Agreement of 2D transthoracic echocardiography with cardiovascular magnetic resonance imaging after ST-elevation myocardial infarction.


Journal

European journal of radiology
ISSN: 1872-7727
Titre abrégé: Eur J Radiol
Pays: Ireland
ID NLM: 8106411

Informations de publication

Date de publication:
May 2019
Historique:
received: 14 11 2018
revised: 18 02 2019
accepted: 27 02 2019
entrez: 22 4 2019
pubmed: 22 4 2019
medline: 22 6 2019
Statut: ppublish

Résumé

This study was designed to investigate the agreement of 2D transthoracic echocardiography (2D TTE) with cardiovascular magnetic resonance imaging (CMR) in a contemporary population of ST-elevation myocardial infarction (STEMI) patients. In this subanalysis of the GIPS-III trial, a randomized controlled trial investigating the administration of metformin in STEMI patients to prevent reperfusion injury, we studied 259 patients who underwent same-day CMR and 2D TTE assessments four months after hospitalization for a first STEMI. Bland-Altman analyses were performed to assess agreement between LV end-diastolic volume (LVEDV), LV end-systolic volume (LVESV), LV ejection fraction (LVEF), and LV mass measurements. Sensitivity and specificity of 2D TTE to detect categories of LVEF (≤35%, 35-50%, ≥50%) was determined. Linear regression of absolute differences in measurements between imaging modalities was used to investigate whether patient characteristics impact measurement bias. Pairwise difference (bias) and 95% limits of agreement between CMR and 2D TTE measurements were +84 (37, 147) ml for LVEDV, +39 (6, 85) ml for LVESV, -1.1 ± 13.5% for LVEF, and -75 (-154, -14) g for LV mass. Sensitivity and specificity of 2D TTE to detect subjects with moderately depressed LVEF (35-50%) as measured by CMR were 52% and 88% respectively. We observed a significant effect of enzymatic infarct size on bias between 2D TTE and CMR in measuring LVESV and LVEF (P = 0.029, P = 0.001 respectively), of age and sex on bias between 2D TTE and CMR in measuring LV mass (P = 0.027, P < 0.001) and LVEDV (P = 0.001, P = 0.039), and of heart rate on bias between 2D TTE and CMR in LV volume measurements (P = 0.004, P = 0.016). Wide limits of agreement, underestimation of LV volumes and overestimation of LV mass was observed when comparing 2D TTE to CMR. Enzymatic infarct size, age, sex, and heart rate are potential sources of bias between imaging modalities.

Sections du résumé

BACKGROUND BACKGROUND
This study was designed to investigate the agreement of 2D transthoracic echocardiography (2D TTE) with cardiovascular magnetic resonance imaging (CMR) in a contemporary population of ST-elevation myocardial infarction (STEMI) patients.
METHODS METHODS
In this subanalysis of the GIPS-III trial, a randomized controlled trial investigating the administration of metformin in STEMI patients to prevent reperfusion injury, we studied 259 patients who underwent same-day CMR and 2D TTE assessments four months after hospitalization for a first STEMI. Bland-Altman analyses were performed to assess agreement between LV end-diastolic volume (LVEDV), LV end-systolic volume (LVESV), LV ejection fraction (LVEF), and LV mass measurements. Sensitivity and specificity of 2D TTE to detect categories of LVEF (≤35%, 35-50%, ≥50%) was determined. Linear regression of absolute differences in measurements between imaging modalities was used to investigate whether patient characteristics impact measurement bias.
RESULTS RESULTS
Pairwise difference (bias) and 95% limits of agreement between CMR and 2D TTE measurements were +84 (37, 147) ml for LVEDV, +39 (6, 85) ml for LVESV, -1.1 ± 13.5% for LVEF, and -75 (-154, -14) g for LV mass. Sensitivity and specificity of 2D TTE to detect subjects with moderately depressed LVEF (35-50%) as measured by CMR were 52% and 88% respectively. We observed a significant effect of enzymatic infarct size on bias between 2D TTE and CMR in measuring LVESV and LVEF (P = 0.029, P = 0.001 respectively), of age and sex on bias between 2D TTE and CMR in measuring LV mass (P = 0.027, P < 0.001) and LVEDV (P = 0.001, P = 0.039), and of heart rate on bias between 2D TTE and CMR in LV volume measurements (P = 0.004, P = 0.016).
CONCLUSIONS CONCLUSIONS
Wide limits of agreement, underestimation of LV volumes and overestimation of LV mass was observed when comparing 2D TTE to CMR. Enzymatic infarct size, age, sex, and heart rate are potential sources of bias between imaging modalities.

Identifiants

pubmed: 31005178
pii: S0720-048X(19)30092-0
doi: 10.1016/j.ejrad.2019.02.039
pii:
doi:

Substances chimiques

Cardiovascular Agents 0
Metformin 9100L32L2N

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

6-13

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Tom Hendriks (T)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: t.hendriks@umcg.nl.

Lawien Al Ali (L)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: l.al.ali@umcg.nl.

Carlijn G Maagdenberg (CG)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: c.g.maagdenberg@umcg.nl.

Joost P van Melle (JP)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: j.p.van.melle@umcg.nl.

Yoran M Hummel (YM)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: y.m.hummel@umcg.nl.

Matthijs Oudkerk (M)

University of Groningen, University Medical Center Groningen, Center for Medical Imaging, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: m.oudkerk@umcg.nl.

Dirk J van Veldhuisen (DJ)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: d.j.van.veldhuisen@umcg.nl.

Robin Nijveldt (R)

VU University Medical Center, Department of Cardiology, Amsterdam, PO Box 7057, 1007 MB, the Netherlands. Electronic address: robin@nijveldt.net.

Iwan C C van der Horst (ICC)

University of Groningen, University Medical Center Groningen, Department of Critical Care, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: i.c.c.van.der.horst@umcg.nl.

Erik Lipsic (E)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: e.lipsic@umcg.nl.

Pim van der Harst (P)

University of Groningen, University Medical Center Groningen, Department of Cardiology, Groningen, PO box 30.001, 9700 RB, the Netherlands; University of Groningen, University Medical Center Groningen, Center for Medical Imaging, Groningen, PO box 30.001, 9700 RB, the Netherlands. Electronic address: p.van.der.harst@umcg.nl.

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