The renin-angiotensin system: going beyond the classical paradigms.
Angiotensin I
/ metabolism
Angiotensin-Converting Enzyme 2
Animals
Cardiovascular Agents
/ therapeutic use
Cardiovascular Diseases
/ drug therapy
Cardiovascular System
/ drug effects
Congresses as Topic
Humans
Oligopeptides
/ metabolism
Peptide Fragments
/ metabolism
Peptidyl-Dipeptidase A
/ metabolism
Receptor, Angiotensin, Type 2
/ metabolism
Receptors, G-Protein-Coupled
/ metabolism
Renin-Angiotensin System
/ drug effects
Signal Transduction
/ drug effects
alamandine
angiotensin-(1−7)
angiotensin-(1−9)
angiotensin-converting enzyme 2
heart failure
Journal
American journal of physiology. Heart and circulatory physiology
ISSN: 1522-1539
Titre abrégé: Am J Physiol Heart Circ Physiol
Pays: United States
ID NLM: 100901228
Informations de publication
Date de publication:
01 05 2019
01 05 2019
Historique:
pubmed:
2
2
2019
medline:
17
3
2020
entrez:
2
2
2019
Statut:
ppublish
Résumé
Thirty years ago, a novel axis of the renin-angiotensin system (RAS) was unveiled by the discovery of angiotensin-(1-7) [ANG-(1-7)] generation in vivo. Later, angiotensin-converting enzyme 2 (ACE2) was shown to be the main mediator of this reaction, and Mas was found to be the receptor for the heptapeptide. The functional analysis of this novel axis of the RAS that followed its discovery revealed numerous protective actions in particular for cardiovascular diseases. In parallel, similar protective actions were also described for one of the two receptors of ANG II, the ANG II type 2 receptor (AT
Identifiants
pubmed: 30707614
doi: 10.1152/ajpheart.00723.2018
pmc: PMC7191626
doi:
Substances chimiques
AGTR2 protein, human
0
Cardiovascular Agents
0
MRGPRD protein, human
0
Oligopeptides
0
Peptide Fragments
0
Receptor, Angiotensin, Type 2
0
Receptors, G-Protein-Coupled
0
alamandine
0
Angiotensin I
9041-90-1
Peptidyl-Dipeptidase A
EC 3.4.15.1
ACE2 protein, human
EC 3.4.17.23
Angiotensin-Converting Enzyme 2
EC 3.4.17.23
angiotensin I (1-7)
IJ3FUK8MOF
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
H958-H970Subventions
Organisme : CIHR
Pays : Canada
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