Increased Pulmonary-Systemic Pulse Pressure Ratio Is Associated With Increased Mortality in Group 1 Pulmonary Hypertension.


Journal

Heart, lung & circulation
ISSN: 1444-2892
Titre abrégé: Heart Lung Circ
Pays: Australia
ID NLM: 100963739

Informations de publication

Date de publication:
Jul 2019
Historique:
received: 06 09 2017
revised: 14 03 2018
accepted: 29 05 2018
pubmed: 15 7 2018
medline: 28 11 2019
entrez: 15 7 2018
Statut: ppublish

Résumé

Pulmonary arterial hypertension (PAH) is characterised by remodelling of the pulmonary vasculature leading to right ventricular (RV) failure. The failing RV, through interventricular uncoupling, deleteriously impacts the left ventricle and overall cardiac efficiency. We hypothesised that the ratio of the pulmonary artery pulse pressure to the systemic pulse pressure ("pulmonary-systemic pulse pressure ratio", or PS-PPR) would be associated with mortality in PAH. We conducted a retrospective analysis of 262 patients in the National Institute of Health Primary Pulmonary Hypertension Registry (NIH-PPH). We evaluated the association between the PS-PPR and mortality after adjustment for the Pulmonary Hypertension Connection (PHC) risk equation. Among 262 patients (mean age 37.5±15.8years, 62.2% female), median PS-PPR was 1.04 (IQR 0.79-1.30). In the Cox proportional hazards regression model, each one unit increase in the PS-PPR was associated with more than a two-fold increase in mortality during follow-up (HR 2.06, 95% CI 1.40-3.02, p=0.0002), and this association of PS-PPR with mortality remained significant in the multivariable Cox model adjusted for the PHC risk equation, mean pulmonary artery pressure, and body mass index (BMI) (adjusted HR 1.81, 95% CI 1.13-2.88, p=0.01). Furthermore, PS-PPR in the upper quartile (>1.30) versus quartiles 1-3 was associated with a 68% increase in mortality after adjustment for these same covariates (adjusted HR 1.68, 95% CI 1.13-2.50, p=0.01). Pulmonary-systemic pulse pressure ratio, a marker of biventricular efficiency, is associated with survival in PAH even after adjustment for the PHC risk equation. Further studies are needed on the wider applications of PS-PPR in PAH patients.

Sections du résumé

BACKGROUND BACKGROUND
Pulmonary arterial hypertension (PAH) is characterised by remodelling of the pulmonary vasculature leading to right ventricular (RV) failure. The failing RV, through interventricular uncoupling, deleteriously impacts the left ventricle and overall cardiac efficiency. We hypothesised that the ratio of the pulmonary artery pulse pressure to the systemic pulse pressure ("pulmonary-systemic pulse pressure ratio", or PS-PPR) would be associated with mortality in PAH.
METHODS METHODS
We conducted a retrospective analysis of 262 patients in the National Institute of Health Primary Pulmonary Hypertension Registry (NIH-PPH). We evaluated the association between the PS-PPR and mortality after adjustment for the Pulmonary Hypertension Connection (PHC) risk equation.
RESULTS RESULTS
Among 262 patients (mean age 37.5±15.8years, 62.2% female), median PS-PPR was 1.04 (IQR 0.79-1.30). In the Cox proportional hazards regression model, each one unit increase in the PS-PPR was associated with more than a two-fold increase in mortality during follow-up (HR 2.06, 95% CI 1.40-3.02, p=0.0002), and this association of PS-PPR with mortality remained significant in the multivariable Cox model adjusted for the PHC risk equation, mean pulmonary artery pressure, and body mass index (BMI) (adjusted HR 1.81, 95% CI 1.13-2.88, p=0.01). Furthermore, PS-PPR in the upper quartile (>1.30) versus quartiles 1-3 was associated with a 68% increase in mortality after adjustment for these same covariates (adjusted HR 1.68, 95% CI 1.13-2.50, p=0.01).
CONCLUSIONS CONCLUSIONS
Pulmonary-systemic pulse pressure ratio, a marker of biventricular efficiency, is associated with survival in PAH even after adjustment for the PHC risk equation. Further studies are needed on the wider applications of PS-PPR in PAH patients.

Identifiants

pubmed: 30006114
pii: S1443-9506(18)31770-0
doi: 10.1016/j.hlc.2018.05.199
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1059-1066

Informations de copyright

Copyright © 2018 Australian and New Zealand Society of Cardiac and Thoracic Surgeons (ANZSCTS) and the Cardiac Society of Australia and New Zealand (CSANZ). Published by Elsevier B.V. All rights reserved.

Auteurs

Benjamin K Ruth (BK)

Department of Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Kenneth C Bilchick (KC)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Manu M Mysore (MM)

Department of Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Hunter Mwansa (H)

St Vincent Charity Medical Center, Case Western Reserve University, Cleveland, OH, USA.

William C Harding (WC)

Department of Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Younghoon Kwon (Y)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Jamie L W Kennedy (JLW)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Jeremy A Mazurek (JA)

Division of Cardiovascular Medicine, University of Pennsylvania, Philadelphia, PA, USA.

Andrew D Mihalek (AD)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

LaVone A Smith (LA)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Eliany Mejia-Lopez (E)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Alex M Parker (AM)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA.

Timothy S Welch (TS)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA; Cardiology Service Walter Reed National Military Medical Center, Bethesda, MD, USA.

Sula Mazimba (S)

Division of Cardiovascular Medicine, University of Virginia Health System, Charlottesville, VA, USA. Electronic address: SM8SD@hscmail.mcc.virginia.edu.

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