HIV-1 residual risk and pre-treatment drug resistance among blood donors: A sentinel surveillance from Gabon.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2024
Historique:
received: 07 02 2024
accepted: 06 06 2024
medline: 3 9 2024
pubmed: 3 9 2024
entrez: 3 9 2024
Statut: epublish

Résumé

Surveillance of HIV-1 pre-treatment drug resistance (PDR) is essential for ensuring the success of first-line antiretroviral therapy (ART). Beside population-based surveys, sentinel surveillance of PDR and circulating HIV-1 clades in specific populations such as blood donors could efficiently inform decision-making on ART program. We therefore sought to ascertain HIV-1 residual infection, the threshold of PDR and viral diversity among recently-diagnosed blood donors in Gabon. A sentinel surveillance was conducted among 381 consenting blood donors at the National Blood Transfusion Center (NBTC) in Gabon from August 3,2020 to August, 31, 2021. In order to determine the residual risk of HIV transmission, viral load and HIV-1 Sanger-sequencing were performed at the Chantal BIYA International Reference Center (CIRCB)-Cameroon on HIV samples previously tested seronegative with ELISA in Gabon. Phylogeny was performed using MEGA X, PDR threshold>10% was considered high and data were analysed using p≤0.05 for statistical significance. Five HIV-negative blood donors had a detectable viral load indicating a high residual risk of HIV transmission. Among the samples successfully sequenced, four participants had major drug resistance mutations (DRMs), giving a threshold of PDR of 25% (4/16). By drug class, major DRMs targeting NNRTI (K103N, E138G), NRTIs (L210W) and PI/r (M46L). The most representative viral clades were CRF02_AG and subtype A1. The genetic diversity of HIV-1 had no significant effect on the residual risk in blood transfusion (CRF02_AG, P = 0.3 and Recombinants, P = 0.5). This sentinel surveillance indicates a high residual risk of HIV-1 transfusion in Gabon, thereby underscoring the need for optimal screening strategy for blood safety. Moreover, HIV-1 transmission goes with high-risk of PDR, suggesting suboptimal efficacy of ART. Nonetheless, the genetic diversity has limited (if any effect) on the residual risk of infection and PDR in blood donors.

Sections du résumé

BACKGROUND BACKGROUND
Surveillance of HIV-1 pre-treatment drug resistance (PDR) is essential for ensuring the success of first-line antiretroviral therapy (ART). Beside population-based surveys, sentinel surveillance of PDR and circulating HIV-1 clades in specific populations such as blood donors could efficiently inform decision-making on ART program. We therefore sought to ascertain HIV-1 residual infection, the threshold of PDR and viral diversity among recently-diagnosed blood donors in Gabon.
METHODS METHODS
A sentinel surveillance was conducted among 381 consenting blood donors at the National Blood Transfusion Center (NBTC) in Gabon from August 3,2020 to August, 31, 2021. In order to determine the residual risk of HIV transmission, viral load and HIV-1 Sanger-sequencing were performed at the Chantal BIYA International Reference Center (CIRCB)-Cameroon on HIV samples previously tested seronegative with ELISA in Gabon. Phylogeny was performed using MEGA X, PDR threshold>10% was considered high and data were analysed using p≤0.05 for statistical significance.
RESULTS RESULTS
Five HIV-negative blood donors had a detectable viral load indicating a high residual risk of HIV transmission. Among the samples successfully sequenced, four participants had major drug resistance mutations (DRMs), giving a threshold of PDR of 25% (4/16). By drug class, major DRMs targeting NNRTI (K103N, E138G), NRTIs (L210W) and PI/r (M46L). The most representative viral clades were CRF02_AG and subtype A1. The genetic diversity of HIV-1 had no significant effect on the residual risk in blood transfusion (CRF02_AG, P = 0.3 and Recombinants, P = 0.5).
CONCLUSION CONCLUSIONS
This sentinel surveillance indicates a high residual risk of HIV-1 transfusion in Gabon, thereby underscoring the need for optimal screening strategy for blood safety. Moreover, HIV-1 transmission goes with high-risk of PDR, suggesting suboptimal efficacy of ART. Nonetheless, the genetic diversity has limited (if any effect) on the residual risk of infection and PDR in blood donors.

Identifiants

pubmed: 39226273
doi: 10.1371/journal.pone.0305935
pii: PONE-D-24-02129
doi:

Substances chimiques

Anti-HIV Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0305935

Informations de copyright

Copyright: © 2024 Mangala et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Déclaration de conflit d'intérêts

The authors have declared that there are no interests competitors.

Auteurs

Christian Mangala (C)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Virology Department, National Public Health Laboratory (NPHL), Libreville, Gabon.

Désiré Takou (D)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Denis Maulot-Bangola (D)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Virology Department, National Blood Transfusion Centre (NBTC), Libreville, Gabon.

Grace Beloumou (G)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Olivier Rebienot Pellegrin (O)

Virology Department, National Blood Transfusion Centre (NBTC), Libreville, Gabon.

Samuel Martin Sosso (SM)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Collins Ambe Chenwi (C)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
Faculty of Medicine and Surgery, University of Rome Tor Vergata, Rome, Italy.

Ezechiel Ngoufack Jagni Semengue (E)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Franck Vigan Codjo (F)

Virology Department, National Blood Transfusion Centre (NBTC), Libreville, Gabon.

Olga Boussougou (O)

Virology Department, National Blood Transfusion Centre (NBTC), Libreville, Gabon.

Alex Durand Nka (AD)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Michel Tommo (M)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.

Nadine Fainguem (N)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Rachel Kamgaing (R)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.

Vicky Ama Moor (V)

School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Faculty of Medicine and Biomedical Sciences, University of Yaoundé 1 (FMBS-UY1), Yaoundé, Cameroon.
Laboratories of Biochemistry and Microbiology, University Teaching Hospital, Yaoundé, Cameroon.

Hortense Kamga Gonsu (H)

School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Faculty of Medicine and Biomedical Sciences, University of Yaoundé 1 (FMBS-UY1), Yaoundé, Cameroon.
Laboratories of Biochemistry and Microbiology, University Teaching Hospital, Yaoundé, Cameroon.

Veronique Penlap (V)

School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Biotechnology Centre, University of Yaounde I, Yaounde, Cameroon.

Thérèse Nkoa (T)

School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Faculty of Medicine and Biomedical Sciences, University of Yaoundé 1 (FMBS-UY1), Yaoundé, Cameroon.

Vittorio Colizzi (V)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
Faculty of Science and Technology, Evangelic University of Cameroon, Bandjoun, Cameroon.
UNESCO Chair of Biotechnology, University of Rome Tor Vergata, Rome, Italy.

Carlo-Federico Perno (CF)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
Department of Microbiology, Bambino Gesu Pediatric Hospital, Rome, Italy.

Joseph Fokam (J)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
School of Health Sciences, Catholic University of Central Africa (ESS-UCAC), Yaoundé, Cameroon.
Faculty of Medicine and Biomedical Sciences, University of Yaoundé 1 (FMBS-UY1), Yaoundé, Cameroon.
Faculty of Health Sciences, University of Buea (FHS-UB), Buea, Cameroon.
National HIV Drug Resistance working group, Ministry of Public Health, Yaounde, Cameroon.

Alexis Ndjolo (A)

Chantal BIYA International Reference Centre for research on HIV/AIDS Prevention and Management (CIRCB), Yaoundé, Cameroon.
Faculty of Medicine and Biomedical Sciences, University of Yaoundé 1 (FMBS-UY1), Yaoundé, Cameroon.

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