The spectrum of systemic sclerosis-associated pulmonary hypertension: Insights from the ASPIRE registry.


Journal

The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation
ISSN: 1557-3117
Titre abrégé: J Heart Lung Transplant
Pays: United States
ID NLM: 9102703

Informations de publication

Date de publication:
Oct 2024
Historique:
received: 04 04 2024
revised: 07 06 2024
accepted: 13 06 2024
medline: 12 9 2024
pubmed: 12 9 2024
entrez: 11 9 2024
Statut: ppublish

Résumé

There are limited data assessing the spectrum of systemic sclerosis-associated pulmonary hypertension (PH). Data for 912 systemic sclerosis patients assessed between 2000 and 2020 were retrieved from the Assessing the Spectrum of Pulmonary hypertension Identified at a REferral centre (ASPIRE) registry and classified based on 2022 European Society of Cardiology/European Respiratory Society (ESC/ERS) guidelines and multimodality investigations. Reduction in pulmonary vascular resistance (PVR) diagnostic threshold to >2WU resulted in a 19% increase in precapillary PH diagnoses. Patients with PVR ≤2WU had superior survival to PVR >2-3WU which was similar to PVR >3-4WU. Survival in pulmonary arterial hypertension (PAH) was superior to PH associated with lung disease. However, patients with mild parenchymal disease on CT had similar characteristics and outcomes to patients without lung disease. Combined pre- and postcapillary PH had significantly poorer survival than isolated postcapillary PH. Patients with mean pulmonary arterial wedge pressure (PAWP) 13-15 mm Hg had similar haemodynamics and left atrial volumes to those with PAWP >15 mm Hg. Unclassified-PH had more frequently dilated left atria and higher PAWP than PAH. Although Unclassified-PH had a similar survival to No-PH, 36% were subsequently diagnosed with PAH or PH associated with left heart disease. The presence of 2-3 radiological signs of pulmonary veno-occlusive disease was noted in 7% of PAH patients and was associated with worse survival. Improvement in incremental shuttle walking distance of ≥30 m following initiation of PAH therapy was associated with superior survival. PAH patients diagnosed after 2011 had greater use of combination therapy and superior survival. A number of systemic sclerosis PH phenotypes can be recognized and characterized using haemodynamics, lung function and multimodality imaging.

Sections du résumé

BACKGROUND BACKGROUND
There are limited data assessing the spectrum of systemic sclerosis-associated pulmonary hypertension (PH).
METHODS METHODS
Data for 912 systemic sclerosis patients assessed between 2000 and 2020 were retrieved from the Assessing the Spectrum of Pulmonary hypertension Identified at a REferral centre (ASPIRE) registry and classified based on 2022 European Society of Cardiology/European Respiratory Society (ESC/ERS) guidelines and multimodality investigations.
RESULTS RESULTS
Reduction in pulmonary vascular resistance (PVR) diagnostic threshold to >2WU resulted in a 19% increase in precapillary PH diagnoses. Patients with PVR ≤2WU had superior survival to PVR >2-3WU which was similar to PVR >3-4WU. Survival in pulmonary arterial hypertension (PAH) was superior to PH associated with lung disease. However, patients with mild parenchymal disease on CT had similar characteristics and outcomes to patients without lung disease. Combined pre- and postcapillary PH had significantly poorer survival than isolated postcapillary PH. Patients with mean pulmonary arterial wedge pressure (PAWP) 13-15 mm Hg had similar haemodynamics and left atrial volumes to those with PAWP >15 mm Hg. Unclassified-PH had more frequently dilated left atria and higher PAWP than PAH. Although Unclassified-PH had a similar survival to No-PH, 36% were subsequently diagnosed with PAH or PH associated with left heart disease. The presence of 2-3 radiological signs of pulmonary veno-occlusive disease was noted in 7% of PAH patients and was associated with worse survival. Improvement in incremental shuttle walking distance of ≥30 m following initiation of PAH therapy was associated with superior survival. PAH patients diagnosed after 2011 had greater use of combination therapy and superior survival.
CONCLUSION CONCLUSIONS
A number of systemic sclerosis PH phenotypes can be recognized and characterized using haemodynamics, lung function and multimodality imaging.

Identifiants

pubmed: 39260921
pii: S1053-2498(24)01699-1
doi: 10.1016/j.healun.2024.06.007
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1629-1639

Informations de copyright

Copyright © 2024 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Howard Smith (H)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK.

A A Roger Thompson (AAR)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK; Division of Clinical Medicine, School of Medicine and Population Health, University of Sheffield, Sheffield, UK.

Mohammed Akil (M)

Department of Rheumatology, Royal Hallamshire Hospital, Sheffield, UK.

Samer Alabed (S)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK.

Athanasios Charalampopoulos (A)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK.

Krit Dwivedi (K)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK.

Charlie A Elliot (CA)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK.

Abdul Hameed (A)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK; Division of Clinical Medicine, School of Medicine and Population Health, University of Sheffield, Sheffield, UK.

Ashraful Haque (A)

Department of Rheumatology, Royal Hallamshire Hospital, Sheffield, UK.

Neil Hamilton (N)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK.

Catherine Hill (C)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK.

Judith Hurdman (J)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK.

Rachael Kilding (R)

Department of Rheumatology, Royal Hallamshire Hospital, Sheffield, UK.

Kar-Ping Kuet (KP)

Department of Rheumatology, Royal Hallamshire Hospital, Sheffield, UK.

Smitha Rajaram (S)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK.

Alexander M K Rothman (AMK)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK; Division of Clinical Medicine, School of Medicine and Population Health, University of Sheffield, Sheffield, UK; National Institute for Health and Care Research Sheffield Biomedical Research Centre, Sheffield, UK.

Andrew J Swift (AJ)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK; Insigneo Institute, University of Sheffield, Sheffield, UK; National Institute for Health and Care Research Sheffield Biomedical Research Centre, Sheffield, UK.

James M Wild (JM)

Department of Radiology, Royal Hallamshire Hospital, Sheffield, UK; Insigneo Institute, University of Sheffield, Sheffield, UK; National Institute for Health and Care Research Sheffield Biomedical Research Centre, Sheffield, UK.

David G Kiely (DG)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK; Division of Clinical Medicine, School of Medicine and Population Health, University of Sheffield, Sheffield, UK; Insigneo Institute, University of Sheffield, Sheffield, UK; National Institute for Health and Care Research Sheffield Biomedical Research Centre, Sheffield, UK.

Robin Condliffe (R)

Sheffield Pulmonary Vascular Disease Unit, Royal Hallamshire Hospital, Sheffield, UK; Division of Clinical Medicine, School of Medicine and Population Health, University of Sheffield, Sheffield, UK. Electronic address: robin.condliffe@nhs.net.

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