Modulation of large rhythmic depolarizations in human large basket cells by norepinephrine and acetylcholine.


Journal

Communications biology
ISSN: 2399-3642
Titre abrégé: Commun Biol
Pays: England
ID NLM: 101719179

Informations de publication

Date de publication:
20 Jul 2024
Historique:
received: 13 04 2024
accepted: 03 07 2024
medline: 21 7 2024
pubmed: 21 7 2024
entrez: 20 7 2024
Statut: epublish

Résumé

Rhythmic brain activity is critical to many brain functions and is sensitive to neuromodulation, but so far very few studies have investigated this activity on the cellular level in vitro in human brain tissue samples. This study reveals and characterizes a novel rhythmic network activity in the human neocortex. Using intracellular patch-clamp recordings of human cortical neurons, we identify large rhythmic depolarizations (LRDs) driven by glutamate release but not by GABA. These LRDs are intricate events made up of multiple depolarizing phases, occurring at ~0.3 Hz, have large amplitudes and long decay times. Unlike human tissue, rat neocortex layers 2/3 exhibit no such activity under identical conditions. LRDs are mainly observed in a subset of L2/3 interneurons that receive substantial excitatory inputs and are likely large basket cells based on their morphology. LRDs are highly sensitive to norepinephrine (NE) and acetylcholine (ACh), two neuromodulators that affect network dynamics. NE increases LRD frequency through β-adrenergic receptor activity while ACh decreases it via M

Identifiants

pubmed: 39033173
doi: 10.1038/s42003-024-06546-2
pii: 10.1038/s42003-024-06546-2
doi:

Substances chimiques

Acetylcholine N9YNS0M02X
Norepinephrine X4W3ENH1CV

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

885

Subventions

Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : FOR2715

Informations de copyright

© 2024. The Author(s).

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Auteurs

Danqing Yang (D)

Research Center Juelich, Institute of Neuroscience and Medicine 10, Research Center Juelich, 52425, Juelich, Germany.
Department of Psychiatry, Psychotherapy, and Psychosomatics, RWTH Aachen University Hospital, 52074, Aachen, Germany.

Guanxiao Qi (G)

Research Center Juelich, Institute of Neuroscience and Medicine 10, Research Center Juelich, 52425, Juelich, Germany.

Jonas Ort (J)

Department of Neurosurgery, Faculty of Medicine, RWTH Aachen University Hospital, Aachen, Germany.
Neurosurgical Artificial Intelligence Laboratory Aachen (NAILA), RWTH Aachen University Hospital, 52074, Aachen, Germany.
Center for Integrated Oncology, Universities Aachen, Bonn, Cologne, Düsseldorf (CIO ABCD), Bonn, Germany.

Victoria Witzig (V)

Department of Neurology, RWTH Aachen University Hospital, 52074, Aachen, Germany.

Aniella Bak (A)

Department of Neurology, Section Epileptology, RWTH Aachen University Hospital, 52074, Aachen, Germany.

Daniel Delev (D)

Department of Neurosurgery, Faculty of Medicine, RWTH Aachen University Hospital, Aachen, Germany.
Neurosurgical Artificial Intelligence Laboratory Aachen (NAILA), RWTH Aachen University Hospital, 52074, Aachen, Germany.
Center for Integrated Oncology, Universities Aachen, Bonn, Cologne, Düsseldorf (CIO ABCD), Bonn, Germany.

Henner Koch (H)

Department of Neurology, Section Epileptology, RWTH Aachen University Hospital, 52074, Aachen, Germany.

Dirk Feldmeyer (D)

Research Center Juelich, Institute of Neuroscience and Medicine 10, Research Center Juelich, 52425, Juelich, Germany. d.feldmeyer@fz-juelich.de.
Department of Psychiatry, Psychotherapy, and Psychosomatics, RWTH Aachen University Hospital, 52074, Aachen, Germany. d.feldmeyer@fz-juelich.de.
Jülich-Aachen Research Alliance, Translational Brain Medicine (JARA Brain), Aachen, Germany. d.feldmeyer@fz-juelich.de.

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