Letrozole versus testosterone for promotion of endogenous puberty in boys with constitutional delay of growth and puberty: a randomised controlled phase 3 trial.


Journal

The Lancet. Child & adolescent health
ISSN: 2352-4650
Titre abrégé: Lancet Child Adolesc Health
Pays: England
ID NLM: 101712925

Informations de publication

Date de publication:
02 2019
Historique:
received: 28 09 2018
revised: 15 11 2018
accepted: 17 11 2018
pubmed: 8 1 2019
medline: 13 5 2020
entrez: 8 1 2019
Statut: ppublish

Résumé

The treatment of constitutional delay of growth and puberty (CDGP) is an underinvestigated area in adolescent medicine. We tested the hypothesis that peroral aromatase inhibition with letrozole is more efficacious than intramuscular injection of low-dose testosterone in inducing puberty in boys with CDGP. We did a randomised, controlled, open-label trial at four paediatric centres in Finland. Boys aged at least 14 years with CDGP who wanted medical intervention and exhibited the first signs of puberty were randomly assigned in blocks of ten to receive either six intramuscular injections of low-dose testosterone (about 1 mg/kg bodyweight) every 4 weeks for 6 months or peroral letrozole 2·5 mg once daily for 6 months. All boys were followed up for 6 months after the end of treatment. The primary outcomes were changes in testicular volume and hormonal markers of puberty at 6 months after treatment initiation, which were assessed in all participants who received the assigned treatment. All patients were included in the safety analysis. This study is registered with ClinicalTrials.gov, number NCT01797718. Between Aug 1, 2013, and Jan 30, 2017, 30 boys were randomly assigned to receive testosterone (n=15) or letrozole (n=15). One boy in the testosterone group was excluded from the primary analyses because of a protocol deviation. During treatment, boys in the letrozole group had higher serum concentrations of luteinising hormone, follicle-stimulating hormone, testosterone, and inhibin B than did boys in the testosterone group. Testicular growth from baseline to 6 months was greater in the letrozole group than in the testosterone group (7·2 mL [95% CI 5·2-9·3] vs 2·2 mL [1·4-2·9]; between-group difference per month 0·9 mL [95% CI 0·6-1·2], p<0·0001). Most adverse events were mild. One boy in the testosterone group had aggressive behaviour for 1 week after each injection, and one boy in the letrozole group had increased irritability at 6 months. Letrozole might be a feasible alternative treatment to low-dose testosterone for boys with CDGP who opt for medical intervention. However, the risks and benefits of manipulating the reproductive axis during early puberty should be weighed carefully. Helsinki University Hospital, Academy of Finland, and Finnish Foundation for Pediatric Research.

Sections du résumé

BACKGROUND
The treatment of constitutional delay of growth and puberty (CDGP) is an underinvestigated area in adolescent medicine. We tested the hypothesis that peroral aromatase inhibition with letrozole is more efficacious than intramuscular injection of low-dose testosterone in inducing puberty in boys with CDGP.
METHODS
We did a randomised, controlled, open-label trial at four paediatric centres in Finland. Boys aged at least 14 years with CDGP who wanted medical intervention and exhibited the first signs of puberty were randomly assigned in blocks of ten to receive either six intramuscular injections of low-dose testosterone (about 1 mg/kg bodyweight) every 4 weeks for 6 months or peroral letrozole 2·5 mg once daily for 6 months. All boys were followed up for 6 months after the end of treatment. The primary outcomes were changes in testicular volume and hormonal markers of puberty at 6 months after treatment initiation, which were assessed in all participants who received the assigned treatment. All patients were included in the safety analysis. This study is registered with ClinicalTrials.gov, number NCT01797718.
FINDINGS
Between Aug 1, 2013, and Jan 30, 2017, 30 boys were randomly assigned to receive testosterone (n=15) or letrozole (n=15). One boy in the testosterone group was excluded from the primary analyses because of a protocol deviation. During treatment, boys in the letrozole group had higher serum concentrations of luteinising hormone, follicle-stimulating hormone, testosterone, and inhibin B than did boys in the testosterone group. Testicular growth from baseline to 6 months was greater in the letrozole group than in the testosterone group (7·2 mL [95% CI 5·2-9·3] vs 2·2 mL [1·4-2·9]; between-group difference per month 0·9 mL [95% CI 0·6-1·2], p<0·0001). Most adverse events were mild. One boy in the testosterone group had aggressive behaviour for 1 week after each injection, and one boy in the letrozole group had increased irritability at 6 months.
INTERPRETATION
Letrozole might be a feasible alternative treatment to low-dose testosterone for boys with CDGP who opt for medical intervention. However, the risks and benefits of manipulating the reproductive axis during early puberty should be weighed carefully.
FUNDING
Helsinki University Hospital, Academy of Finland, and Finnish Foundation for Pediatric Research.

Identifiants

pubmed: 30612946
pii: S2352-4642(18)30377-8
doi: 10.1016/S2352-4642(18)30377-8
pii:
doi:

Substances chimiques

Androgens 0
Aromatase Inhibitors 0
Biomarkers 0
Hormones 0
Testosterone 3XMK78S47O
Letrozole 7LKK855W8I

Banques de données

ClinicalTrials.gov
['NCT01797718']

Types de publication

Clinical Trial, Phase III Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

109-120

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Tero Varimo (T)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Hanna Huopio (H)

Kuopio University Hospital, University of Eastern Finland, Kuopio, Finland.

Laura Kariola (L)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Sirpa Tenhola (S)

Kymenlaakso Central Hospital, Kotka, Finland.

Raimo Voutilainen (R)

Kuopio University Hospital, University of Eastern Finland, Kuopio, Finland.

Jorma Toppari (J)

Department of Pediatrics, Turku University Hospital, and Institute of Biomedicine, Research Centre for Integrative Physiology and Pharmacology, University of Turku, Turku, Finland.

Sanna Toiviainen-Salo (S)

Medical Imaging Center, Department of Pediatric Radiology, Helsinki University Hospital, Helsinki, Finland.

Esa Hämäläinen (E)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland; Department of Clinical Chemistry, University of Helsinki, Helsinki, Finland.

Mari-Anne Pulkkinen (MA)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Mitja Lääperi (M)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Annika Tarkkanen (A)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland; Department of Physiology, Medicum Unit, and Translational Stem Cell Biology and Metabolism Research Program, Faculty of Medicine, University of Helsinki, Helsinki, Finland.

Kirsi Vaaralahti (K)

Department of Physiology, Medicum Unit, and Translational Stem Cell Biology and Metabolism Research Program, Faculty of Medicine, University of Helsinki, Helsinki, Finland.

Päivi J Miettinen (PJ)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Matti Hero (M)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland.

Taneli Raivio (T)

Pediatric Research Center, Children's Hospital, Helsinki University Hospital, Helsinki, Finland; Department of Physiology, Medicum Unit, and Translational Stem Cell Biology and Metabolism Research Program, Faculty of Medicine, University of Helsinki, Helsinki, Finland. Electronic address: taneli.raivio@helsinki.fi.

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Classifications MeSH