Attenuated effector T cells are linked to control of chronic HBV infection.


Journal

Nature immunology
ISSN: 1529-2916
Titre abrégé: Nat Immunol
Pays: United States
ID NLM: 100941354

Informations de publication

Date de publication:
Sep 2024
Historique:
received: 19 04 2024
accepted: 12 07 2024
medline: 31 8 2024
pubmed: 31 8 2024
entrez: 28 8 2024
Statut: ppublish

Résumé

Hepatitis B virus (HBV)-specific CD8

Identifiants

pubmed: 39198634
doi: 10.1038/s41590-024-01928-4
pii: 10.1038/s41590-024-01928-4
doi:

Substances chimiques

Transforming Growth Factor beta 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1650-1662

Subventions

Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : TRR 179 project no. 272983813
Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : CRC 1160 project no. 256073931
Organisme : Deutsche Forschungsgemeinschaft (German Research Foundation)
ID : CRC 1479 project no. 441891347

Informations de copyright

© 2024. The Author(s).

Références

World Health Organization. Hepatitis B. https://www.who.int/news-room/fact-sheets/detail/hepatitis-b (2024).
EASL. 2017 Clinical Practice Guidelines on the management of hepatitis B virus infection. J. Hepatol. 67, 370–398 (2017).
doi: 10.1016/j.jhep.2017.03.021
Thimme, R. et al. CD8(+) T cells mediate viral clearance and disease pathogenesis during acute hepatitis B virus infection. J. Virol. 77, 68–76 (2003).
pubmed: 12477811 pmcid: 140637 doi: 10.1128/JVI.77.1.68-76.2003
Maini, M. K. et al. The role of virus-specific CD8(+) cells in liver damage and viral control during persistent hepatitis B virus infection. J. Exp. Med. 191, 1269–1280 (2000).
pubmed: 10770795 pmcid: 2193131 doi: 10.1084/jem.191.8.1269
Webster, G. J. et al. Longitudinal analysis of CD8
pubmed: 15140968 pmcid: 415806 doi: 10.1128/JVI.78.11.5707-5719.2004
Boni, C. et al. Characterization of hepatitis B virus (HBV)-specific T-cell dysfunction in chronic HBV infection. J. Virol. 81, 4215–4225 (2007).
pubmed: 17287266 pmcid: 1866111 doi: 10.1128/JVI.02844-06
Das, A. et al. Functional skewing of the global CD8 T cell population in chronic hepatitis B virus infection. J. Exp. Med. 205, 2111–2124 (2008).
pubmed: 18695005 pmcid: 2526205 doi: 10.1084/jem.20072076
Kurktschiev, P. D. et al. Dysfunctional CD8
pubmed: 25225458 pmcid: 4172217 doi: 10.1084/jem.20131333
Schurich, A. et al. Distinct metabolic requirements of exhausted and functional virus-specific CD8 T cells in the same host. Cell Rep. 16, 1243–1252 (2016).
pubmed: 27452473 pmcid: 4977274 doi: 10.1016/j.celrep.2016.06.078
Fisicaro, P. et al. Targeting mitochondrial dysfunction can restore antiviral activity of exhausted HBV-specific CD8 T cells in chronic hepatitis B. Nat. Med. 23, 327–336 (2017).
pubmed: 28165481 doi: 10.1038/nm.4275
Heim, K. et al. TOX defines the degree of CD8
pubmed: 33097558 doi: 10.1136/gutjnl-2020-322404
Wherry, E. J. & Kurachi, M. Molecular and cellular insights into T cell exhaustion. Nat. Rev. Immunol. 15, 486–499 (2015).
pubmed: 26205583 pmcid: 4889009 doi: 10.1038/nri3862
McLane, L. M., Abdel-Hakeem, M. S. & Wherry, E. J. CD8 T cell exhaustion during chronic viral infection and cancer. Annu. Rev. Immunol. 37, 457–495 (2019).
pubmed: 30676822 doi: 10.1146/annurev-immunol-041015-055318
Bengsch, B. et al. Coexpression of PD-1, 2B4, CD160 and KLRG1 on exhausted HCV-specific CD8
pubmed: 20548953 pmcid: 2883597 doi: 10.1371/journal.ppat.1000947
Alfei, F. et al. TOX reinforces the phenotype and longevity of exhausted T cells in chronic viral infection. Nature 571, 265–269 (2019).
pubmed: 31207605 doi: 10.1038/s41586-019-1326-9
Blackburn, S. D., Shin, H., Freeman, G. J. & Wherry, E. J. Selective expansion of a subset of exhausted CD8 T cells by αPD-L1 blockade. PNAS 105, 15016–15021 (2008).
pubmed: 18809920 pmcid: 2567485 doi: 10.1073/pnas.0801497105
Utzschneider, D. T. et al. T cell factor 1-expressing memory-like CD8(+) T cells sustain the immune response to chronic viral infections. Immunity 45, 415–427 (2016).
pubmed: 27533016 doi: 10.1016/j.immuni.2016.07.021
Schuch, A. et al. Phenotypic and functional differences of HBV core-specific versus HBV polymerase-specific CD8
pubmed: 30622109 doi: 10.1136/gutjnl-2018-316641
Hoogeveen, R. C. et al. Phenotype and function of HBV-specific T cells is determined by the targeted epitope in addition to the stage of infection. Gut 68, 893–904 (2019).
pubmed: 30580250 doi: 10.1136/gutjnl-2018-316644
Winkler, F. et al. Enolase represents a metabolic checkpoint controlling the differential exhaustion programmes of hepatitis virus-specific CD8(+) T cells. Gut 72, 1971–1984 (2023).
pubmed: 37541771 doi: 10.1136/gutjnl-2022-328734
Cheng, Y. et al. Multifactorial heterogeneity of virus-specific T cells and association with the progression of human chronic hepatitis B infection. Science Immunol. 4, eaau6905 (2019).
doi: 10.1126/sciimmunol.aau6905
Aliabadi, E. et al. Impact of HBsAg and HBcrAg levels on phenotype and function of HBV-specific T cells in patients with chronic hepatitis B virus infection. Gut 71, 2300–2312 (2022).
pubmed: 34702717 doi: 10.1136/gutjnl-2021-324646
Bertoletti, A. & Ferrari, C. Adaptive immunity in HBV infection. J. Hepatol. 64, S71–s83 (2016).
pubmed: 27084039 doi: 10.1016/j.jhep.2016.01.026
Herman, J. S., Sagar & Grün, D. FateID infers cell fate bias in multipotent progenitors from single-cell RNA-seq data. Nat. Methods 15, 379–386 (2018).
pubmed: 29630061 doi: 10.1038/nmeth.4662
Hensel, N. et al. Memory-like HCV-specific CD8(+) T cells retain a molecular scar after cure of chronic HCV infection. Nat. Immunol. 22, 229–239 (2021).
pubmed: 33398179 doi: 10.1038/s41590-020-00817-w
Hao, Y. et al. Integrated analysis of multimodal single-cell data. Cell 184, 3573–3587.e3529 (2021).
pubmed: 34062119 pmcid: 8238499 doi: 10.1016/j.cell.2021.04.048
Korsunsky, I. et al. Fast, sensitive and accurate integration of single-cell data with Harmony. Nat. Methods 16, 1289–1296 (2019).
pubmed: 31740819 pmcid: 6884693 doi: 10.1038/s41592-019-0619-0
Giles, J. R. et al. Shared and distinct biological circuits in effector, memory and exhausted CD8(+) T cells revealed by temporal single-cell transcriptomics and epigenetics. Nat. Immunol. 23, 1600–1613 (2022).
pubmed: 36271148 pmcid: 10408358 doi: 10.1038/s41590-022-01338-4
Beltra, J. C. et al. Stat5 opposes the transcription factor Tox and rewires exhausted CD8(+) T cells toward durable effector-like states during chronic antigen exposure. Immunity 56, 2699–2718.e2611 (2023).
pubmed: 38091951 doi: 10.1016/j.immuni.2023.11.005
Giles, J. R. et al. Human epigenetic and transcriptional T cell differentiation atlas for identifying functional T cell-specific enhancers. Immunity 55, 557–574.e557 (2022).
pubmed: 35263570 pmcid: 9214622 doi: 10.1016/j.immuni.2022.02.004
Haghverdi, L., Büttner, M., Wolf, F. A., Buettner, F. & Theis, F. J. Diffusion pseudotime robustly reconstructs lineage branching. Nat. Methods 13, 845–848 (2016).
pubmed: 27571553 doi: 10.1038/nmeth.3971
Aibar, S. et al. SCENIC: single-cell regulatory network inference and clustering. Nat. Methods 14, 1083–1086 (2017).
pubmed: 28991892 pmcid: 5937676 doi: 10.1038/nmeth.4463
Guan, T. et al. ZEB1, ZEB2, and the miR-200 family form a counterregulatory network to regulate CD8(+) T cell fates. J. Exp. Med. 215, 1153–1168 (2018).
pubmed: 29449309 pmcid: 5881466 doi: 10.1084/jem.20171352
Genshaft, A. S. et al. Single-cell RNA sequencing of liver fine-needle aspirates captures immune diversity in the blood and liver in chronic hepatitis B patients. Hepatology 78, 1525–1541 (2023).
pubmed: 37158243
Dominguez, C. X. et al. The transcription factors ZEB2 and T-bet cooperate to program cytotoxic T cell terminal differentiation in response to LCMV viral infection. J. Exp. Med. 212, 2041–2056 (2015).
pubmed: 26503446 pmcid: 4647261 doi: 10.1084/jem.20150186
Omilusik, K. D. et al. Transcriptional repressor ZEB2 promotes terminal differentiation of CD8
doi: 10.1084/jem.20150194
Daniel, B. et al. Divergent clonal differentiation trajectories of T cell exhaustion. Nat. Immunol. 23, 1614–1627 (2022).
pubmed: 36289450 pmcid: 11225711 doi: 10.1038/s41590-022-01337-5
Iezzi, G., Karjalainen, K. & Lanzavecchia, A. The duration of antigenic stimulation determines the fate of naive and effector T cells. Immunity 8, 89–95 (1998).
pubmed: 9462514 doi: 10.1016/S1074-7613(00)80461-6
Shedlock, D. J. & Shen, H. Requirement for CD4 T cell help in generating functional CD8 T cell memory. Science 300, 337–339 (2003).
pubmed: 12690201 doi: 10.1126/science.1082305
Kolumam, G. A., Thomas, S., Thompson, L. J., Sprent, J. & Murali-Krishna, K. Type I interferons act directly on CD8 T cells to allow clonal expansion and memory formation in response to viral infection. J. Exp. Med. 202, 637–650 (2005).
pubmed: 16129706 pmcid: 2212878 doi: 10.1084/jem.20050821
Mescher, M. F. et al. Signals required for programming effector and memory development by CD8
pubmed: 16824119 doi: 10.1111/j.0105-2896.2006.00382.x
Williams, M. A., Tyznik, A. J. & Bevan, M. J. Interleukin-2 signals during priming are required for secondary expansion of CD8
pubmed: 16778891 pmcid: 2776073 doi: 10.1038/nature04790
Joshi, N. S. et al. Inflammation directs memory precursor and short-lived effector CD8(+) T cell fates via the graded expression of T-bet transcription factor. Immunity 27, 281–295 (2007).
pubmed: 17723218 pmcid: 2034442 doi: 10.1016/j.immuni.2007.07.010
Ruby, C. E., Redmond, W. L., Haley, D. & Weinberg, A. D. Anti-OX40 stimulation in vivo enhances CD8
pubmed: 17183611 doi: 10.1002/eji.200636428
Khakpoor, A. et al. Spatiotemporal differences in presentation of CD8 T cell epitopes during hepatitis B virus infection. J. Virol. 93, e01457–18 (2019).
pubmed: 30518652 pmcid: 6364024 doi: 10.1128/JVI.01457-18
Wiegand, S. B. et al. Soluble immune markers in the different phases of chronic hepatitis B virus infection. Sci. Rep. 9, 14118 (2019).
pubmed: 31575964 pmcid: 6773856 doi: 10.1038/s41598-019-50729-5
Dahmani, A. & Delisle, J. S. TGF-β in T cell biology: implications for cancer immunotherapy. Cancers 10, 194 (2018).
pubmed: 29891791 pmcid: 6025055 doi: 10.3390/cancers10060194
Metropulos, A. E. & Munshi, H. G. & Principe, D.R. The difficulty in translating the preclinical success of combined TGFβ and immune checkpoint inhibition to clinical trial. eBioMedicine 86, 104380 (2022).
pubmed: 36455409 pmcid: 9706619 doi: 10.1016/j.ebiom.2022.104380
Rivino, L. et al. Hepatitis B virus-specific T cells associate with viral control upon nucleos(t)ide-analogue therapy discontinuation. J. Clin. Investig. 128, 668–681 (2018).
pubmed: 29309050 pmcid: 5785266 doi: 10.1172/JCI92812
Tian, Y., James Ou, J. H., Hepatitis, B. & Virus-Specific, T. Cells as a biomarker for discontinuation of nucleos(t)ide analogue therapy for chronic hepatitis B. Hepatology 69, 1342–1344 (2019).
pubmed: 30168616 doi: 10.1002/hep.30243
Collins, D. R., Gaiha, G. D. & Walker, B. D. CD8(+) T cells in HIV control, cure and prevention. Nat. Rev. Immunol. 20, 471–482 (2020).
pubmed: 32051540 pmcid: 7222980 doi: 10.1038/s41577-020-0274-9
Pallett, L. J. & Maini, M. K. Liver-resident memory T cells: life in lockdown. Semin. Immunopathol. https://doi.org/10.1007/s00281-022-00932-w (2022).
Crowell H. L., Zanotelli, V. R. T., Chevrier S., Robinson M. CATALYST: Cytometry dATa anALYSis Tools. R package version 1.15.0 https://github.com/HelenaLC/CATALYST (2020).
Hashimshony, T. et al. CEL-Seq2: sensitive highly-multiplexed single-cell RNA-seq. Genome Biol. 17, 77 (2016).
pubmed: 27121950 pmcid: 4848782 doi: 10.1186/s13059-016-0938-8
Li, H. & Durbin, R. Fast and accurate long-read alignment with Burrows-Wheeler transform. Bioinformatics 26, 589–595 (2010).
pubmed: 20080505 pmcid: 2828108 doi: 10.1093/bioinformatics/btp698
Baker, S. C. et al. The External RNA Controls Consortium: a progress report. Nat. Methods 2, 731–734 (2005).
pubmed: 16179916 doi: 10.1038/nmeth1005-731
Grun, D., Kester, L. & van Oudenaarden, A. Validation of noise models for single-cell transcriptomics. Nat. Methods 11, 637–640 (2014).
pubmed: 24747814 doi: 10.1038/nmeth.2930
Aizarani, N. et al. A human liver cell atlas reveals heterogeneity and epithelial progenitors. Nature 572, 199–204 (2019).
pubmed: 31292543 pmcid: 6687507 doi: 10.1038/s41586-019-1373-2
Sankowski, R. et al. Mapping microglia states in the human brain through the integration of high-dimensional techniques. Nat. Neurosci. 22, 2098–2110 (2019).
pubmed: 31740814 doi: 10.1038/s41593-019-0532-y
Nusser, A. et al. Developmental dynamics of two bipotent thymic epithelial progenitor types. Nature 606, 165–171 (2022).
pubmed: 35614226 pmcid: 9159946 doi: 10.1038/s41586-022-04752-8
Grun, D. et al. De novo prediction of stem cell identity using single-cell transcriptome data. Cell Stem Cell 19, 266–277 (2016).
pubmed: 27345837 pmcid: 4985539 doi: 10.1016/j.stem.2016.05.010

Auteurs

Kathrin Heim (K)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Özlem Sogukpinar (Ö)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Sian Llewellyn-Lacey (S)

Division of Infection and Immunity, Cardiff University School of Medicine, University Hospital of Wales, Cardiff, UK.

David A Price (DA)

Division of Infection and Immunity, Cardiff University School of Medicine, University Hospital of Wales, Cardiff, UK.
Systems Immunity Research Institute, Cardiff University School of Medicine, University Hospital of Wales, Cardiff, UK.

Florian Emmerich (F)

Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Institute for Transfusion Medicine and Gene Therapy, University Medical Center, University of Freiburg, Freiburg, Germany.

Anke R M Kraft (ARM)

Department of Gastroenterology, Hepatology, Infectious Diseases and Endocrinology, Hannover Medical School, Hannover, Germany.
German Centre for Infection Research (DZIF), partner site Hannover-Braunschweig, Braunschweig, Germany.
Centre for Individualised Infection Medicine (CiiM), Hannover, Germany.
Cluster of Excellence Resolving Infection Susceptibility (RESIST; EXC), Hannover Medical School, Hannover, Germany.

Markus Cornberg (M)

Department of Gastroenterology, Hepatology, Infectious Diseases and Endocrinology, Hannover Medical School, Hannover, Germany.
German Centre for Infection Research (DZIF), partner site Hannover-Braunschweig, Braunschweig, Germany.
Centre for Individualised Infection Medicine (CiiM), Hannover, Germany.
Cluster of Excellence Resolving Infection Susceptibility (RESIST; EXC), Hannover Medical School, Hannover, Germany.

Sophie Kielbassa (S)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Percy Knolle (P)

Institute of Molecular Immunology, School of Medicine, Technical University of Munich (TUM), Munich, Germany.
German Center for Infection Research, Munich, Germany.
Institute of Molecular Immunology, School of Life Science, TUM, Munich, Germany.

Dirk Wohlleber (D)

Institute of Molecular Immunology, School of Medicine, Technical University of Munich (TUM), Munich, Germany.

Bertram Bengsch (B)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.
Signaling Research Centres BIOSS and CIBSS, University of Freiburg, Freiburg, Germany.

Tobias Boettler (T)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Christoph Neumann-Haefelin (C)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany.
Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Robert Thimme (R)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany. robert.thimme@uniklinik-freiburg.de.
Faculty of Medicine, University of Freiburg, Freiburg, Germany. robert.thimme@uniklinik-freiburg.de.

Maike Hofmann (M)

Department of Medicine II, Medical Center - University of Freiburg, Freiburg, Germany. maike.hofmann@uniklinik-freiburg.de.
Faculty of Medicine, University of Freiburg, Freiburg, Germany. maike.hofmann@uniklinik-freiburg.de.

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