Clinical relevance of aortic conduit and reservoir function.


Journal

Open heart
ISSN: 2053-3624
Titre abrégé: Open Heart
Pays: England
ID NLM: 101631219

Informations de publication

Date de publication:
19 Aug 2024
Historique:
received: 16 04 2024
accepted: 07 08 2024
medline: 20 8 2024
pubmed: 20 8 2024
entrez: 19 8 2024
Statut: epublish

Résumé

Aortic conduit and reservoir functions can be directly measured by four-dimensional flow (4D flow) cardiovascular magnetic resonance (CMR). Twenty healthy controls (10 young and 10 age-gender-matched old controls) and 20 patients with heart failure with preserved ejection fraction (HFpEF) were recruited. All had 4D flow CMR. Flow was quantified at the ascending and descending aorta levels. In addition, at the ascending aorta level, we quantified systolic flow displacement (FDs) and systolic flow reversal ratio (sFRR). The aortic conduit function was defined as the relative drop in systolic flow from the ascending to the descending aorta (∆Fs). Aortic reservoir function was defined as descending aortic diastolic stroke volume (DAo SV Both ∆Fs (R=0.51, p=0.001) and DAo SV Both aortic conduit and reservoir function decline with age and this decline in aortic function is also independently associated with renal functional decline. Ascending aortic turbulent flow signatures are associated with loss of aortic conduit and reservoir functions. Finally, in HFpEF, aortic conduit and reservoir function demonstrate progressive decline. NCT05114785.

Sections du résumé

BACKGROUND BACKGROUND
Aortic conduit and reservoir functions can be directly measured by four-dimensional flow (4D flow) cardiovascular magnetic resonance (CMR).
METHODS METHODS
Twenty healthy controls (10 young and 10 age-gender-matched old controls) and 20 patients with heart failure with preserved ejection fraction (HFpEF) were recruited. All had 4D flow CMR. Flow was quantified at the ascending and descending aorta levels. In addition, at the ascending aorta level, we quantified systolic flow displacement (FDs) and systolic flow reversal ratio (sFRR). The aortic conduit function was defined as the relative drop in systolic flow from the ascending to the descending aorta (∆Fs). Aortic reservoir function was defined as descending aortic diastolic stroke volume (DAo SV
RESULTS RESULTS
Both ∆Fs (R=0.51, p=0.001) and DAo SV
CONCLUSION CONCLUSIONS
Both aortic conduit and reservoir function decline with age and this decline in aortic function is also independently associated with renal functional decline. Ascending aortic turbulent flow signatures are associated with loss of aortic conduit and reservoir functions. Finally, in HFpEF, aortic conduit and reservoir function demonstrate progressive decline.
TRIALS REGISTRATION NUMBER BACKGROUND
NCT05114785.

Identifiants

pubmed: 39160086
pii: openhrt-2024-002713
doi: 10.1136/openhrt-2024-002713
pii:
doi:

Banques de données

ClinicalTrials.gov
['NCT05114785']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© Author(s) (or their employer(s)) 2024. Re-use permitted under CC BY. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: PG is a clinical advisor for Neosoft, Pie Medical Imaging and Medis Medical Imaging. PG consults for Anteris and Edward Life Sciences. All other authors have no competing interests to declare.

Auteurs

Hosamadin Assadi (H)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Chris Sawh (C)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Hilmar Spohr (H)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Faye Nelthorpe (F)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Sunil Nair (S)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Marina Hughes (M)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

David Ashman (D)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Alisdair Ryding (A)

Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Gareth Matthews (G)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Rui Li (R)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Ciaran Grafton-Clarke (C)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Zia Mehmood (Z)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Abdallah Al-Mohammad (A)

Department of Infection, Immunity & Cardiovascular Medicine, The University of Sheffield, Sheffield, UK.
South Yorkshire Cardiothoracic Centre, Sheffield Teaching Hospitals NHS Foundation Trust, Sheffield, UK.

Bahman Kasmai (B)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

Vassilios S Vassiliou (VS)

University of East Anglia, Norwich, UK.

Pankaj Garg (P)

Department of Cardiovascular and Metabolic Health, University of East Anglia, Norwich, UK pankajvic@gmail.com.
Norfolk and Norwich University Hospitals NHS Foundation Trust, Norwich, UK.

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Classifications MeSH