Anti-Mesothelin Recombinant Immunotoxin Therapy for Colorectal Cancer.


Journal

Clinical colorectal cancer
ISSN: 1938-0674
Titre abrégé: Clin Colorectal Cancer
Pays: United States
ID NLM: 101120693

Informations de publication

Date de publication:
09 2019
Historique:
received: 04 04 2019
revised: 20 06 2019
accepted: 25 06 2019
pubmed: 28 7 2019
medline: 7 7 2020
entrez: 27 7 2019
Statut: ppublish

Résumé

Mesothelin (MSLN) is a cell surface glycoprotein expressed at a high level on many malignancies, including pancreatic adenocarcinoma, serous ovarian cancer, and epithelioid mesothelioma. MSLN-targeted recombinant immunotoxins (RITs) consist of an anti-MSLN Fv fused to the catalytic domain of Pseudomonas exotoxin A. Recent data has also shown that MSLN is expressed at clinically relevant levels on the surface of colorectal cancer (CRC). In this study, CRC cell lines were tested for MSLN expression and susceptibility to MSLN-targeted RITs. CRC cell lines were tested for membranous MSLN expression via flow cytometry. Cell lines expressing MSLN were tested by WST-8 cell viability assay for sensitivity to various RITs and chemotherapeutic agents. CRC cell line SW-48 was tested in a mouse model for response to RIT as a single agent or in combination with actinomycin D and oxaliplatin. CRC cell lines were susceptible to anti-MSLN RITs at half maximal inhibitory concentration levels comparable with those previously described in pancreatic cancer cell lines. In a nude mouse model, MSLN-targeted RIT treatment of SW48 CRC tumors resulted in a significant decrease in tumor volume. Although combination therapy with standard of care chemotherapeutic oxaliplatin did not improve tumor regressions, combination therapy with actinomycin D resulted in > 90% tumor volume reduction with 50% complete regressions. These data support the development of anti-MSLN RITs as well as other MSLN-targeted therapies for CRC.

Sections du résumé

BACKGROUND
Mesothelin (MSLN) is a cell surface glycoprotein expressed at a high level on many malignancies, including pancreatic adenocarcinoma, serous ovarian cancer, and epithelioid mesothelioma. MSLN-targeted recombinant immunotoxins (RITs) consist of an anti-MSLN Fv fused to the catalytic domain of Pseudomonas exotoxin A. Recent data has also shown that MSLN is expressed at clinically relevant levels on the surface of colorectal cancer (CRC). In this study, CRC cell lines were tested for MSLN expression and susceptibility to MSLN-targeted RITs.
MATERIALS AND METHODS
CRC cell lines were tested for membranous MSLN expression via flow cytometry. Cell lines expressing MSLN were tested by WST-8 cell viability assay for sensitivity to various RITs and chemotherapeutic agents. CRC cell line SW-48 was tested in a mouse model for response to RIT as a single agent or in combination with actinomycin D and oxaliplatin.
RESULTS
CRC cell lines were susceptible to anti-MSLN RITs at half maximal inhibitory concentration levels comparable with those previously described in pancreatic cancer cell lines. In a nude mouse model, MSLN-targeted RIT treatment of SW48 CRC tumors resulted in a significant decrease in tumor volume. Although combination therapy with standard of care chemotherapeutic oxaliplatin did not improve tumor regressions, combination therapy with actinomycin D resulted in > 90% tumor volume reduction with 50% complete regressions.
CONCLUSIONS
These data support the development of anti-MSLN RITs as well as other MSLN-targeted therapies for CRC.

Identifiants

pubmed: 31345777
pii: S1533-0028(19)30176-8
doi: 10.1016/j.clcc.2019.06.006
pmc: PMC8317202
mid: NIHMS1533408
pii:
doi:

Substances chimiques

Antigens, Neoplasm 0
Antineoplastic Agents, Immunological 0
Bacterial Toxins 0
Exotoxins 0
GPI-Linked Proteins 0
Immunotoxins 0
MSLN protein, human 0
Msln protein, mouse 0
Recombinant Proteins 0
Virulence Factors 0
ADP Ribose Transferases EC 2.4.2.-
Mesothelin J27WDC343N

Types de publication

Journal Article Research Support, N.I.H., Intramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

192-199.e1

Subventions

Organisme : Intramural NIH HHS
ID : ZIA BC008753
Pays : United States

Informations de copyright

Published by Elsevier Inc.

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Auteurs

Adam Cerise (A)

Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD.

Tapan K Bera (TK)

Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD.

Xiufen Liu (X)

Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD.

Junxia Wei (J)

Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD.

Ira Pastan (I)

Laboratory of Molecular Biology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD. Electronic address: pastani@mail.nih.gov.

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Classifications MeSH