Establishment and characterization of a patient-derived metastatic extraskeletal Ewing sarcoma cell line ES-ZSS-1.


Journal

Human cell
ISSN: 1749-0774
Titre abrégé: Hum Cell
Pays: Japan
ID NLM: 8912329

Informations de publication

Date de publication:
30 Oct 2024
Historique:
received: 10 07 2024
accepted: 04 10 2024
medline: 31 10 2024
pubmed: 30 10 2024
entrez: 30 10 2024
Statut: epublish

Résumé

The methods available for treating metastatic Ewing sarcoma (ES) are inadequate; thus, innovative therapeutic approaches need to be developed. However, the lack of clinically relevant ES models has hindered the discovery of drugs for this disease. In this study, we established and characterized a patient-derived xenograft (PDX) cell line model, which was constructed using tumor tissue from a patient with metastatic extraskeletal ES. The cells were found to recapitulate the morphological and histopathological features of the patient tumor and were designated as ES-ZSS-1. The cells harbor the characteristic EWSR1-FLI1 infusion and underwent successive passages in vitro. By performing gene expression profiling, we found that the mutation in STAG2 was the most frequent. An increase in Twist1 and epithelial-to-mesenchymal transition (EMT) was recorded. These genetic features might be relevant to metastasis and resistance to chemotherapy. To summarize, the novel patient-derived ES cell line we developed closely mimics the phenotype and genotype of patient tumors, making it a reliable tool for research on metastatic ES.

Identifiants

pubmed: 39475964
doi: 10.1007/s13577-024-01133-3
pii: 10.1007/s13577-024-01133-3
doi:

Substances chimiques

STAG2 protein, human 0
Twist-Related Protein 1 0
TWIST1 protein, human 0
RNA-Binding Protein EWS 0
Nuclear Proteins 0
Oncogene Proteins, Fusion 0
Proto-Oncogene Protein c-fli-1 0
EWSR1-FLI1 fusion protein, human 0
Cohesins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

12

Informations de copyright

© 2024. The Author(s) under exclusive licence to Japan Human Cell Society.

Références

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Auteurs

Chenlu Zhang (C)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Mengling Liu (M)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Lijuan Luan (L)

Department of Pathology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Xi Guo (X)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Yang You (Y)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Zhiming Wang (Z)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Wei Li (W)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Nanhang Lu (N)

Department of Plastic and Reconstructive Surgery, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Yingyong Hou (Y)

Department of Pathology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Lili Lu (L)

Department of Biotherapy Center, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China.

Weiqi Lu (W)

Department of General Surgery, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China. lu.weiqi@zs-hospital.sh.cn.

Yuhong Zhou (Y)

Department of Medical Oncology, Zhongshan Hospital, Fudan University, 180 Fenglin Road, Shanghai, China. zhou.yuhong@zs-hospital.sh.cn.

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