Viral eradication is required for sustained improvement of patient-reported outcomes in patients with hepatitis C.


Journal

Liver international : official journal of the International Association for the Study of the Liver
ISSN: 1478-3231
Titre abrégé: Liver Int
Pays: United States
ID NLM: 101160857

Informations de publication

Date de publication:
01 2019
Historique:
received: 05 03 2018
accepted: 05 06 2018
pubmed: 13 6 2018
medline: 9 1 2020
entrez: 13 6 2018
Statut: ppublish

Résumé

Clearance of chronic HCV infection improves quality of life and other patient-reported outcomes (PROs). Lack of placebo-controlled data led to concerns about the extent of contribution of viral eradication to PRO improvement. To assess PRO changes in HCV patients initially randomized to placebo treatment who received SOF/VEL/VOX in a deferred treatment substudy. HCV-infected direct-acting antivirals-experienced patients who received placebo treatment in POLARIS-1 subsequently received SOF/VEL/VOX (400/100/100 mg) daily for 12 weeks. PROs were prospectively collected using SF-36v2, CLDQ-HCV, FACIT-F, WPAI:SHP. Of 147 patients treated, most were male (79%), white (82%), 33% had cirrhosis, 99% had HCV genotype 1 with SVR-12 of 97%. During treatment with placebo, there were no significant changes in any PROs from patients' own baseline (all P > .05) except for the Worry domain of CLDQ-HCV. However, soon after initiation of treatment with SOF/VEL/VOX, significant PRO improvements were noted: +2.4% to +8.1% of a PRO range size, P < .05 for 6 of the 26 studied PROs, by treatment week 4; +2.0% to +8.3%, P < .05 for 14/26 PROs by treatment week 12. Achieving SVR was associated with similar or greater PRO improvement: +2.5% to +11.9%, P < .05 for 24/26 PROs, by SVR-12; +3.2% to +14.9%, P < .05 for 23/26 PROs, by SVR-24. In multivariate regression analysis, being viraemic was associated with PRO impairment: beta from -2.4% to -8.5%, P < .05 for all but one PRO. Treatment with SOF/VEL/VOX for 12 weeks led to significant and sustainable improvement in patient-reported outcomes in patients who had previously failed another direct-acting antiviral regimen.

Sections du résumé

BACKGROUND
Clearance of chronic HCV infection improves quality of life and other patient-reported outcomes (PROs). Lack of placebo-controlled data led to concerns about the extent of contribution of viral eradication to PRO improvement.
AIM
To assess PRO changes in HCV patients initially randomized to placebo treatment who received SOF/VEL/VOX in a deferred treatment substudy.
METHODS
HCV-infected direct-acting antivirals-experienced patients who received placebo treatment in POLARIS-1 subsequently received SOF/VEL/VOX (400/100/100 mg) daily for 12 weeks. PROs were prospectively collected using SF-36v2, CLDQ-HCV, FACIT-F, WPAI:SHP.
RESULTS
Of 147 patients treated, most were male (79%), white (82%), 33% had cirrhosis, 99% had HCV genotype 1 with SVR-12 of 97%. During treatment with placebo, there were no significant changes in any PROs from patients' own baseline (all P > .05) except for the Worry domain of CLDQ-HCV. However, soon after initiation of treatment with SOF/VEL/VOX, significant PRO improvements were noted: +2.4% to +8.1% of a PRO range size, P < .05 for 6 of the 26 studied PROs, by treatment week 4; +2.0% to +8.3%, P < .05 for 14/26 PROs by treatment week 12. Achieving SVR was associated with similar or greater PRO improvement: +2.5% to +11.9%, P < .05 for 24/26 PROs, by SVR-12; +3.2% to +14.9%, P < .05 for 23/26 PROs, by SVR-24. In multivariate regression analysis, being viraemic was associated with PRO impairment: beta from -2.4% to -8.5%, P < .05 for all but one PRO.
CONCLUSION
Treatment with SOF/VEL/VOX for 12 weeks led to significant and sustainable improvement in patient-reported outcomes in patients who had previously failed another direct-acting antiviral regimen.

Identifiants

pubmed: 29893462
doi: 10.1111/liv.13900
doi:

Substances chimiques

Aminoisobutyric Acids 0
Antiviral Agents 0
Carbamates 0
Cyclopropanes 0
Heterocyclic Compounds, 4 or More Rings 0
Lactams, Macrocyclic 0
Macrocyclic Compounds 0
Quinoxalines 0
Sulfonamides 0
voxilaprevir 0570F37359
Proline 9DLQ4CIU6V
Leucine GMW67QNF9C
velpatasvir KCU0C7RS7Z
Sofosbuvir WJ6CA3ZU8B

Types de publication

Clinical Trial, Phase III Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

54-59

Informations de copyright

© 2018 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Auteurs

Zobair M Younossi (ZM)

Center for Liver Diseases, Department of Medicine, Inova Fairfax Hospital, Falls Church, VA, USA.
Betty and Guy Beatty Center for Integrated Research, Inova Health System, Falls Church, VA, USA.

Maria Stepanova (M)

Center for Outcomes Research in Liver Diseases, Washington, DC, USA.

Rajender Reddy (R)

University of Pennsylvania, Philadelphia, PA, USA.

Michael P Manns (MP)

Department of Gastroenterology, Hepatology and Endocrinology, Hannover Medical School, Meintz, Germany.

Marc Bourliere (M)

Department of Hepato-Gastroenterology, Hospital Saint Joseph, Marseille, France.

Stuart C Gordon (SC)

Department of Gastroenterology, Henry Ford Hospital, Detroit, MI, USA.

Eugene Schiff (E)

Schiff Center For Liver Diseases, University of Miami, Miami, FL, USA.

Tram Tran (T)

Liver Disease and Transplant Center, Cedars-Sinai Medical Center, Los Angeles, LA, USA.

Issah Younossi (I)

Center for Outcomes Research in Liver Diseases, Washington, DC, USA.

Andrei Racila (A)

Center for Outcomes Research in Liver Diseases, Washington, DC, USA.

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Classifications MeSH