Effect of Neprilysin Inhibition on Various Natriuretic Peptide Assays.


Journal

Journal of the American College of Cardiology
ISSN: 1558-3597
Titre abrégé: J Am Coll Cardiol
Pays: United States
ID NLM: 8301365

Informations de publication

Date de publication:
26 03 2019
Historique:
received: 05 07 2018
revised: 27 11 2018
accepted: 05 12 2018
entrez: 23 3 2019
pubmed: 23 3 2019
medline: 28 2 2020
Statut: ppublish

Résumé

With sacubitril/valsartan treatment, B-type natriuretic peptide (BNP) concentrations increase; it remains unclear whether change in BNP concentrations is similar across all assays for its measurement. Effects of sacubitril/valsartan on atrial natriuretic peptide (ANP) concentrations in patients are unknown. Lastly, the impact of neprilysin inhibition on mid-regional pro-ANP (MR-proANP), N-terminal pro-BNP (NT-proBNP), proBNP This study sought to examine the effects of sacubitril/valsartan on results from different natriuretic peptide assays. Twenty-three consecutive stable patients with heart failure and reduced ejection fraction were initiated and titrated on sacubitril/valsartan. Change in ANP, MR-proANP, BNP (using 5 assays), NT-proBNP (3 assays), proBNP Average time to 3 follow-up visits was 22, 46, and 84 days. ANP rapidly and substantially increased with initiation and titration of sacubitril/valsartan, more than doubling by the first follow-up visit (+105.8%). Magnitude of ANP increase was greatest in those with concentrations above the median at baseline (+188%) compared with those with lower baseline concentrations (+44%); ANP increases were sustained. Treatment with sacubitril/valsartan led to inconsistent changes in BNP, which varied across methods assessed. Concentrations of MR-proANP, NT-proBNP, and proBNP Initiation and titration of sacubitril/valsartan led to variable changes in concentrations of multiple natriuretic peptides. These results provide important insights into the effects of sacubitril/valsartan treatment on individual patient results, and further suggest the benefit of neprilysin inhibition may be partially mediated by increased ANP concentrations.

Sections du résumé

BACKGROUND
With sacubitril/valsartan treatment, B-type natriuretic peptide (BNP) concentrations increase; it remains unclear whether change in BNP concentrations is similar across all assays for its measurement. Effects of sacubitril/valsartan on atrial natriuretic peptide (ANP) concentrations in patients are unknown. Lastly, the impact of neprilysin inhibition on mid-regional pro-ANP (MR-proANP), N-terminal pro-BNP (NT-proBNP), proBNP
OBJECTIVES
This study sought to examine the effects of sacubitril/valsartan on results from different natriuretic peptide assays.
METHODS
Twenty-three consecutive stable patients with heart failure and reduced ejection fraction were initiated and titrated on sacubitril/valsartan. Change in ANP, MR-proANP, BNP (using 5 assays), NT-proBNP (3 assays), proBNP
RESULTS
Average time to 3 follow-up visits was 22, 46, and 84 days. ANP rapidly and substantially increased with initiation and titration of sacubitril/valsartan, more than doubling by the first follow-up visit (+105.8%). Magnitude of ANP increase was greatest in those with concentrations above the median at baseline (+188%) compared with those with lower baseline concentrations (+44%); ANP increases were sustained. Treatment with sacubitril/valsartan led to inconsistent changes in BNP, which varied across methods assessed. Concentrations of MR-proANP, NT-proBNP, and proBNP
CONCLUSIONS
Initiation and titration of sacubitril/valsartan led to variable changes in concentrations of multiple natriuretic peptides. These results provide important insights into the effects of sacubitril/valsartan treatment on individual patient results, and further suggest the benefit of neprilysin inhibition may be partially mediated by increased ANP concentrations.

Identifiants

pubmed: 30898202
pii: S0735-1097(19)30288-8
doi: 10.1016/j.jacc.2018.12.063
pii:
doi:

Substances chimiques

Aminobutyrates 0
Angiotensin Receptor Antagonists 0
Biphenyl Compounds 0
Drug Combinations 0
Tetrazoles 0
Natriuretic Peptide, Brain 114471-18-0
Valsartan 80M03YXJ7I
Neprilysin EC 3.4.24.11
sacubitril and valsartan sodium hydrate drug combination WB8FT61183

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1273-1284

Commentaires et corrections

Type : CommentIn
Type : CommentIn

Informations de copyright

Copyright © 2019 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Auteurs

Nasrien E Ibrahim (NE)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts; Harvard Medical School, Boston, Massachusetts.

Cian P McCarthy (CP)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts.

Shreya Shrestha (S)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts.

Hanna K Gaggin (HK)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts; Harvard Medical School, Boston, Massachusetts.

Renata Mukai (R)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts.

Jackie Szymonifka (J)

New York University Langone Medical Center, New York, New York.

Fred S Apple (FS)

Laboratory Medicine & Pathology, Hennepin County Medical Center & University of Minnesota, Minneapolis, Minnesota.

John C Burnett (JC)

Mayo Clinic, Rochester, Minnesota.

Seethalakshmi Iyer (S)

Mayo Clinic, Rochester, Minnesota.

James L Januzzi (JL)

Cardiology Division, Massachusetts General Hospital, Boston, Massachusetts; Harvard Medical School, Boston, Massachusetts; Baim Institute for Clinical Research, Boston, Massachusetts. Electronic address: jjanuzzi@partners.org.

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Classifications MeSH