Layer-specific longitudinal strain in Anderson-Fabry disease at diagnosis: A speckle tracking echocardiography analysis.

Fabry disease left ventricular hypertrophy left ventricular strain left ventricular systolic function speckle tracking echocardiography

Journal

Echocardiography (Mount Kisco, N.Y.)
ISSN: 1540-8175
Titre abrégé: Echocardiography
Pays: United States
ID NLM: 8511187

Informations de publication

Date de publication:
07 2019
Historique:
received: 06 03 2019
revised: 09 05 2019
accepted: 10 05 2019
pubmed: 28 6 2019
medline: 2 10 2020
entrez: 28 6 2019
Statut: ppublish

Résumé

Speckle tracking advancements make now available the analysis of layer-specific myocardial deformation. This study investigated multilayer longitudinal strain in Anderson-Fabry disease (AFD) patients at diagnosis. In a case-control study, 33 newly diagnosed, untreated AFD patients and 33 healthy age- and sex-matched healthy controls underwent a complete echocardiogram, including assessment of left ventricular (LV) transmural global longitudinal strain (GLS), subendocardial longitudinal strain (LSsubendo), subepicardial longitudinal strain (LSsubepi), and strain gradient (LSsubendo-LSsubpepi). Anderson-Fabry disease patients had similar blood pressure, heart rate, and ejection fraction but higher body mass index in comparison with controls. LV mass index, maximal, and relative wall thickness were significantly greater in AFD patients. LSsubendo was significantly higher than LSsubepi in both groups, but GLS (P < 0.0001), LSsubendo (P = 0.003), and particularly LSsubepi (21.4 ± 1.7 vs 18.8 ± 1.4%, P < 0.0001) were lower in AFD patients than in controls. Accordingly, LS gradient was higher in AFD patients (P = 0.003). Three patients symptomatic for dyspnoea presented a combination of LV hypertrophy and reduced LSsubepi. After adjusting for confounders by multivariate analyses, LV mass index or maximal wall thickness were independently and inversely associated with transmural GLS and LSsubepi, but not with LSsubendo in the AFD group. At receiver operating curve curves, LSsubepi best discriminated AFD and normals. In newly diagnosed, untreated AFD patients, layer-specific strain imaging highlights an impairment of LV longitudinal deformation, mainly involving subepicardial strain and causing increase in longitudinal strain myocardial gradient. These findings could be useful for identifying the mechanisms underlying early LV dysfunction in AFD patients.

Sections du résumé

BACKGROUND
Speckle tracking advancements make now available the analysis of layer-specific myocardial deformation. This study investigated multilayer longitudinal strain in Anderson-Fabry disease (AFD) patients at diagnosis.
METHODS
In a case-control study, 33 newly diagnosed, untreated AFD patients and 33 healthy age- and sex-matched healthy controls underwent a complete echocardiogram, including assessment of left ventricular (LV) transmural global longitudinal strain (GLS), subendocardial longitudinal strain (LSsubendo), subepicardial longitudinal strain (LSsubepi), and strain gradient (LSsubendo-LSsubpepi).
RESULTS
Anderson-Fabry disease patients had similar blood pressure, heart rate, and ejection fraction but higher body mass index in comparison with controls. LV mass index, maximal, and relative wall thickness were significantly greater in AFD patients. LSsubendo was significantly higher than LSsubepi in both groups, but GLS (P < 0.0001), LSsubendo (P = 0.003), and particularly LSsubepi (21.4 ± 1.7 vs 18.8 ± 1.4%, P < 0.0001) were lower in AFD patients than in controls. Accordingly, LS gradient was higher in AFD patients (P = 0.003). Three patients symptomatic for dyspnoea presented a combination of LV hypertrophy and reduced LSsubepi. After adjusting for confounders by multivariate analyses, LV mass index or maximal wall thickness were independently and inversely associated with transmural GLS and LSsubepi, but not with LSsubendo in the AFD group. At receiver operating curve curves, LSsubepi best discriminated AFD and normals.
CONCLUSIONS
In newly diagnosed, untreated AFD patients, layer-specific strain imaging highlights an impairment of LV longitudinal deformation, mainly involving subepicardial strain and causing increase in longitudinal strain myocardial gradient. These findings could be useful for identifying the mechanisms underlying early LV dysfunction in AFD patients.

Identifiants

pubmed: 31246327
doi: 10.1111/echo.14399
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1273-1281

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Auteurs

Roberta Esposito (R)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.
Mediterranea Cardiocentro, Naples, Italy.

Ciro Santoro (C)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

Regina Sorrentino (R)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

Eleonora Riccio (E)

Department of Public Medicine, University Federico II, Naples, Italy.

Rodolfo Citro (R)

Department of Cardiology, University Hospital 'San Giovanni di Dio e Ruggi d'Aragona', Salerno, Italy.

Agostino Buonauro (A)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

Teodolinda Di Risi (T)

Department of Molecular Medicine and Medical Biology, University Federico II, Naples, Italy.

Massimo Imbriaco (M)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

Bruno Trimarco (B)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

Antonio Pisani (A)

Department of Public Medicine, University Federico II, Naples, Italy.

Maurizio Galderisi (M)

Department of Advanced Biomedical Sciences, University Federico II, Naples, Italy.

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