Anti-IL-2R blockers comparing with polyclonal antibodies: Higher risk of rejection without negative mid-term outcomes after ABO-incompatible kidney transplantation.


Journal

Clinical transplantation
ISSN: 1399-0012
Titre abrégé: Clin Transplant
Pays: Denmark
ID NLM: 8710240

Informations de publication

Date de publication:
10 2019
Historique:
received: 23 05 2019
revised: 12 07 2019
accepted: 23 07 2019
pubmed: 2 8 2019
medline: 12 9 2020
entrez: 2 8 2019
Statut: ppublish

Résumé

There is no recommendation regarding the type of induction therapy to use in ABO-incompatible (ABOi) kidney transplantation. The aim of this retrospective study was to compare the outcome of ABOi living donor kidney transplant (LDKT) recipients who received either polyclonal antibodies or anti-interleukin-2 receptor (IL-2R) blockers as an induction agent. All ABOi HLA-compatible patients that received a LDKT between 03/11 and 03/18 in three French transplantation center (Paris Saint-Louis, Paris Necker, and Toulouse) were included in the study. Fifty-eight patients were given polyclonal antibodies and 39 patients received anti-IL-2R blockers. We identified by a Cox proportional hazard model the use of polyclonal antibodies as a protective factor against acute rejection (HR = 0.4, 95%CI [0-0.9], P < .05). However, pathological findings on protocol biopsies at 1 year were similar in both groups, as were patient and graft survivals, renal function, and complications. We conclude that the acute rejection rate was significantly higher in patients given anti-IL-2R blockers compared to polyclonal antibodies. However, in our series, there was no negative impact on mid-term outcome.

Identifiants

pubmed: 31369170
doi: 10.1111/ctr.13681
doi:

Substances chimiques

ABO Blood-Group System 0
Antibodies 0
Receptors, Interleukin-2 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13681

Informations de copyright

© 2019 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd.

Références

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Auteurs

Arnaud Del Bello (A)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.
Université Paul Sabatier, Toulouse, France.

Gillian Divard (G)

Paris Translational Research Center for Organ Transplantation, Paris, France.

Julie Belliere (J)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.
Université Paul Sabatier, Toulouse, France.

Nicolas Congy-Jolivet (N)

Université Paul Sabatier, Toulouse, France.
Molecular Immunogenetics Laboratory, EA 3034, Faculté de Médecine Purpan, IFR150 (INSERM), Toulouse, France.
Department of Immunology, Hôpital de Rangueil, CHU de Toulouse, Toulouse, France.

Luca Lanfranco (L)

Department of Nephrology, CHU de Brest, Brest, France.

Régine Ricard (R)

Etablissement Francais du Sang, CHU de Purpan, Toulouse, France.

Audrey Delas (A)

Department of Pathology, Institut Universitaire du Cancer, Toulouse, France.

Magali Colombat (M)

Université Paul Sabatier, Toulouse, France.
Department of Pathology, Institut Universitaire du Cancer, Toulouse, France.

Laure Esposito (L)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.

Anne-Laure Hebral (AL)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.

Olivier Cointault (O)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.

Carmen Lefaucheur (C)

Paris Translational Research Center for Organ Transplantation, Paris, France.

Alexandre Loupy (A)

Paris Translational Research Center for Organ Transplantation, Paris, France.

Nassim Kamar (N)

Department of Nephrology and Organ Transplant, CHU Rangueil, Toulouse, France.
Université Paul Sabatier, Toulouse, France.
INSERM U1043, IFR-BMT, CHU Purpan, Toulouse, France.

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