Impact of smokeless tobacco on psychological and oxidative stress in unemployed indian youth.


Journal

Amino acids
ISSN: 1438-2199
Titre abrégé: Amino Acids
Pays: Austria
ID NLM: 9200312

Informations de publication

Date de publication:
12 Oct 2024
Historique:
received: 05 11 2023
accepted: 20 08 2024
medline: 13 10 2024
pubmed: 13 10 2024
entrez: 12 10 2024
Statut: epublish

Résumé

In India, tobacco (nicotine) addiction among youth has increased, leading to substantial socioeconomic burdens, mortality, and morbidity. While minimal short-term nicotine consumption may have antioxidant effects, chronic exposure results in various adverse health outcomes. This study examines the impact of chronic nicotine consumption on cellular oxidative stress and psychological stress, and their correlation with Homocysteine (Hcy) levels in unemployed tobacco consumers. This case-control study included 156 healthy, educated, unemployed male volunteers aged 20-40 years, divided into nicotine-addicted (n = 80) and non-addicted (n = 76) groups. Psychological stress was assessed using perceived stress scales (PSS) and coping self-efficacy (CSE) scales. Oxidative stress markers, including Malondialdehyde (MDA), Superoxide Dismutase (SOD), and Catalase, were measured. Hcy levels were quantified using high-performance liquid chromatography (HPLC). Nicotine-addicted participants exhibited significantly higher perceived stress (p = 0.0001) and lower coping self-efficacy (p = 0.0001) compared to non-addicted individuals. MDA levels in erythrocytes were significantly increased (p = 0.0006), while SOD (p = 0.0001) and Catalase (p = 0.02) activities were significantly decreased in the addicted group. Nicotine intake influenced Hcy concentrations, with 55% of addicted individuals falling into moderate, 27.5% into intermediate, and 7.5% into severe Hcy categories. Chronic nicotine intake also reflected the hematological parameters (WBCs, RBCs, HGB, and Platelets). Chronic tobacco consumption induces oxidative stress and perceived psychological stress, leading to elevated Hcy levels in nicotine consumers. The study highlights the detrimental effects of nicotine addiction on cellular defensive mechanisms, emphasizing the need for targeted interventions to address this growing health issue among unemployed Indian youth.

Identifiants

pubmed: 39395920
doi: 10.1007/s00726-024-03416-3
pii: 10.1007/s00726-024-03416-3
doi:

Substances chimiques

Homocysteine 0LVT1QZ0BA
Malondialdehyde 4Y8F71G49Q
Superoxide Dismutase EC 1.15.1.1
Catalase EC 1.11.1.6

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

59

Informations de copyright

© 2024. The Author(s).

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Auteurs

Anurag Mishra (A)

Department of Biochemistry, Faculty of Science, University of Allahabad, Prayagraj, 211002, UP, India.

Rishabh Kumar (R)

Department of Biochemistry, Faculty of Science, University of Allahabad, Prayagraj, 211002, UP, India.

Satya Narayan Mishra (SN)

Maa Gayatri College of Pharmacy, Dr. A. P. J. Abdul Kalam Technical University, Prayagraj, 211008, UP, India.

Sivakumar Vijayaraghavalu (S)

Department of Life Sciences (Zoology), Manipur University (A Central University), Imphal, 795003, Manipur, India. drshiva@manipuruniv.ac.in.

Girish C Shukla (GC)

Department of Biological, Geological and Environmental Sciences, 2121 Euclid Ave, Cleveland, OH, 44115, USA.
Center for Gene Regulation in Health and Disease, 2121 Euclid Ave, Cleveland, OH, 44115, USA.

Munish Kumar (M)

Department of Biochemistry, Faculty of Science, University of Allahabad, Prayagraj, 211002, UP, India. munish@allduniv.ac.in.

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