Mechanistic role of the CREB-regulated transcription coactivator 1 in cardiac hypertrophy.
Animals
Cardiomegaly
/ diagnostic imaging
Cyclic AMP-Dependent Protein Kinases
/ metabolism
HEK293 Cells
Humans
Mice, Inbred C57BL
Myocytes, Cardiac
/ metabolism
Phosphorylation
Promoter Regions, Genetic
RGS Proteins
/ genetics
RNA, Messenger
/ genetics
Rats, Wistar
Receptors, Adrenergic, beta
/ metabolism
Signal Transduction
Transcription Factors
/ deficiency
CREB-regulated transcription coactivator 1
Hypertrophy
Regulator of G protein signaling 2
β-Adrenergic signaling
Journal
Journal of molecular and cellular cardiology
ISSN: 1095-8584
Titre abrégé: J Mol Cell Cardiol
Pays: England
ID NLM: 0262322
Informations de publication
Date de publication:
02 2019
02 2019
Historique:
received:
30
07
2018
revised:
27
11
2018
accepted:
02
12
2018
pubmed:
7
12
2018
medline:
9
1
2020
entrez:
7
12
2018
Statut:
ppublish
Résumé
The sympathetic nervous system is the main stimulator of cardiac function. While acute activation of the β-adrenoceptors exerts positive inotropic and lusitropic effects by increasing cAMP and Ca
Identifiants
pubmed: 30521840
pii: S0022-2828(18)30725-9
doi: 10.1016/j.yjmcc.2018.12.001
pii:
doi:
Substances chimiques
CRTC1 protein, human
0
Crtc1 protein, mouse
0
RGS Proteins
0
RNA, Messenger
0
Receptors, Adrenergic, beta
0
Rgs2 protein, mouse
0
Transcription Factors
0
Cyclic AMP-Dependent Protein Kinases
EC 2.7.11.11
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
31-43Informations de copyright
Copyright © 2018 Elsevier Ltd. All rights reserved.