Apolipoprotein-B mRNA-editing complex 3B could be a new potential therapeutic target in endometriosis.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
23 10 2024
Historique:
received: 05 08 2024
accepted: 15 10 2024
medline: 24 10 2024
pubmed: 24 10 2024
entrez: 24 10 2024
Statut: epublish

Résumé

This study investigated the correlation of Apolipoprotein-B mRNA-editing complex 3B (APOBEC3B) expression with hypoxia inducible factor 1α (HIF-1α), Kirsten rat sarcoma virus (KRAS) and phosphatidylinositol-4,5-bisphosphate 3-kinase catalytic subunit alpha (PIK3CA) in endometriosis patients, and the inhibitory effects of APOBEC3B knockdown in a human endometriotic cell line. Here, APOBEC3B, HIF-1α, KRAS, and PIK3CA were examined in patients with and without endometriosis using reverse transcription polymerase chain reaction (RT-PCR). The apoptosis, cell proliferation, invasion, migration, and biological function of APOBEC3B knockdown were explored in 12Z immortalized human endometriotic cell line. We observed APOBEC3B, HIF-1α, KRAS and PIK3CA expressions were significantly higher in endometriosis patients (p < 0.001, p < 0.001, p = 0.029, p = 0.001). Knockdown of APOBEC3B increased apoptosis, which was 28.03% and 22.27% higher than in mock and control siRNA samples, respectively. APOBEC3B knockdown also decreased PIK3CA expression and increased Caspase 8 expression, suggesting a potential role in the regulation of apoptosis. Furthermore, knockdown of APOBEC3B significantly inhibited cell proliferation, invasion, and migration compared to mock and control siRNA. (Cell proliferation: mock: p < 0.001 and control siRNA: p = 0.049. Cell invasion: mock: p < 0.001 and control siRNA: p = 0.029. Cell migration: mock: p = 0.004, and control siRNA: p = 0.014). In conclusion, this study suggests that APOBEC3B may be a new potential therapeutic target for endometriosis.

Identifiants

pubmed: 39443671
doi: 10.1038/s41598-024-76589-2
pii: 10.1038/s41598-024-76589-2
doi:

Substances chimiques

APOBEC3B protein, human EC 3.5.4.5
Cytidine Deaminase EC 3.5.4.5
Minor Histocompatibility Antigens 0
Class I Phosphatidylinositol 3-Kinases EC 2.7.1.137
PIK3CA protein, human EC 2.7.1.137
Proto-Oncogene Proteins p21(ras) EC 3.6.5.2
KRAS protein, human 0
HIF1A protein, human 0
Hypoxia-Inducible Factor 1, alpha Subunit 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

24968

Subventions

Organisme : Japan Society for the Promotion of Science
ID : 22K09619
Organisme : Japan Society for the Promotion of Science
ID : 23K15815

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Thuy Ha Vu (TH)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.
Department of Histopathology, Haiphong University of Medicine and Pharmacy, 72A Nguyen Binh Khiem St, Ngo Quyen Dist, Hai Phong, 180000, Vietnam.

Keiichiro Nakamura (K)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan. k-nakamu@cc.okayama-u.ac.jp.

Kunitoshi Shigeyasu (K)

Department of Gastroenterological Surgery, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

Chiaki Kashino (C)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

Kazuhiro Okamoto (K)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

Kotaro Kubo (K)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

Yasuhiko Kamada (Y)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

Hisashi Masuyama (H)

Department of Obstetrics and Gynecology, Dentistry and Pharmaceutical Sciences, Okayama University Graduate School of Medicine, 2-5-1 Shikata-cho, Kita-ku, Okayama, 700- 8558, Japan.

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