Concurrent isolated retroperitoneal HGSC and STIC defined by somatic mutation analysis: a case report.


Journal

Diagnostic pathology
ISSN: 1746-1596
Titre abrégé: Diagn Pathol
Pays: England
ID NLM: 101251558

Informations de publication

Date de publication:
11 Feb 2019
Historique:
received: 26 11 2018
accepted: 01 02 2019
entrez: 13 2 2019
pubmed: 13 2 2019
medline: 14 6 2019
Statut: epublish

Résumé

Retroperitoneal high-grade serous carcinoma (HGSC) is extremely rare and the origin remains unclear. We present a case of retroperitoneal HGSC and coexisting serous tubal intraepithelial carcinoma (STIC), which is considered as the main origin of ovarian HGSC. We reviewed the available literature and discussed about the origin of this rare disease. A 58-year-old female with a 93 × 65 × 62 mm-solid tumor with a cystic part was located immediately dorsal to the rectum underwent bilateral salpingo-oophorectomy, total abdominal hysterectomy, and en bloc resection of the retroperitoneal tumor together with lower anterior resection of the rectum. Histological diagnosis was retroperitoneal HGSC and STIC at the right fallopian tube. Two deleterious somatic mutations in TP53 and BRCA2 genes were shared between retroperitoneal HGSC and STIC. In addition to clinical features in the previous reports, our genetic findings suggest the origin of retroperitoneal HGSC might be STIC.

Sections du résumé

BACKGROUND BACKGROUND
Retroperitoneal high-grade serous carcinoma (HGSC) is extremely rare and the origin remains unclear. We present a case of retroperitoneal HGSC and coexisting serous tubal intraepithelial carcinoma (STIC), which is considered as the main origin of ovarian HGSC. We reviewed the available literature and discussed about the origin of this rare disease.
CASE PRESENTATION METHODS
A 58-year-old female with a 93 × 65 × 62 mm-solid tumor with a cystic part was located immediately dorsal to the rectum underwent bilateral salpingo-oophorectomy, total abdominal hysterectomy, and en bloc resection of the retroperitoneal tumor together with lower anterior resection of the rectum. Histological diagnosis was retroperitoneal HGSC and STIC at the right fallopian tube. Two deleterious somatic mutations in TP53 and BRCA2 genes were shared between retroperitoneal HGSC and STIC.
CONCLUSIONS CONCLUSIONS
In addition to clinical features in the previous reports, our genetic findings suggest the origin of retroperitoneal HGSC might be STIC.

Identifiants

pubmed: 30744657
doi: 10.1186/s13000-019-0795-3
pii: 10.1186/s13000-019-0795-3
pmc: PMC6371506
doi:

Substances chimiques

BRCA2 Protein 0
BRCA2 protein, human 0
TP53 protein, human 0
Tumor Suppressor Protein p53 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

17

Subventions

Organisme : Japan Society for the Promotion of Science
ID : JP16H06267

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Auteurs

Kazuaki Suda (K)

Department of Obstetrics and Gynecology, Niigata University Graduate School of Medical and Dental Sciences, 1-757 Asahimachi-dori, Niigata, 951-8510, Japan.

Hirofumi Nakaoka (H)

Department of Genetics, School of Life Sciences, Graduate University for Advanced Studies (SOKENDAI), Hayama, Japan.
Division of Human Genetics, National Institute of Genetics, Mishima, Japan.

Chihiro Hata (C)

Department of Genetics, School of Life Sciences, Graduate University for Advanced Studies (SOKENDAI), Hayama, Japan.
Division of Human Genetics, National Institute of Genetics, Mishima, Japan.

Natsumi Yahata (N)

Department of Obstetrics and Gynecology, Niigata Prefectural Shibata Hospital, Shibata, Japan.

Masanori Isobe (M)

Department of Obstetrics and Gynecology, Niigata University Graduate School of Medical and Dental Sciences, 1-757 Asahimachi-dori, Niigata, 951-8510, Japan.

Hitoshi Kameyama (H)

Division of Digestive and General Surgery, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Toshifumi Wakai (T)

Division of Digestive and General Surgery, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Teiichi Motoyama (T)

Department of Molecular and Diagnostic Pathology, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Ituro Inoue (I)

Department of Genetics, School of Life Sciences, Graduate University for Advanced Studies (SOKENDAI), Hayama, Japan.
Division of Human Genetics, National Institute of Genetics, Mishima, Japan.

Kosuke Yoshihara (K)

Department of Obstetrics and Gynecology, Niigata University Graduate School of Medical and Dental Sciences, 1-757 Asahimachi-dori, Niigata, 951-8510, Japan. yoshikou@med.niigata-u.ac.jp.

Takayuki Enomoto (T)

Department of Obstetrics and Gynecology, Niigata University Graduate School of Medical and Dental Sciences, 1-757 Asahimachi-dori, Niigata, 951-8510, Japan.

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Classifications MeSH