A Broad Test Based on Fluorescent-Multiplex PCR for Noninvasive Prenatal Diagnosis of Cystic Fibrosis.


Journal

Fetal diagnosis and therapy
ISSN: 1421-9964
Titre abrégé: Fetal Diagn Ther
Pays: Switzerland
ID NLM: 9107463

Informations de publication

Date de publication:
2019
Historique:
received: 07 02 2018
accepted: 02 05 2018
pubmed: 20 8 2018
medline: 7 1 2020
entrez: 20 8 2018
Statut: ppublish

Résumé

Analysis of cell-free fetal DNA in maternal plasma is very promising for early diagnosis of monogenic diseases. However, it has been limited by the need to set up patient- or disease-specific custom-made approaches. Here we propose a universal test based on fluorescent multiplex PCR and size fragment analysis for an indirect diagnosis of cystic fibrosis (CF). The test, based on haplotyping, includes nine intra- and extragenic short tandem repeats of the CFTR locus, the coamplification of p.Phe508del (the most frequent mutation in CF patients worldwide), and a specific SRY sequence. The assay is able to determine the inherited paternal allele. Our simple approach was successfully applied to 30 couples and provided clear results from the maternal plasma. The mean rate of informative markers was sufficient to propose it for use in indirect diagnosis. This noninvasive prenatal diagnosis test, focused on indirect diagnosis of CF, offers many advantages over current methods: it is simple, rapid, and cost-effective. It allows for the testing of a large number of couples with high risk of CF, whatever the familial mutation of the CFTR gene. It provides an alternative method to reduce the number of invasive tests.

Sections du résumé

BACKGROUND BACKGROUND
Analysis of cell-free fetal DNA in maternal plasma is very promising for early diagnosis of monogenic diseases. However, it has been limited by the need to set up patient- or disease-specific custom-made approaches. Here we propose a universal test based on fluorescent multiplex PCR and size fragment analysis for an indirect diagnosis of cystic fibrosis (CF).
METHODS METHODS
The test, based on haplotyping, includes nine intra- and extragenic short tandem repeats of the CFTR locus, the coamplification of p.Phe508del (the most frequent mutation in CF patients worldwide), and a specific SRY sequence. The assay is able to determine the inherited paternal allele.
RESULTS RESULTS
Our simple approach was successfully applied to 30 couples and provided clear results from the maternal plasma. The mean rate of informative markers was sufficient to propose it for use in indirect diagnosis.
CONCLUSIONS CONCLUSIONS
This noninvasive prenatal diagnosis test, focused on indirect diagnosis of CF, offers many advantages over current methods: it is simple, rapid, and cost-effective. It allows for the testing of a large number of couples with high risk of CF, whatever the familial mutation of the CFTR gene. It provides an alternative method to reduce the number of invasive tests.

Identifiants

pubmed: 30121677
pii: 000489776
doi: 10.1159/000489776
doi:

Substances chimiques

CFTR protein, human 0
Cell-Free Nucleic Acids 0
Cystic Fibrosis Transmembrane Conductance Regulator 126880-72-6

Types de publication

Journal Article Validation Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

403-412

Informations de copyright

© 2018 S. Karger AG, Basel.

Auteurs

Claire Guissart (C)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France.

Frédéric Tran Mau Them (F)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.

Vanessa Debant (V)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.

Victoria Viart (V)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.

Charlotte Dubucs (C)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.

Victoria Pritchard (V)

Ology Bioservices, Alachua, Florida, USA.

Cécile Rouzier (C)

CHU de Nice, Service de Génétique Médicale, Nice, France.

Amandine Boureau-Wirth (A)

CHU de Nice, Service de Génétique Médicale, Nice, France.

Emmanuelle Haquet (E)

CHU de Montpellier, Service de Génétique Médicale, Montpellier, France.

Jacques Puechberty (J)

CHU de Montpellier, Service de Génétique Médicale, Montpellier, France.

Eric Bieth (E)

CHU de Toulouse, Service de Génétique Médicale, Toulouse, France.

Philippe Khau Van Kien (P)

CHU de Nîmes, Laboratoire de Cytologie Clinique et Cytogénétique, Nîmes, France.

Marie-Pierre Brechard (MP)

Hôpital Saint-Joseph, Service de Diagnostic Prénatal, Marseille, France.

Caroline Raynal (C)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France.

Anne Girardet (A)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France.

Mireille Claustres (M)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France.

Michel Koenig (M)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France.

Marie-Claire Vincent (MC)

Laboratoire de Génétique Moléculaire, IURC, CHRU de Montpellier, Montpellier, France, marie-claire.vincent@inserm.fr.
Equipe Accueil EA7402, Université Montpellier, Montpellier, France, marie-claire.vincent@inserm.fr.

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Classifications MeSH