Co-expression of carcinoembryonic antigen-related cell adhesion molecule 6 and 8 inhibits proliferation and invasiveness of breast carcinoma cells.


Journal

Clinical & experimental metastasis
ISSN: 1573-7276
Titre abrégé: Clin Exp Metastasis
Pays: Netherlands
ID NLM: 8409970

Informations de publication

Date de publication:
10 2019
Historique:
received: 21 02 2019
accepted: 18 06 2019
pubmed: 22 6 2019
medline: 11 4 2020
entrez: 22 6 2019
Statut: ppublish

Résumé

The carcinoembryonic antigen-related cell adhesion molecule (CEACAM) 6 and CEACAM8 form heterodimers and exert their effects. Therefore, we examined the effects of CEACAM6 and CEACAM8 co-expression in breast cancer. We first studied CEACAM6/8 expression using immunohistochemistry in 109 patients with breast cancer. We then established MCF-7 cells that were stably transfected with CEACAM8 and studied cell proliferation, invasion and adhesion. The number of CEACAM6 and CEACAM8 double-positive breast carcinoma cells significantly increased in patients with low histopathological grade and stage. Proximity ligation assay (PLA) confirmed high CEACAM6/8 expression in MCF-7 cells. CEACAM6/8 expression promoted the adhesion of MCF-7 cells to endothelial cell monolayers but inhibited their invasion and proliferation. Furthermore, CEACAM6 status in carcinoma cells was significantly higher in bone than in lung metastases. CEACAM6/8 expression is associated with the inhibition of vascular invasion and cell proliferation. CEACAM6 expression was also considered to be involved in bone metastases of breast cancer. This is the first study to demonstrate the possible role of CEACAM6/8 heterodimer and CEACAM6 expression in breast cancer patients.

Identifiants

pubmed: 31222613
doi: 10.1007/s10585-019-09981-2
pii: 10.1007/s10585-019-09981-2
doi:

Substances chimiques

Antigens, CD 0
Biomarkers, Tumor 0
CEACAM6 protein, human 0
CEACAM8 protein, human 0
Cell Adhesion Molecules 0
GPI-Linked Proteins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

423-432

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Auteurs

Erina Iwabuchi (E)

Department of Pathology, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, 980-8575, Japan.
Japan Society for the Promotion of Science, Tokyo, Japan.

Yasuhiro Miki (Y)

Department of Disaster Obstetrics and Gynecology, International Research Institute of Disaster Science (IRIDes), Tohoku University, Sendai, Japan.

Yoshiaki Onodera (Y)

Department of Pathology, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, 980-8575, Japan.

Yukiko Shibahara (Y)

Department of Pathology, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, 980-8575, Japan.
Laboratory Medicine & Pathobiology, University of Toronto, Toronto, ON, Canada.

Kiyoshi Takagi (K)

Department of Pathology and Histotechnology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Takashi Suzuki (T)

Department of Pathology and Histotechnology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Takanori Ishida (T)

Department of Breast and Endocrine Surgical Oncology, Tohoku University Graduate School of Medicine, Sendai, Japan.

Hironobu Sasano (H)

Department of Pathology, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, 980-8575, Japan. hsasano@patholo2.med.tohoku.ac.jp.

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Classifications MeSH