Contractility kits promote assembly of the mechanoresponsive cytoskeletal network.


Journal

Journal of cell science
ISSN: 1477-9137
Titre abrégé: J Cell Sci
Pays: England
ID NLM: 0052457

Informations de publication

Date de publication:
16 01 2019
Historique:
received: 17 10 2018
accepted: 05 12 2018
pubmed: 19 12 2018
medline: 14 2 2020
entrez: 19 12 2018
Statut: epublish

Résumé

Cellular contractility is governed by a control system of proteins that integrates internal and external cues to drive diverse shape change processes. This contractility controller includes myosin II motors, actin crosslinkers and protein scaffolds, which exhibit robust and cooperative mechanoaccumulation. However, the biochemical interactions and feedback mechanisms that drive the controller remain unknown. Here, we use a proteomics approach to identify direct interactors of two key nodes of the contractility controller in the social amoeba

Identifiants

pubmed: 30559246
pii: jcs.226704
doi: 10.1242/jcs.226704
pmc: PMC6362397
pii:
doi:

Substances chimiques

Actins 0
Microfilament Proteins 0
Protozoan Proteins 0
ctxA protein, Dictyostelium discoideum 0
Myosin Type II EC 3.6.1.-

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : NIGMS NIH HHS
ID : T32 GM007445
Pays : United States
Organisme : NIGMS NIH HHS
ID : F31 GM122258
Pays : United States
Organisme : NIGMS NIH HHS
ID : R01 GM066817
Pays : United States
Organisme : NIA NIH HHS
ID : R21 AG042332
Pays : United States
Organisme : NIH HHS
ID : S10 OD016374
Pays : United States
Organisme : Howard Hughes Medical Institute
Pays : United States

Informations de copyright

© 2019. Published by The Company of Biologists Ltd.

Déclaration de conflit d'intérêts

Competing interestsThe authors declare no competing or financial interests.

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Auteurs

Priyanka Kothari (P)

Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.

Vasudha Srivastava (V)

Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
Department of Chemical and Biomolecular Engineering, Johns Hopkins University, Baltimore, MD 21218, USA.

Vasudha Aggarwal (V)

Department of Biophysics and Biophysical Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.

Irina Tchernyshyov (I)

Department of Medicine, The Smidt Heart Institute and Advanced Clinical Biosystems Institute, Cedar-Sinai Medical Center, Los Angeles, CA 90048, USA.

Jennifer E Van Eyk (JE)

Department of Medicine, The Smidt Heart Institute and Advanced Clinical Biosystems Institute, Cedar-Sinai Medical Center, Los Angeles, CA 90048, USA.

Taekjip Ha (T)

Department of Biophysics and Biophysical Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
Department of Biophysics, Johns Hopkins University, Baltimore, MD 21218, USA.
Department of Biomedical Engineering, Johns Hopkins University, Baltimore, MD 21205, USA.
Howard Hughes Medical Institute, Baltimore, MD 21205, USA.

Douglas N Robinson (DN)

Department of Cell Biology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA dnr@jhmi.edu.
Department of Chemical and Biomolecular Engineering, Johns Hopkins University, Baltimore, MD 21218, USA.
Department of Pharmacology and Molecular Sciences, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.

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