Enhancing Abiraterone Acetate Efficacy in Androgen Receptor-positive Triple-negative Breast Cancer: Chk1 as a Potential Target.


Journal

Clinical cancer research : an official journal of the American Association for Cancer Research
ISSN: 1557-3265
Titre abrégé: Clin Cancer Res
Pays: United States
ID NLM: 9502500

Informations de publication

Date de publication:
15 01 2019
Historique:
received: 24 05 2018
revised: 28 08 2018
accepted: 18 10 2018
pubmed: 26 10 2018
medline: 28 2 2020
entrez: 25 10 2018
Statut: ppublish

Résumé

Our aim was to identify predictive factors of abiraterone acetate efficacy and putative new druggable targets in androgen receptor (AR)-positive triple-negative breast cancer (TNBC) treated in the UCBG 2012-1 trial. Classic IHC apocrine markers including AR, FOXA1, GGT1, and GCDFP15, from patients' tumors allowed identifying abiraterone acetate-responders and nonresponders. All responders had clear apocrine features. Transcriptome analysis revealed that 31 genes were differentially expressed in the two subgroups, 9 of them being linked to proliferation and DNA damage repair. One of the most significant differences was the overexpression, in nonresponders, of This study suggests that apocrine features can be helpful in the identification of abiraterone acetate-responders. We identified Chk1 as a putative drug target in AR-positive TNBCs.

Identifiants

pubmed: 30352905
pii: 1078-0432.CCR-18-1469
doi: 10.1158/1078-0432.CCR-18-1469
doi:

Substances chimiques

Antineoplastic Agents 0
Biomarkers, Tumor 0
Protein Kinase Inhibitors 0
Receptors, Androgen 0
CHEK1 protein, human EC 2.7.11.1
Checkpoint Kinase 1 EC 2.7.11.1
Abiraterone Acetate EM5OCB9YJ6

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

856-867

Informations de copyright

©2018 American Association for Cancer Research.

Auteurs

Thomas Grellety (T)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France. t.grellety@bordeaux.unicancer.fr.
University of Bordeaux, Bordeaux, France.
Department of Medical Oncology, Institut Bergonié, Comprehensive Cancer Centre Bordeaux, France.

Celine Callens (C)

Pharmacogenomic Unit, Genetics Laboratory, Institut Curie, Paris, France.

Elodie Richard (E)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France.

Adrien Briaux (A)

Pharmacogenomic Unit, Genetics Laboratory, Institut Curie, Paris, France.

Valérie Vélasco (V)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France.
Department of Pathology, Institut Bergonié, Comprehensive Cancer Centre Bordeaux, France.

Marina Pulido (M)

Clinical and Epidemiological Research Unit, Institut Bergonié, INSERM CIC1401, Bordeaux, France.

Anthony Gonçalves (A)

Aix-Marseille Univ, CNRS, INSERM, Institut Paoli-Calmettes, Department of Medical Oncology, CRCM, Marseille, France.

Pierre Gestraud (P)

Institut Curie, PSL Research University, Mines Paris Tech, Bioinformatics and Computational Systems Biology of Cancer, INSERM U900, Paris, France.

Gaetan MacGrogan (G)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France.
Department of Pathology, Institut Bergonié, Comprehensive Cancer Centre Bordeaux, France.

Hervé Bonnefoi (H)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France.
University of Bordeaux, Bordeaux, France.
Department of Medical Oncology, Institut Bergonié, Comprehensive Cancer Centre Bordeaux, France.

Bruno Cardinaud (B)

Institut National de la Santé et de la Recherche Médicale (INSERM) UNIT U1218, Institut Bergonié, Bordeaux, France.
Bordeaux Institut National Polytechnique, Bordeaux, France.

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Classifications MeSH