BRL37344 stimulates GLUT4 translocation and glucose uptake in skeletal muscle via β
Adrenergic beta-2 Receptor Agonists
/ pharmacology
Animals
Cell Line
Cyclic AMP
/ metabolism
Ethanolamines
/ pharmacology
Female
Glucose
/ metabolism
Glucose Transporter Type 4
/ genetics
Humans
Kinetics
Mechanistic Target of Rapamycin Complex 2
/ metabolism
Mice, Knockout
Muscle, Skeletal
/ cytology
Myoblasts, Skeletal
/ drug effects
Protein Transport
Rats
Receptors, Adrenergic, beta-2
/ drug effects
Receptors, Adrenergic, beta-3
/ genetics
Signal Transduction
BRL37344
GLUT4
glucose uptake
isoprenaline
receptor desensitization
skeletal muscle
β-adrenoceptor
β-arrestin
Journal
American journal of physiology. Regulatory, integrative and comparative physiology
ISSN: 1522-1490
Titre abrégé: Am J Physiol Regul Integr Comp Physiol
Pays: United States
ID NLM: 100901230
Informations de publication
Date de publication:
01 05 2019
01 05 2019
Historique:
pubmed:
21
3
2019
medline:
20
2
2020
entrez:
21
3
2019
Statut:
ppublish
Résumé
The type 2 diabetes epidemic makes it important to find insulin-independent ways to improve glucose homeostasis. This study examines the mechanisms activated by a dual β
Identifiants
pubmed: 30892909
doi: 10.1152/ajpregu.00285.2018
doi:
Substances chimiques
ADRB2 protein, human
0
ADRB2 protein, mouse
0
Adrb2 protein, rat
0
Adrb3 protein, mouse
0
Adrenergic beta-2 Receptor Agonists
0
Ethanolamines
0
Glucose Transporter Type 4
0
Receptors, Adrenergic, beta-2
0
Receptors, Adrenergic, beta-3
0
SLC2A4 protein, human
0
Slc2a4 protein, mouse
0
BRL 37344
5DZZ1926YW
Cyclic AMP
E0399OZS9N
Mechanistic Target of Rapamycin Complex 2
EC 2.7.11.1
Glucose
IY9XDZ35W2
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM