Malignant Hyperthermia and Cerebral Venous Sinus Thrombosis After Ventriculoperitoneal Shunt in Infant with Schizencephaly and COL4A1 Mutation.


Journal

World neurosurgery
ISSN: 1878-8769
Titre abrégé: World Neurosurg
Pays: United States
ID NLM: 101528275

Informations de publication

Date de publication:
Jul 2019
Historique:
received: 21 02 2019
revised: 16 04 2019
accepted: 17 04 2019
pubmed: 29 4 2019
medline: 21 1 2020
entrez: 29 4 2019
Statut: ppublish

Résumé

Schizencephaly is a rare congenital central nervous system malformation characterized by linear, thickened clefts of the cerebral mantle. Recently, germline mutations in collagen type IV alpha 1 (COL4A1) have been reported to be a genetic cause of schizencephaly as a result of prenatal stroke. Patients with COL4A1 mutation demonstrate a variety of disease phenotypes. However, little is known about the potential complications of patients with COL4A1 mutations before and after neurologic surgery. A 9-month-old boy with schizencephaly and a congenital cataract underwent a ventriculoperitoneal shunt for progressive hydrocephalus. Postoperatively, he developed malignant hyperthermia and cerebral venous thrombosis. Early treatment with dantrolene sodium and hydration was effective. Genetic testing revealed a germline COL4A1 mutation. To our knowledge, malignant hyperthermia and cerebral venous thrombosis have not been reported in the literature in patients with COL4A1 mutations after surgery. Schizencephaly arising from COL4A1 mutations might be a disease prone to these adverse effects because this mutation is known to be associated with venous tortuosity, venous vulnerability, and muscle spasms due to basement membrane protein abnormalities. We need to better understand the wide spectrum of clinical phenotypes of COL4A1 mutations and potential complications in order to better manage surgery of patients with schizencephaly.

Sections du résumé

BACKGROUND BACKGROUND
Schizencephaly is a rare congenital central nervous system malformation characterized by linear, thickened clefts of the cerebral mantle. Recently, germline mutations in collagen type IV alpha 1 (COL4A1) have been reported to be a genetic cause of schizencephaly as a result of prenatal stroke. Patients with COL4A1 mutation demonstrate a variety of disease phenotypes. However, little is known about the potential complications of patients with COL4A1 mutations before and after neurologic surgery.
CASE DESCRIPTION METHODS
A 9-month-old boy with schizencephaly and a congenital cataract underwent a ventriculoperitoneal shunt for progressive hydrocephalus. Postoperatively, he developed malignant hyperthermia and cerebral venous thrombosis. Early treatment with dantrolene sodium and hydration was effective. Genetic testing revealed a germline COL4A1 mutation.
CONCLUSIONS CONCLUSIONS
To our knowledge, malignant hyperthermia and cerebral venous thrombosis have not been reported in the literature in patients with COL4A1 mutations after surgery. Schizencephaly arising from COL4A1 mutations might be a disease prone to these adverse effects because this mutation is known to be associated with venous tortuosity, venous vulnerability, and muscle spasms due to basement membrane protein abnormalities. We need to better understand the wide spectrum of clinical phenotypes of COL4A1 mutations and potential complications in order to better manage surgery of patients with schizencephaly.

Identifiants

pubmed: 31029817
pii: S1878-8750(19)31151-9
doi: 10.1016/j.wneu.2019.04.156
pii:
doi:

Substances chimiques

COL4A1 protein, human 0
Collagen Type IV 0
Muscle Relaxants, Central 0
Dantrolene F64QU97QCR

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

446-450

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Jun Watanabe (J)

Department of Neurosurgery, Brain Research Institute, Niigata University, Niigata, Japan. Electronic address: jwatanabe@bri.niigata-u.ac.jp.

Kouichirou Okamoto (K)

Department of Translational Research, Brain Research Institute, Niigata University, Niigata, Japan.

Tsukasa Ohashi (T)

Department of Pediatrics, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.

Manabu Natsumeda (M)

Department of Neurosurgery, Brain Research Institute, Niigata University, Niigata, Japan.

Hitoshi Hasegawa (H)

Department of Neurosurgery, Brain Research Institute, Niigata University, Niigata, Japan.

Makoto Oishi (M)

Department of Neurosurgery, Brain Research Institute, Niigata University, Niigata, Japan.

Satoko Miyatake (S)

Department of Human Genetics, Yokohama City University Graduate School of Medicine, Yokohama, Kanagawa, Japan.

Naomichi Matsumoto (N)

Department of Human Genetics, Yokohama City University Graduate School of Medicine, Yokohama, Kanagawa, Japan.

Yukihiko Fujii (Y)

Department of Neurosurgery, Brain Research Institute, Niigata University, Niigata, Japan.

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