, the data safety monitoring board approved respective dose escalation. In the therapeutic dose cohort (n = 5) in patients with PRA <50% without donor specific antibodies, 2 patients received 8 mg/kg and 3 patients 10 mg/kg. CD3 + T cell depletion <100/mm 3 at day 2 was observed in all patients who received 6, 8, and 10 mg/kg of LIS1. The terminal half-life of LIS1 was 33.7 d with linearity in its disposition. Lymphocyte repopulation was fast and pretransplant lymphocyte subpopulation counts recovered within 2-4 wk. LIS1 was well tolerated, neither cytokine release syndrome nor severe thrombocytopenia or leukopenia were noticed. Antibodies to LIS1 were not detected. In this first-in-human trial, genome-edited swine-derived polyclonal LIS1 ALG was well tolerated, did not elicit antidrug antibodies, and caused time-limited T cell depletion in low- and medium-risk kidney transplant recipients.

Sections du résumé

BACKGROUND BACKGROUND
Polyclonal rabbit antithymocyte globulins (ATGs) are commonly used in organ transplantation as induction. Anti- N -glycolylneuraminic acid carbohydrate antibodies which develop in response to rabbit carbohydrate antigens might lead to unwanted systemic inflammation. LIS1, the first new generation of antilymphocyte globulins (ALGs) derived from double knockout swine, lacking carbohydrate xenoantigens was already tested in nonhuman primates and rodent models.
METHODS METHODS
This open-label, single-site, dose escalation, first-in-human, phase 1 study evaluated the safety, T cell depletion, pharmacokinetics, and pharmacodynamics of LIS1. In an ascending dose cohort (n = 5), a primary kidney transplant recipient at low immunologic risk (panel reactive antibody [PRA] < 20%), received LIS1 for 5 d at either 0.6, 1, 3, 6, or 8 mg/kg. After each patient completed treatment, the data safety monitoring board approved respective dose escalation. In the therapeutic dose cohort (n = 5) in patients with PRA <50% without donor specific antibodies, 2 patients received 8 mg/kg and 3 patients 10 mg/kg.
RESULTS RESULTS
CD3 + T cell depletion <100/mm 3 at day 2 was observed in all patients who received 6, 8, and 10 mg/kg of LIS1. The terminal half-life of LIS1 was 33.7 d with linearity in its disposition. Lymphocyte repopulation was fast and pretransplant lymphocyte subpopulation counts recovered within 2-4 wk. LIS1 was well tolerated, neither cytokine release syndrome nor severe thrombocytopenia or leukopenia were noticed. Antibodies to LIS1 were not detected.
CONCLUSIONS CONCLUSIONS
In this first-in-human trial, genome-edited swine-derived polyclonal LIS1 ALG was well tolerated, did not elicit antidrug antibodies, and caused time-limited T cell depletion in low- and medium-risk kidney transplant recipients.

Identifiants

pubmed: 38985979
doi: 10.1097/TP.0000000000004967
pii: 00007890-202407000-00026
doi:

Substances chimiques

Antilymphocyte Serum 0
Immunosuppressive Agents 0
alpha-1,3-galactosyltransferase 1, porcine EC 2.4.1.-
Galactosyltransferases EC 2.4.1.-

Banques de données

ClinicalTrials.gov
['NCT04431219']

Types de publication

Journal Article Clinical Trial, Phase I

Langues

eng

Sous-ensembles de citation

IM

Pagination

e139-e147

Informations de copyright

Copyright © 2024 Wolters Kluwer Health, Inc. All rights reserved.

Déclaration de conflit d'intérêts

J.R., P.-J.R., G.E., O.D., and B.V. are employees of Xenothera. O.D., J.-P.S., J.-M.B., and C.G. are cofounders of Xenothera. The other authors declare no conflicts of interest.

Références

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Auteurs

Ondrej Viklicky (O)

Department of Nephrology, Institute of Clinical and Experimental Medicine, Prague, Czech Republic.

Janka Slatinska (J)

Department of Nephrology, Institute of Clinical and Experimental Medicine, Prague, Czech Republic.

Libor Janousek (L)

Department of Nephrology, Institute of Clinical and Experimental Medicine, Prague, Czech Republic.

Juliette Rousse (J)

Xenothera, Nantes, France.

Pierre-Joseph Royer (PJ)

Xenothera, Nantes, France.

Pierre-Louis Toutain (PL)

Comparative Biomedical Sciences, The Royal Veterinary College, London, United Kingdom.
INTHERES, Université de Toulouse, INRAE, ENVT, Toulouse, France.

Emanuele Cozzi (E)

Transplantation Immunology Unit, Padua University Hospital, Padova, Italy.

Cesare Galli (C)

Avantea, Laboratory of Reproductive Technologies, Cremona, Italy.

Gwenaelle Evanno (G)

Xenothera, Nantes, France.

Odile Duvaux (O)

Xenothera, Nantes, France.

Jean-Marie Bach (JM)

Oniris, INRAE, IECM, USC 1383, Nantes, France.

Jean-Paul Soulillou (JP)

Institut de Transplantation Urologie Néphrologie (ITUN), CHU Nantes, Nantes, France.
Nantes Université, INSERM UMR1064, Center for Research in Transplantation and Translational Immunology, Nantes, France.

Magali Giral (M)

Institut de Transplantation Urologie Néphrologie (ITUN), CHU Nantes, Nantes, France.
Nantes Université, INSERM UMR1064, Center for Research in Transplantation and Translational Immunology, Nantes, France.

Bernard Vanhove (B)

Xenothera, Nantes, France.

Gilles Blancho (G)

Institut de Transplantation Urologie Néphrologie (ITUN), CHU Nantes, Nantes, France.
Nantes Université, INSERM UMR1064, Center for Research in Transplantation and Translational Immunology, Nantes, France.

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