Potential spironolactone effects on collagen metabolism biomarkers in patients with uncontrolled blood pressure.


Journal

Heart (British Cardiac Society)
ISSN: 1468-201X
Titre abrégé: Heart
Pays: England
ID NLM: 9602087

Informations de publication

Date de publication:
02 2019
Historique:
received: 21 02 2018
revised: 21 07 2018
accepted: 26 07 2018
pubmed: 20 8 2018
medline: 8 5 2020
entrez: 20 8 2018
Statut: ppublish

Résumé

An increase in myocardial collagen content may contribute to the development of heart failure; this might be inhibited or reversed by mineralocorticoid receptor antagonists (MRAs). We investigated changes in serum concentrations of the collagen synthesis biomarkers N-terminal propeptide of procollagen type III (PIIINP) (primary outcome) and C-terminal propeptide of procollagen type I (PICP) (secondary outcome) after non-randomised initiation of spironolactone as add-on therapy among patients with resistant hypertension enrolled in the 'Anglo-Scandinavian Cardiac Outcomes' trial (ASCOT). An age/sex matching plus propensity-scored logistic regression model incorporating variables related to the outcome and spironolactone treatment was created to compare patients treated with spironolactone for a 9-month period versus matched controls. A within-person analysis comparing changes in serum biomarker concentrations in the 9 months before versus after spironolactone treatment was also performed. Patients included in the between-person analysis (n=146) were well matched: the mean age was 63±7 years and 11% were woman. Serum concentrations of PIIINP and PICP rose in 'controls' and fell during spironolactone treatment (adjusted means +0.52 (-0.05 to 1.09) vs -0.41 (-0.97 to 0.16) ng/mL, p=0.031 for PIIINP and +4.54(-1.77 to 10.9) vs -6.36 (-12.5 to -0.21) ng/mL, p=0.023 for PICP). For the within-person analysis (n=173), spironolactone treatment was also associated with a reduction in PICP (beta estimate=-11.82(-17.53 to -6.10) ng/mL, p<0.001) but not in PIIINP levels. Treatment with spironolactone was associated with a reduction in serum biomarkers of collagen synthesis independently of blood pressure in patients with hypertension, suggesting that spironolactone might exert favourable effects on myocardial collagen synthesis and fibrosis. Whether this effect might contribute to slowing the progression to heart failure is worth investigating.

Sections du résumé

BACKGROUND
An increase in myocardial collagen content may contribute to the development of heart failure; this might be inhibited or reversed by mineralocorticoid receptor antagonists (MRAs). We investigated changes in serum concentrations of the collagen synthesis biomarkers N-terminal propeptide of procollagen type III (PIIINP) (primary outcome) and C-terminal propeptide of procollagen type I (PICP) (secondary outcome) after non-randomised initiation of spironolactone as add-on therapy among patients with resistant hypertension enrolled in the 'Anglo-Scandinavian Cardiac Outcomes' trial (ASCOT).
METHODS
An age/sex matching plus propensity-scored logistic regression model incorporating variables related to the outcome and spironolactone treatment was created to compare patients treated with spironolactone for a 9-month period versus matched controls. A within-person analysis comparing changes in serum biomarker concentrations in the 9 months before versus after spironolactone treatment was also performed.
RESULTS
Patients included in the between-person analysis (n=146) were well matched: the mean age was 63±7 years and 11% were woman. Serum concentrations of PIIINP and PICP rose in 'controls' and fell during spironolactone treatment (adjusted means +0.52 (-0.05 to 1.09) vs -0.41 (-0.97 to 0.16) ng/mL, p=0.031 for PIIINP and +4.54(-1.77 to 10.9) vs -6.36 (-12.5 to -0.21) ng/mL, p=0.023 for PICP). For the within-person analysis (n=173), spironolactone treatment was also associated with a reduction in PICP (beta estimate=-11.82(-17.53 to -6.10) ng/mL, p<0.001) but not in PIIINP levels.
CONCLUSIONS
Treatment with spironolactone was associated with a reduction in serum biomarkers of collagen synthesis independently of blood pressure in patients with hypertension, suggesting that spironolactone might exert favourable effects on myocardial collagen synthesis and fibrosis. Whether this effect might contribute to slowing the progression to heart failure is worth investigating.

Identifiants

pubmed: 30121630
pii: heartjnl-2018-313182
doi: 10.1136/heartjnl-2018-313182
doi:

Substances chimiques

Biomarkers 0
Mineralocorticoid Receptor Antagonists 0
Peptide Fragments 0
Procollagen 0
procollagen Type III-N-terminal peptide 0
procollagen type I carboxy terminal peptide 0
Spironolactone 27O7W4T232
Collagen 9007-34-5

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

307-314

Informations de copyright

© Author(s) (or their employer(s)) 2019. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

Auteurs

João Pedro Ferreira (JP)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.
Department of Physiology, University of Porto, Porto, Portugal.
Department of Cardiothoracic Surgery, University of Porto, Porto, Portugal.

Patrick Rossignol (P)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

Anne Pizard (A)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

Jean-Loup Machu (JL)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

Timothy Collier (T)

Department of Medical Statistics, London School of Hygiene and Tropical Medicine, London, UK.

Nicolas Girerd (N)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

Anne-Cécile Huby (AC)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

Arantxa Gonzalez (A)

Program of Cardiovascular Diseases, CIMA, University of Navarra and Instituto de Investigación Sanitaria de Navarra (IdiSNA), Pamplona, Spain.
CIBERCV, Carlos III Institute of Health, Madrid, Spain.

Javier Diez (J)

Program of Cardiovascular Diseases, CIMA, University of Navarra and Instituto de Investigación Sanitaria de Navarra (IdiSNA), Pamplona, Spain.
CIBERCV, Carlos III Institute of Health, Madrid, Spain.
Department of Cardiology and Cardiac Surgery, University of Navarra Clinic, Pamplona, Spain.

Begoña López (B)

CIBERCV, Carlos III Institute of Health, Madrid, Spain.

Naveed Sattar (N)

Institute of Cardiovascular and Medical Sciences, British Heart Foundation Glasgow Cardiovascular Research Centre, University of Glasgow, Glasgow, UK.

John G Cleland (JG)

Robertson Centre for Biostatistics and Clinical Trials, University of Glasgow, Glasgow, UK.
National Heart and Lung Institute, Imperial College London, London, UK.

Peter S Sever (PS)

International Centre for Circulatory Health, Imperial College London, London, UK.

Faiez Zannad (F)

Centre d'Investigations Cliniques Plurithématique Inserm 1433, Université de Lorraine, Nancy, France.
CHRU de Nancy, Inserm U1116, Université de Lorraine, Nancy, France.
FCRIN INI-CRCT, Université de Lorraine, Nancy, France.

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