Association of childhood pulmonary tuberculosis with exposure to indoor air pollution: a case control study.


Journal

BMC public health
ISSN: 1471-2458
Titre abrégé: BMC Public Health
Pays: England
ID NLM: 100968562

Informations de publication

Date de publication:
07 Mar 2019
Historique:
received: 25 09 2018
accepted: 27 02 2019
entrez: 9 3 2019
pubmed: 9 3 2019
medline: 7 5 2019
Statut: epublish

Résumé

Crude measures of exposure to indicate indoor air pollution have been associated with the increased risk for acquiring tuberculosis. Our study aimed to determine an association between childhood pulmonary tuberculosis (PTB) and exposure to indoor air pollution (IAP), based on crude exposure predictors and directly sampled and modelled pollutant concentrations. In this case control study, children diagnosed with PTB were compared to children without PTB. Questionnaires about children's health; and house characteristics and activities (including household air pollution) and secondhand smoke (SHS) exposure were administered to caregivers of participants. A subset of the participants' homes was sampled for measurements of PM Of the 234 participants, 107 were cases and 127 were controls. Pollutants concentrations (μg/m Our study suggests increased risk of childhood tuberculosis disease when children are exposed to SHS, dirty cooking fuel, and dampness in their homes. Yet, HIV status, age and TB contact are the most important risk factors of childhood PTB in this population.

Sections du résumé

BACKGROUND BACKGROUND
Crude measures of exposure to indicate indoor air pollution have been associated with the increased risk for acquiring tuberculosis. Our study aimed to determine an association between childhood pulmonary tuberculosis (PTB) and exposure to indoor air pollution (IAP), based on crude exposure predictors and directly sampled and modelled pollutant concentrations.
METHODS METHODS
In this case control study, children diagnosed with PTB were compared to children without PTB. Questionnaires about children's health; and house characteristics and activities (including household air pollution) and secondhand smoke (SHS) exposure were administered to caregivers of participants. A subset of the participants' homes was sampled for measurements of PM
RESULTS RESULTS
Of the 234 participants, 107 were cases and 127 were controls. Pollutants concentrations (μg/m
CONCLUSION CONCLUSIONS
Our study suggests increased risk of childhood tuberculosis disease when children are exposed to SHS, dirty cooking fuel, and dampness in their homes. Yet, HIV status, age and TB contact are the most important risk factors of childhood PTB in this population.

Identifiants

pubmed: 30845944
doi: 10.1186/s12889-019-6604-9
pii: 10.1186/s12889-019-6604-9
pmc: PMC6407209
doi:

Substances chimiques

Air Pollutants 0
Tobacco Smoke Pollution 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

275

Subventions

Organisme : FIC NIH HHS
ID : U2R TW007373
Pays : United States
Organisme : National Institutes for Health-Fogarty International (Bethesda, MD, USA) and South African TB/AIDS Research Training (SATBAT, Johannesburg, South Africa) Grant
ID : 5U2RTW00773 and 5U2RTW007373

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Auteurs

Nkosana Jafta (N)

Discipline of Occupational and Environmental Health, School of Nursing and Public Health, University of KwaZulu-Natal, 321 George Campbell Building, Howard College Campus, Durban, 4041, South Africa. jaftan@ukzn.ac.za.

Prakash M Jeena (PM)

Discipline of Pediatrics and Child Health, School of Clinical Medicine, University of KwaZulu-Natal, Private Bag X1, Congella, Durban, 4013, South Africa.

Lars Barregard (L)

Department of Occupational and Environmental Medicine, Sahlgrenska University Hospital and Sahlgrenska Academy at Gothenburg University, Box 414, S-405 30, Gothenburg, Sweden.

Rajen N Naidoo (RN)

Discipline of Occupational and Environmental Health, School of Nursing and Public Health, University of KwaZulu-Natal, 321 George Campbell Building, Howard College Campus, Durban, 4041, South Africa.

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Classifications MeSH