Neurotransmitter recognition by human vesicular monoamine transporter 2.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
16 Sep 2024
Historique:
received: 15 11 2023
accepted: 20 08 2024
medline: 17 9 2024
pubmed: 17 9 2024
entrez: 16 9 2024
Statut: epublish

Résumé

Human vesicular monoamine transporter 2 (VMAT2), a member of the SLC18 family, plays a crucial role in regulating neurotransmitters in the brain by facilitating their uptake and storage within vesicles, preparing them for exocytotic release. Because of its central role in neurotransmitter signalling and neuroprotection, VMAT2 is a target for neurodegenerative diseases and movement disorders, with its inhibitor being used as therapeutics. Despite the importance of VMAT2 in pharmacophysiology, the molecular basis of VMAT2-mediated neurotransmitter transport and its inhibition remains unclear. Here we show the cryo-electron microscopy structure of VMAT2 in the substrate-free state, in complex with the neurotransmitter dopamine, and in complex with the inhibitor tetrabenazine. In addition to these structural determinations, monoamine uptake assays, mutational studies, and pKa value predictions were performed to characterize the dynamic changes in VMAT2 structure. These results provide a structural basis for understanding VMAT2-mediated vesicular transport of neurotransmitters and a platform for modulation of current inhibitor design.

Identifiants

pubmed: 39284862
doi: 10.1038/s41467-024-51960-z
pii: 10.1038/s41467-024-51960-z
doi:

Substances chimiques

Vesicular Monoamine Transport Proteins 0
SLC18A2 protein, human 0
Tetrabenazine Z9O08YRN8O
Dopamine VTD58H1Z2X
Neurotransmitter Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

7661

Subventions

Organisme : MEXT | Japan Society for the Promotion of Science (JSPS)
ID : 19H00923, 22KK0099, 23K06357
Organisme : Japan Agency for Medical Research and Development (AMED)
ID : JP21am0101079, JP23ama121007, JP23ama121001, 23809479

Informations de copyright

© 2024. The Author(s).

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Auteurs

Dohyun Im (D)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan. im.dohyun.3s@kyoto-u.ac.jp.

Mika Jormakka (M)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.

Narinobu Juge (N)

Department of Genomics and Proteomics, Advanced Science Research Center, Okayama University, Okayama, Japan.
Department of Molecular Membrane Biology, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama, Japan.

Jun-Ichi Kishikawa (JI)

Department of Applied Biology, Kyoto Institute of Technology, Kyoto, Japan.
Institute for Protein Research, Osaka University, Suita, Japan.

Takayuki Kato (T)

Institute for Protein Research, Osaka University, Suita, Japan.

Yukihiko Sugita (Y)

Laboratory of Ultrastructural Virology, Institute for Life and Medical Sciences, Kyoto University, Kyoto, Japan.
Laboratory of Ultrastructural Virology, Graduate School of Biostudies, Kyoto University, Kyoto, Japan.
Hakubi Center for Advanced Research, Kyoto University, Kyoto, Japan.

Takeshi Noda (T)

Laboratory of Ultrastructural Virology, Institute for Life and Medical Sciences, Kyoto University, Kyoto, Japan.
Laboratory of Ultrastructural Virology, Graduate School of Biostudies, Kyoto University, Kyoto, Japan.
CREST, Japan Science and Technology Agency, Kawaguchi, Japan.

Tomoko Uemura (T)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.

Yuki Shiimura (Y)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.
Institute of Life Science, Kurume University, Kurume, Japan.

Takaaki Miyaji (T)

Department of Genomics and Proteomics, Advanced Science Research Center, Okayama University, Okayama, Japan.
Department of Molecular Membrane Biology, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama, Japan.

Hidetsugu Asada (H)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan.

So Iwata (S)

Department of Cell Biology, Graduate School of Medicine, Kyoto University, Kyoto, Japan. iwata.so.2z@kyoto-u.ac.jp.
RIKEN SPring-8 Center, Sayo-gun, Japan. iwata.so.2z@kyoto-u.ac.jp.

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