Neonatal diabetes caused by the heterozygous Pro1198Leu mutation in the ABCC8 gene in a male infant: 6-year clinical course.


Journal

Journal of diabetes investigation
ISSN: 2040-1124
Titre abrégé: J Diabetes Investig
Pays: Japan
ID NLM: 101520702

Informations de publication

Date de publication:
Mar 2020
Historique:
received: 29 05 2019
revised: 01 08 2019
accepted: 05 08 2019
pubmed: 8 8 2019
medline: 24 11 2020
entrez: 8 8 2019
Statut: ppublish

Résumé

Neonatal diabetes is a rare disease, often caused by a monogenic abnormality. A male infant patient developed diabetic ketoacidosis at 2 months-of-age due to the heterozygous ABCC8 gene mutation (p.Pro1198Leu). After genetic diagnosis, insulin therapy was successfully transitioned to oral sulfonylurea therapy. For >6 years, oral sulfonylurea therapy has been safe and effective, and the required amount of sulfonylureas has progressively decreased. The mutation was transmitted in an autosomal-dominant fashion across three generations of his family, but the severity of diabetes varied among members from neonatal diabetes to mild diabetes. One family member had normal glucose tolerance despite having the mutation. This case presentation could help in the understanding of neonatal diabetes caused by the ABCC8 gene mutation.

Identifiants

pubmed: 31390154
doi: 10.1111/jdi.13127
pmc: PMC7078085
doi:

Substances chimiques

ABCC8 protein, human 0
Glycated Hemoglobin A 0
Hypoglycemic Agents 0
Insulin 0
Sulfonylurea Receptors 0
hemoglobin A1c protein, human 0
Glyburide SX6K58TVWC

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

502-505

Subventions

Organisme : Japan Society for the Promotion of Science
ID : JP17K09842

Informations de copyright

© 2019 The Authors. Journal of Diabetes Investigation published by Asian Association for the Study of Diabetes (AASD) and John Wiley & Sons Australia, Ltd.

Références

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pubmed: 7502040
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pubmed: 25182307
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pubmed: 17446535

Auteurs

Shinsuke Uraki (S)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Hiroto Furuta (H)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Masakazu Miyawaki (M)

Department of Pediatrics, Wakayama Medical University, Wakayama, Japan.

Norihiko Matsutani (N)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Yuko Shima (Y)

Department of Pediatrics, Wakayama Medical University, Wakayama, Japan.

Miki Iwamoto (M)

Department of Pediatrics, Wakayama Medical University, Wakayama, Japan.

Shohei Matsuno (S)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Shuhei Morita (S)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Machi Furuta (M)

Clinical Laboratory Medicine, Wakayama Medical University, Wakayama, Japan.

Asako Doi (A)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Hiroshi Iwakura (H)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Hiroyuki Ariyasu (H)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

Masahiro Nishi (M)

Department of Clinical Nutrition and Metabolism, Wakayama Medical University, Wakayama, Japan.

Hiroyuki Suzuki (H)

Department of Pediatrics, Wakayama Medical University, Wakayama, Japan.

Takashi Akamizu (T)

First Department of Internal Medicine, Wakayama Medical University, Wakayama, Japan.

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Classifications MeSH