Circulating interleukin-6 as a biomarker in a randomized controlled trial of modified-release prednisone vs immediate-release prednisolone, in newly diagnosed patients with giant cell arteritis.


Journal

International journal of rheumatic diseases
ISSN: 1756-185X
Titre abrégé: Int J Rheum Dis
Pays: England
ID NLM: 101474930

Informations de publication

Date de publication:
Oct 2019
Historique:
received: 06 03 2019
revised: 03 07 2019
accepted: 12 07 2019
pubmed: 19 9 2019
medline: 9 4 2020
entrez: 19 9 2019
Statut: ppublish

Résumé

To measure serial interleukin (IL)-6 levels in newly diagnosed patients with giant cell arteritis (GCA), treated in a randomized controlled trial of modified-release prednisone (MR) vs immediate-release prednisolone (IR) used in a tapering regimen conforming to British Society for Rheumatology GCA guidelines. Patients (n = 12) were randomized into 2 treatment arms (7 MR, 5 IR) and followed over 26 weeks. We measured IL-6 with additional markers. A significantly higher overall mean IL-6 level (P < .05) was seen in IR (mean = 12.15, standard error [SE] = 1.90) compared with MR (mean = 4.39, SE = 1.84). Mean collagen type 1 cross-linked C-telopeptide (CTX) concentration was significantly higher (P < .05) in both groups at week 4 (mean = 0.29, SE = 0.04) compared with week 26 (mean = 0.13, SE = 0.02). MR patients had adrenocorticotropic hormone (ACTH) suppression compared with IR (P < .05) throughout without differences in cortisol levels (P = .34). No significant differences were seen between arms in other markers. Our study suggests that elevated levels of IL-6 in new GCA are better suppressed by MR prednisone compared with IR prednisolone. CTX was significantly reduced in both treatment arms indicating early metabolic effect of glucocorticoids on bone. ACTH suppression with MR prednisone may reflect a greater impact on the hypothalamic-pituitary-adrenal axis although cortisol was not affected. MR prednisone warrants further investigation in GCA.

Identifiants

pubmed: 31531960
doi: 10.1111/1756-185X.13702
doi:

Substances chimiques

Biomarkers 0
Delayed-Action Preparations 0
Glucocorticoids 0
Interleukin-6 0
Prednisolone 9PHQ9Y1OLM
Prednisone VB0R961HZT

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

1900-1904

Subventions

Organisme : Napp Pharmaceuticals Limited

Informations de copyright

© 2019 Asia Pacific League of Associations for Rheumatology and John Wiley & Sons Australia, Ltd.

Références

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GraphPad Software. GraphPad prism version 7.01 for windows 2016.

Auteurs

Emma Miler (E)

Department of Biochemistry, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Philip P Stapleton (PP)

Department of Rheumatology, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Sarah Mapplebeck (S)

Department of Biochemistry, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Craig Mackerness (C)

Research & Development, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Dawn Gayford (D)

Department of Rheumatology, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Tin Aung (T)

Department of Rheumatology, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Lisa Wilson (L)

Department of Biochemistry, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.

Paul Schofield (P)

Napp Pharmaceuticals Limited, Cambridge, UK.

Bhaskar Dasgupta (B)

Department of Rheumatology, Southend University Hospital NHS Foundation Trust, Westcliff-on-Sea, UK.
Honorary Professorship at Essex University, Westcliff-on-Sea, UK.
Visiting Professorship at Anglia Ruskin University, Westcliff-on-Sea, UK.

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